scholarly journals Barrett esophagus

Cell Cycle ◽  
2012 ◽  
Vol 11 (23) ◽  
pp. 4328-4338 ◽  
Author(s):  
Michael Quante ◽  
Julian A. Abrams ◽  
Yoomi Lee ◽  
Timothy C. Wang
Keyword(s):  
Author(s):  
James Saller ◽  
Kun Jiang ◽  
Yin Xiong ◽  
Sean J. Yoder ◽  
Kevin Neill ◽  
...  

2011 ◽  
Vol 33 (7) ◽  
pp. 559-561 ◽  
Author(s):  
Amalia Schiavetti ◽  
Giovanni Di Nardo ◽  
Annapaola Ingrosso ◽  
Damiano Chiriacò ◽  
Salvatore Cucchiara

2005 ◽  
Vol 39 (Supplement 2) ◽  
pp. S33-S41 ◽  
Author(s):  
Hiroshi Mashimo ◽  
Mihir S Wagh ◽  
Raj K Goyal
Keyword(s):  

2010 ◽  
Vol 134 (10) ◽  
pp. 1479-1484 ◽  
Author(s):  
John R. Goldblum

Abstract Context.—Pathologists frequently assess esophageal biopsy specimens to “rule out Barrett esophagus,” as well as to assess for the presence or absence of dysplasia. Objective.—To review some of the recent controversies in the diagnosis of Barrett esophagus and Barrett-related dysplasia. Data Sources.—Sources were the author's experience and review of the English literature from 1978 to 2009. Conclusions.—Although goblet cells are required by the American College of Gastroenterology to confirm a diagnosis of Barrett esophagus, this definition might expand to include columnar-lined esophagus without goblet cells. The recognition of dysplasia in Barrett esophagus remains a difficult task for the surgical pathologist, with difficulties in distinguishing reactive epithelium from dysplasia, low-grade dysplasia from high-grade dysplasia, and even high-grade dysplasia from intramucosal adenocarcinoma.


2020 ◽  
Author(s):  
Jon O. Wee

In most instances, laparoscopy has replaced open procedures as the standard of care. Nevertheless, equipoise remains in the literature regarding the benefits of surgery compared with alternative treatment strategies such as medications in the case of gastroesophageal reflux disease (GERD) or endoscopic procedures in the case of achalasia. According to Society of American Gastrointestinal and Endoscopic Surgeons (SAGES) guidelines published in 2010, indications for surgery include (1) failure of medical management, (2) patient preference, (3) complications of GERD (Barrett esophagus, peptic stricture), and (4) extraesophageal manifestations (asthma, hoarseness, cough, chest pain, aspiration). This chapter is organized by surgical procedure, all of which are derivatives of the laparoscopic Nissen fundoplication. In this chapter, the authors focus on minimally invasive surgical approaches to the treatment of the following benign esophageal disorders: GERD, achalasia, and paraesophageal hernias. New in this chapter is the in-depth coverage of laparoscopic paraesophageal hernia repair. The majority of patients with paraesophageal hernias are asymptomatic, and their hernias are found incidentally with a retrocardiac gastric bubble on an upright chest x-ray or herniated gastroesophageal junction seen on a chest or abdominal computed tomographic scan. For patients who are symptomatic, surgical repair is indicated as there is no medical treatment for this mechanical problem. For asymptomatic patients, clinical judgment needs to be used. All surgical procedures are covered by preoperative evaluation, operative planning, and operative technique, with a troubleshooting note for every step. Procedure complications, postoperative care, and outcome evaluation follow each procedure, listing the most current reports and data. This review contains 10 figures, 9 tables and 49 references Keywords: Minimally invasive surgery, esophagectomy, myotomy, gastroesophageal reflux disease, Barrett esophagus, Nissen fundoplication, fundoplication, paraesophageal hernia


2000 ◽  
Vol 124 (3) ◽  
pp. 411-415 ◽  
Author(s):  
Christopher I. Wilson ◽  
Janna Summerall ◽  
Irvin Willis ◽  
Jack Lubin ◽  
Beria Cabello Inchausti

Abstract We report herein a unique case of an esophageal collision tumor composed of a papillary adenocarcinoma and a large cell neuroendocrine carcinoma arising in a Barrett esophagus. Hematoxylin-eosin and silver staining patterns, immunohistochemistry, and electron microscopy of the large cell neuroendocrine component are discussed.


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