scholarly journals An Extraordinary Case of Silent Extensive Anterior Wall Myocardial Infarction Complicated with Giant Left Ventricular Aneurysm and Dressler Syndrome

2014 ◽  
Vol 04 (06) ◽  
pp. 294-298
Author(s):  
Iat-Lon Leong ◽  
Weng-Chio Tam ◽  
Paul Chan (Chen) ◽  
Zhong-Min Liu
Keyword(s):  
Myocardial Infarction ◽  
Anterior Wall ◽  
Left Ventricular ◽  
Left Ventricular Aneurysm ◽  
Ventricular Aneurysm ◽  
Anterior Wall Myocardial Infarction

Left ventricular aneurysm and prognosis after first anterior wall acute myocardial infarction

1989 ◽  
Vol 63 (5) ◽  
pp. 362-364 ◽  
Author(s):  
Dimitrios Alexopoulos ◽  
Steven F. Horowitz ◽  
Margaret M. Macari-Hinson ◽  
William Slater ◽  
Steven J. Schleifer ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Anterior Wall ◽  
Left Ventricular ◽  
Left Ventricular Aneurysm ◽  
Ventricular Aneurysm

scholarly journals Normal Coronary Artery Patient Presenting with Left Ventricular Aneurysm

2011 ◽  
Vol 2011 ◽  
pp. 1-3 ◽  
Author(s):  
Hakan Altay ◽  
Cihan Altin ◽  
Ali Çoner ◽  
Haldun Muderrisoglu
Keyword(s):  
Myocardial Infarction ◽  
Coronary Artery ◽  
Dilated Cardiomyopathy ◽  
Posterior Wall ◽  
Anterior Wall ◽  
Left Ventricular ◽  
Idiopathic Dilated Cardiomyopathy ◽  
Left Ventricular Aneurysm ◽  
Ventricular Aneurysm ◽  
Distinct Area

Left ventricular aneurysm (LVA) is one of the most important complications of myocardial infarction LVA is strictly defined as a distinct area of abnormal left ventricular diastolic contour with systolic dyskinesia or paradoxical bulging. LVA usually results from myocardial infarction. Other rare aetiologies of LVA include hypertrophic cardiomyopathy, Chagas' disease, sarcoidosis, congenital LVA, and idiopathic However, LVA formation in patients with idiopathic dilated cardiomyopathy is rarely reported, and the incidence, clinical features, and pathogenesis of LVA formation in patients with idiopathic dilated cardiomyopathy is not well understood. Here, we present a 45 years old, idiopathic dilated cardiomyopathy patient with LVA and normal coronary arteries The pathogenesis of LVA formation in patients with idiopathic dilated cardiomyopathy is not clear. One acceptable hypothesis is that coronary artery emboli originate from mural thrombi, present in some patients with idiopathic dilated cardiomyopathy, which develop due to local wall infarction and fibrosis. The local myocardial perfusion differences could be seen in idiopathic dilated cardiomyopathy and predominantly found in the anteroposterior axis of the left ventricle. Local fibrosis occurs more frequently on the anterior wall or posterior wall, and less frequently on the lateral or septal wall. In our patient, LVA existed in the septal segments.We could not define the exact mechanism of the septal aneurysm in our patient but we decided to present this abnormal case, which is different from cases thus far reported in the literature.

SURGICAL MANAGEMENT OF LEFT VENTRICULAR ANEURYSM AS A COMPLICATION OF ACUTE MYOCARDIAL INFARCTION - A SINGLE SURGEON EXPERIENCE

2021 ◽  
pp. 24-26
Author(s):  
Srinivas Kola ◽  
Noel Vijay Paul Bezawada
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Alcohol Consumption ◽  
Anterior Wall ◽  
Left Ventricular ◽  
Left Ventricular Aneurysm ◽  
Care Hospital ◽  
Ventricular Aneurysm ◽  
Surgeon Experience ◽  
Lv Aneurysm

Left ventricular aneurysm is a localized area of the myocardium, with abnormal outward bulging and deformation during systole and diastole, which may be an akinetic, dyskinetic hypokinetic segment. It is due to the weakening of the muscle wall. The aim of this study is the assessment of the Left Ventricular aneurysm, its clinical presentation, Repair technique, and surgical outcome of patients presented with Acute Myocardial infarction with Ventricular wall complications. A retrospective study of the case scenarios that have undergone LV aneurysm repair in a tertiary care hospital facility over four years (2015-2019) after being diagnosed with Acute and evolved Myocardial infarction, which has been rehabilitated by medical management and then referred from cardiology. The Cases with aneurysm were diagnosed by 2d- echocardiography, examined for window period, taken up for surgery, which is re-vascularisation and surgical ventricular remodeling. Of the 6 cases operated, the results were as follows. The mean age of presentation is early for patients with co-morbidities like diabetes and hypertension. Female preponderance is seen. Habitual alcohol consumption causes an early presentation of symptoms. Severe LV dysfunction due to occlusion of the Left coronary artery causes this aneurysm. The most frequent site of aneurysm is the anterior wall with an apex. Apex or Distal anterior wall involvement is repaired by Dor or Linear repair. A septal aneurysm is repaired by septal exclusion by linear Dacron. To conclude, acute MI due to Single or Triple vessel disease can lead to LV aneurysm, which can be Akinetic or Dyskinetic segment.LV Aneurysm as a complication can have a varied presentation, including an Anterior wall, Apex, Apex, and variable extent of the septum and posterior wall, with varied ventricular function with organic valvular MR or Functional MR. Patients with Hypertension and Habitual alcohol consumption have an early age of presentation

scholarly journals Left Ventricular Aneurysm Presenting as a Late Complication of Childhood Chemotherapy

2015 ◽  
Vol 2015 ◽  
pp. 1-5 ◽  
Author(s):  
Braghadheeswar Thyagarajan ◽  
Lubna Bashir Munshi ◽  
Martin Miguel Amor
Keyword(s):  
Myocardial Infarction ◽  
Acute Lymphocytic Leukemia ◽  
Late Complication ◽  
Left Ventricular ◽  
Lymphocytic Leukemia ◽  
Left Ventricular Aneurysm ◽  
Ventricular Aneurysm ◽  
Past Medical History ◽  
History Of ◽  
High Index Of Suspicion

Cardiotoxicity is a well known adverse effect of chemotherapy. Multiple cardiac injuries have been reported including cardiomyopathy, pericarditis, myocarditis, angina, arrhythmias, and myocardial infarction. A left ventricular aneurysm due to chemotherapy is a rare and a dangerous complication which is particularly challenging in diagnosis requiring a high index of suspicion and periodic imaging. We present a case of a young Caucasian male with a past medical history of Acute Lymphocytic Leukemia status after chemotherapy during his childhood diagnosed with left ventricular aneurysm several years later.

scholarly journals COVID-19 Pneumonia Precipitating Acute Anterior Wall Myocardial Infarction With Large Left Ventricular Apical Thrombus

2021 ◽  
Vol 12 (6) ◽  
pp. 251-255
Author(s):  
Sabu John ◽  
Sudhanva Hegde ◽  
Syed Hussain ◽  
Inna Bukharovich ◽  
Suzette Graham-Hill ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Anterior Wall ◽  
Left Ventricular ◽  
Anterior Wall Myocardial Infarction

scholarly journals Improvements in Regional Left Ventricular Function Following Late Percutaneous Coronary Intervention for Anterior Myocardial Infarction

2017 ◽  
Vol 11 ◽  
pp. 117954681774663
Author(s):  
Srilakshmi M Adhyapak ◽  
Prahlad G Menon ◽  
Kiron Varghese ◽  
Abhinav Mehra ◽  
SB Lohitashwa ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Percutaneous Coronary Intervention ◽  
Cardiac Cycle ◽  
Coronary Intervention ◽  
Anterior Wall ◽  
Left Ventricular ◽  
Anterior Myocardial Infarction ◽  
Regional Left Ventricular Function ◽  
Percutaneous Coronary ◽  
Anterior Wall Myocardial Infarction

Background: Late revascularization following a myocardial infarction has questionable clinical benefit. Methods: We studied 13 patients with anterior wall myocardial infarction who underwent percutaneous coronary intervention within 2 weeks of the primary event, by quantitative analysis of 2-dimensional echocardiographic images. Endocardial segmentations of the left ventricular (LV) endocardium from the 4-chamber views were studied over time to establish cumulative wall displacements (CWDs) throughout the cardiac cycle. Results: Left ventricular end-systolic volume decreased to 42 ± 8 mL/body surface area ( P = .034) and LV ejection fraction improved to 52% ± 7% ( P = .04). Analysis of LV endocardial CWD demonstrated significant improvements in mid-systolic to late-systolic phases in the apical LV segments, from 3.5 ± 0.32 to 5.89 ± 0.43 mm ( P = .019). Improvements in CWD were also observed in the late-diastolic phase of the cardiac cycle, from 1.50 ± 0.42 to 1.76 ± 0.52 mm ( P = .04). Conclusions: In our pilot patient cohort, following late establishment of infarct-related artery patency following an anterior wall myocardial infarction, regional improvements were noted in the LV apical segments during systole and late diastole.

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