Fatty and defatted herring scrap fed to mink (Mustela vison) in the reproduction and nursing periods

1999 ◽  
Vol 79 (4) ◽  
pp. 457-465 ◽  
Author(s):  
Tove N. Clausen ◽  
Birthe M. Damgaard ◽  
Christian F. Børsting ◽  
Carsten Hejlesen

The use of fatty herring scrap and defatted herring scrap products for female mink in the reproduction and nursing periods was investigated in two experiments. In exp. 1, diets with 27% fatty herring scrap with 60 vs. 120 mg vitamin E kg−1 feed, and 10 vs. 19% defatted herring scrap ensiled with formic and acetic acids were compared with a control diet without fatty herring scrap or defatted herring scrap silage. Each dietary group consisted of 137 females. In exp. 2, diets with 20% fatty herring scrap with 63 vs. 78% of the dietary fat originating from fish, and diets containing 20 vs. 29% defatted herring scrap were compared with a control diet without fatty herring scrap or defatted herring scrap. Each dietary group consisted of 90 females. The results showed that fatty herring scrap, defatted herring scrap and defatted herring scrap silage can be used at these levels during the winter and reproduction periods until parturition, without any negative effects on the number of kits per female at parturition and weaning. However, use of defatted herring scrap silage in the nursing period resulted in lower kit weights at weaning. There were also some indications that high amounts of dietary fat from herring led to reduced kit weights at weaning; therefore, reducing the fat content in herring seemed to be appropriate in order to reduce the content of fish fat in the diet in the nursing period. The recommended addition of 60 mg vitamin E kg−1 wet feed, with an energy density of 5–6 MJ kg−1 during winter, reproduction and nursing periods, was sufficient according to the plasma content of α -tocopherol. Furthermore, there were no negative effects in the nursing period of feeding fatty or defatted herring scrap during the preceding rearing season of the females. Key words: Mink, marine lipid, polyunsaturated fatty acid, haematology, α -tocopherol, acid silage

Nutrients ◽  
2022 ◽  
Vol 14 (2) ◽  
pp. 275
Author(s):  
Yujie Xu ◽  
Jingyuan Xiong ◽  
Wanke Gao ◽  
Xiaoyu Wang ◽  
Shufang Shan ◽  
...  

Dietary fat and fat quality have been inconsistently associated with puberty timing. The aim of this study was to investigate the prospective associations of dietary fat, saturated fatty acid (SFA), polyunsaturated fatty acid (PUFA), and monounsaturated fatty acid (MUFA) with puberty timing. Using longitudinal data from China Health and Nutrition Survey (CHNS) and Southwest China Childhood Nutrition and Growth (SCCNG) Study, we analyzed dietary data, anthropometric measurements, and potential confounders. Dietary intakes were assessed by 3-day 24-h recalls. Age at Tanner stage 2 for breast/genital development (B2/G2) and age at menarche/voice break (M/VB) were used as puberty development markers. Cox proportional hazard regression models were used to estimate the relevance of dietary intake of total fat, SFA, PUFA, and MUFA on puberty timing. Among 3425 girls and 2495 boys, children with higher intakes of total fat and PUFA were more likely to reach their B2/G2 or M/VB at an earlier age. Associations were not attenuated on additional adjustment for childhood dietary protein intake. However, higher intakes of SFA or MUFA were not independently associated with puberty development. A higher intake of dietary fat and PUFA in prepuberty was associated with earlier puberty timing, which was independent of dietary protein intake.


1966 ◽  
Vol 19 (3) ◽  
pp. 147-157 ◽  
Author(s):  
HABIBA HASSAN ◽  
SAMI A. HASHIM ◽  
THEODORE B. VAN ITALLIE ◽  
WILLIAM H. SEBRELL

1991 ◽  
Vol 65 (3) ◽  
pp. 497-503 ◽  
Author(s):  
T. Carbonell ◽  
M. P. Saiz ◽  
M. T. Mitjavila ◽  
P. Puig-Parellada ◽  
C. Cambon-Gros ◽  
...  

Sprague–Dawley rats were fed for 4 months on a control diet or a polyunsaturated-fatty-acid (PUFA)-deficient diet. The combined effects of iron overload (Fe dextran) or Fe deficiency (desferrioxamine) on carrageenan-induced granuloma were studied. PUFA deficiency induced changes in Fe metabolism, but no alterations in lipid peroxidation variables were observed. Inflammation implied an increase in lipid peroxidation, Fe storage and caeruloplasmin concentration, together with symptoms of anaemia. PUFA deficiency in inflamed rats gave rise to a lower inflammatory response (granuloma weight and prostaglandin E2concentration) and ethane exhalation. Fe overload potentiated inflammatory and lipid peroxidation processes, whereas Fe deficiency decreased them.


2005 ◽  
Vol 30 (03) ◽  
Author(s):  
C Franke ◽  
H Demmelmair ◽  
T Decsi ◽  
C Campoy ◽  
K Müller ◽  
...  

1981 ◽  
Vol 5 (2) ◽  
pp. 145-150 ◽  
Author(s):  
Ruben A. Villalaz ◽  
Nancy Toner ◽  
Malcolm L. Chiswick

Nutrients ◽  
2018 ◽  
Vol 10 (12) ◽  
pp. 1963 ◽  
Author(s):  
Minkyeong Kim ◽  
Kyong Park

Dietary fat intake is associated with the risk of colorectal cancer (CRC); however, the results of epidemiological studies on this are controversial. Therefore, this study aimed to summarize the available scientific evidence regarding the association between dietary fat and the risk of CRC. We conducted a systematic search of PubMed, Web of Science, and the Cochrane library for articles related to dietary fat and the risk of CRC. The summary relative risks with 95% confidence intervals (CI) were calculated via a random effect model. Begg’s test was used to detect publication bias. A total of 18 articles were identified. The pooled relative risk with 95% CI for the risk of CRC were 1.00 (95% CI: 0.90–1.12), 0.97 (95% CI: 0.86–1.10), 1.08 (95% CI: 0.92–1.26), and 0.99 (95% CI: 0.93–1.04) for total fat, saturated fatty acid, monounsaturated fatty acid, and polyunsaturated fatty acid, respectively. No significant associations were found in subgroup analyses. Begg’s test for all exposures revealed no publication bias (total fat, p = 0.3; saturated fatty acid, p = 0.1; monounsaturated fatty acid, p = 0.08; polyunsaturated fatty acid, p = 0.2). The studies included in this review and meta-analysis revealed that dietary fats and fatty acids had no effects on the risk of CRC.


2019 ◽  
Vol 97 (Supplement_2) ◽  
pp. 202-202
Author(s):  
Ding Wang ◽  
Young Dal Jang ◽  
G K Rentfrow ◽  
H J Monegue ◽  
M J Azain ◽  
...  

Abstract The study objective was to assess the contribution of fat source and vitamin E (VE) supplementation on tissue tocopherol concentration, antioxidant status, and fatty acid composition in the backfat of pigs at heavy slaughter weight (149.38 ±1.56 kg). A total of 64 individually-fed pigs (32 barrows, 32 gilts; 28.41 ±0.83 kg) were randomly assigned to 8 dietary treatments in a 4 × 2 factorial arrangement. Fat treatments included corn-starch (CS), tallow (TW), corn oil (CO) and coconut oil (CN). VE supplementation levels were 11 and 200 IU/kg. Loin muscle, backfat, and liver samples were collected at slaughter. Data analysis were performed by ANOVA using GLM in SAS. No interactions between dietary fat source and VE supplementation were observed. Increasing dietary VE level from 11 to 200 IU/kg increased (P < 0.001) the tocopherol concentration in both liver (4.73 vs. 21.06 ppm wet liver) and loin muscle (1.25 vs. 2.67 ppm wet muscle). Pigs from the CO group had higher liver SOD activity (P < 0.05) than the other treatments (CS 22.51, TW 22.67, CO 24.40, and CN 20.92 U/mg protein). Pigs from the CN group had the highest (P < 0.05) total saturated fatty acid content (CS 44.33, TW 40.33, CO 36.29, and CN 51.70%), but the lowest (P < 0.05) total polyunsaturated fatty acid content (CS 9.26, TW 9.80, CO 25.41, and CN 8.75%) and iodine value (CS 56.61, TW 60.66, CO 77.83, and CN 49.90) in the backfat. Pigs from the TW group had the highest (P < 0.05) total monounsaturated fatty acid content (CS 46.40, TW 49.87, CO 38.30, and CN 39.56%) in the backfat. Under conditions of this study, a higher level of VE supplementation increased liver and muscle tocopherol concentrations whereas dietary fat sources altered liver antioxidant activity and fatty acid composition in the backfat.


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