scholarly journals Preoptic activation and connectivity during thermal sweating in humans

Temperature ◽  
2014 ◽  
Vol 1 (2) ◽  
pp. 135-141 ◽  
Author(s):  
Michael J Farrell ◽  
David Trevaks ◽  
Robin M McAllen
Keyword(s):  
2010 ◽  
Vol 158 (1-2) ◽  
pp. 132
Author(s):  
Yoshiyuki Fujita ◽  
Atsushi Kuga ◽  
Megumi Ubano ◽  
Yoshikazu Uesaka ◽  
Masanari Kunimoto

1965 ◽  
Vol 20 (1) ◽  
pp. 134-136 ◽  
Author(s):  
Roy E. Albert

Thermal sweating from the forehead was suppressed by forced air breathing in two normal male subjects. The decreased sweat rate was associated with symptoms of respiratory alkalosis. This sweat suppression was blocked by the introduction of excess carbon dioxide into the respired gas. sweating and hyperventilation; hyperventilation and sweating; respiratory alkalosis and sweating Submitted on February 7, 1964


1979 ◽  
Vol 92 (2) ◽  
pp. 511-512 ◽  
Author(s):  
A. K. Rai ◽  
B. S. Mehta ◽  
M. Singh

Although sheep combat thermal stress mainly by panting, a sizeable amount (40%) of total evaporative heat loss, is from sources other than panting (Hales & Brown, 1974). The frequency of sporadic discharge of sweat glands increases with increase in ambient temperature and is accompanied by a decline in respiration rate (Bligh, 1961). The wool coat can reduce evaporative cooling but sweating may have cooling value in sheep breeds with open fleeces (Rai, Singh & More, 1978). In sheep, the number and size of the sweat glands (Waites & Voglmayr, 1962) and the quantum of sweat production in a particular time period (Ghoshal et al. 1977) varies in different body regions. In view of the possible significance of surface evaporative cooling, thermal sweating in different body regions of sheep was investigated.


1998 ◽  
Vol 30 (Supplement) ◽  
pp. 283 ◽  
Author(s):  
A. Taimura ◽  
M. Sugawara ◽  
M. Yamauchi ◽  
J. B. Lee ◽  
T. Matsumoto ◽  
...  
Keyword(s):  

1987 ◽  
Vol 65 (6) ◽  
pp. 1329-1332 ◽  
Author(s):  
Tetsuo Nagasaka ◽  
Kozo Hirata ◽  
Tadahiro Nunomura ◽  
Michel Cabanac

Blood flow of the finger and the forearm were measured in five male subjects by venous occlusion plethysmography using mercury-in-Silastic strain gauges in either a cool–dry (COOL: 25 °C, 40% relative humidity), a hot–dry (WARM: 35 °C, 40% relative humidity), or a hot–wet (HOT: 35 °C, 80% relative humidity) environment. One hand or forearm was immersed in a water bath, the temperature (Tw) of which was raised every 10 min by steps of 2 °C until it reached 41° or 43 °C. While the other hand or forearm was kept immersed in a water bath (Tw, 35 °C), blood flow in the heated side (BFw) was compared with the corresponding blood flow in the control side (BFc). Under WARM or HOT conditions, linger BFw was significantly lower than finger BFc at a Tw of 39–41 °C in the majority of subjects. When Tw was raised to 43 °C, however, finger BFw became higher than BFc in nearly half of the subjects. In the COOL state, finger BFw did not decrease but increased steadily when Tw increased from 37° to 43 °C. In the forearm, BFw increased steadily with increasing Tw even in WARM–HOT environments. No such heat-induced vasoconstriction was observed in the forearm. From these results we conclude that in hyperthermic subjects, the rise in local temperature to above the core temperature produces vasoconstriction in the fingers, an area where no thermal sweating takes place.


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