scholarly journals Improving hemodynamics – Prolonged intra-aortic balloon pump usage in posterior ventricular septal rupture with right ventricle dysfunction

2020 ◽  
Vol 23 (1) ◽  
pp. 106
Author(s):  
Karthik Raman ◽  
ThapoT Desugari ◽  
S Sowmya ◽  
SamuelS Angula ◽  
Kalaichelvan Uthayakumaran ◽  
...  
Keyword(s):  
Right Ventricle ◽  
Ventricular Septal Rupture ◽  
Intra Aortic Balloon Pump ◽  
Right Ventricle Dysfunction ◽  
Posterior Ventricular Septal Rupture

scholarly journals Right ventricle dysfunction assessment for transcatheter tricuspid valve repair: a matter of debate

2021 ◽  
Author(s):  
Alberto Preda ◽  
Francesco Melillo ◽  
Luca Liberale ◽  
Fabrizio Montecucco ◽  
Eustachio Agricola
Keyword(s):  
Right Ventricle ◽  
Tricuspid Valve ◽  
Tricuspid Valve Repair ◽  
Valve Repair ◽  
Right Ventricle Dysfunction

Posterior Ventricular Septal Rupture: An Anatomical Reconstruction

1998 ◽  
Vol 13 (6) ◽  
pp. 445-450 ◽  
Author(s):  
S. B. Pett ◽  
F. Follis ◽  
K. Allen ◽  
T. Temes ◽  
J. A. Wernly
Keyword(s):  
Anatomical Reconstruction ◽  
Ventricular Septal Rupture ◽  
Posterior Ventricular Septal Rupture

Transatrial approach of acquired posterior ventricular septal rupture and double orifice technique in tricuspid valve repair

2003 ◽  
Vol 76 (2) ◽  
pp. 622-623
Author(s):  
Mario Zogno ◽  
Anna Maizza ◽  
Ernesto Tappainer ◽  
Nicola Pederzolli ◽  
Vinicio Fiorani ◽  
...  
Keyword(s):  
Tricuspid Valve ◽  
Tricuspid Valve Repair ◽  
Ventricular Septal Rupture ◽  
Valve Repair ◽  
Double Orifice ◽  
Posterior Ventricular Septal Rupture

scholarly journals Transcatheter closure of ventricular septal rupture with prolonged support of intra-aortic balloon pump after primary PCI: a case report

2021 ◽  
Vol 21 (1) ◽  
Author(s):  
Chuan Yang ◽  
Yong Sun ◽  
Deling Zou ◽  
Zhaoqing Sun ◽  
Xinzhong Zhang ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Transcatheter Closure ◽  
Interventricular Septum ◽  
Left Ventricular ◽  
Left Ventricular Ejection ◽  
Drug Eluting Stent ◽  
Ventricular Septal Rupture ◽  
Total Occlusion ◽  
Ventricular Ejection Fraction ◽  
Intra Aortic Balloon Pump

Abstract Background Ventricular septal rupture (VSR) is a rare but severe complication of acute myocardial infarction (AMI). For such cases, surgical repair is recommended by major guidelines, but not always possible for such cases. Case presentation A 72-year-old man presented to the emergency room. ECG showed the ST-segment was elevated by 2–3 mm in lead II, III, and aVF, with Q-waves. Coronary angiography (CAG) showed multi-vessel disease with a total occlusion of the right coronary artery (RCA) and severe stenosis of the left anterior descending artery (LAD). A diagnosis of acute inferior myocardial infarction was made. VSR occurred immediately after percutaneous coronary intervention (a 2.5 × 20 mm drug-eluting stent implanted in RCA), and the patient developed cardiogenic shock. An intra-aortic balloon pump (IABP) was used to stabilize the hemodynamics. Transthoracic echocardiography (TTE) revealed an 11.4-mm left-to-right shunt in the interventricular septum. An attempt was made to reduce the IABP augmentation ratio for weaning on day 12 but failed. Transcatheter closure was conducted using a 24-mm double-umbrella occluder on day 28. The patient was weaned from IABP on day 31 and underwent secondary PCI for LAD lesions on day 35. The patient was discharged on day 41. Upon the last follow-up 6 years later, CAG and TTE revealed no in-stent restenosis, no left-to-right shunt, and 51% left ventricular ejection fraction. Conclusions Prolonged implementation of IABP can be a viable option to allow deferred closure of VSR in AMI patients, and transcatheter closure may be considered as a second choice for the selected senior and vulnerable patients, but the risk is still high.

Role of myocardial collagen degradation and fibrosis in right ventricle dysfunction in transposition of the great arteries after atrial switch

2018 ◽  
Vol 258 ◽  
pp. 76-82 ◽  
Author(s):  
Magalie Ladouceur ◽  
Stephanie Baron ◽  
Valérie Nivet-Antoine ◽  
Gérard Maruani ◽  
Gilles Soulat ◽  
...  
Keyword(s):  
Right Ventricle ◽  
Collagen Degradation ◽  
Right Ventricle Dysfunction ◽  
Atrial Switch ◽  
Myocardial Collagen

Using Cardiac Magnetic Resonance Imaging to Evaluate Cardiac Function and Predict Outcomes in Patients with Valvular Heart Disease

2015 ◽  
Vol 10 (1) ◽  
pp. 63-67 ◽  
Author(s):  
Dominic A. Emerson ◽  
Richard L. Amdur ◽  
Jason R. Morrissette ◽  
Federico E. Mordini ◽  
Christian D. Nagy ◽  
...  
Keyword(s):  
Magnetic Resonance Imaging ◽  
Heart Disease ◽  
Cardiac Magnetic Resonance ◽  
Magnetic Resonance ◽  
Right Ventricle ◽  
Valvular Heart Disease ◽  
Regression Models ◽  
Left Ventricular ◽  
Resonance Imaging ◽  
Right Ventricle Dysfunction

Objective In valvular heart disease, elevated left atrial and pulmonary pressures contribute to right ventricular strain and, ultimately, right ventricle failure. Elevated pulmonary artery (PAP) and left ventricular end diastolic pressures are used as markers of right ventricle dysfunction and correlate with poor outcomes. Using cardiac magnetic resonance imaging (CMR), it is possible to directly quantify both left and right ventricular ejection function (LVEF and RVEF), and here, we compare CMR with traditional markers as outcome predictors. Methods A retrospective review of prospectively collected data was performed for patients from January 2004 to February 2008 at a single center (n = 103). Patients were divided into those receiving CMR (n = 56) and those receiving only catheterization (n = 47). Univariate and multivariate logistic regression models were applied to determine predictors of mortality. Finally, predictive models for mortality using PAP, mean PAP, and left ventricular end diastolic pressure were compared to models using LVEF and RVEF obtained from CMR. Results Preoperative average CMR LVEF and RVEF were 57% and 46%, respectively. Only age emerged as an isolated predictor of mortality ( P = 0.01) within the univariate models. Stepwise regression models were created using the catheterization or CMR data. When compared, the CMR model has a slightly better R2, c (prediction accuracy), and sensitivity/specificity (0.22 vs 0.28, 0.77 vs 0.82, and 0.63/0.62 vs 0.69/0.64, respectively). Conclusions Within our population, LVEF and RVEF predict mortality as least as well as traditional catheterization values. Additionally, CMR may identify of elevated PAPs caused by right ventricle dysfunction and those due to other causes, allowing these other causes to be addressed preoperatively.

scholarly journals Mechanisms of right heart disease in pulmonary hypertension (2017 Grover Conference Series)

2017 ◽  
Vol 8 (1) ◽  
pp. 204589321775312 ◽  
Author(s):  
Kewal Asosingh ◽  
Serpil Erzurum
Keyword(s):  
Heart Disease ◽  
Right Ventricle ◽  
Vascular Remodeling ◽  
Cardiac Tissue ◽  
Direct Consequence ◽  
Right Heart Failure ◽  
Right Heart ◽  
Pulmonary Vascular Remodeling ◽  
Right Ventricle Dysfunction ◽  
The Right

Current dogma is that pathological hypertrophy of the right ventricle is a direct consequence of pulmonary vascular remodeling. However, progression of right ventricle dysfunction is not always lung-dependent. Increased afterload caused by pulmonary vascular remodeling initiates the right ventricle hypertrophy, but determinants leading to adaptive or maladaptive hypertrophy and failure remain unknown. Ischemia in a hypertrophic right ventricle may directly contribute to right heart failure. Rapidly enlarging cardiomyocytes switch from aerobic to anaerobic energy generation resulting in cell growth under relatively hypoxic conditions. Cardiac muscle reacts to an increased afterload by over-activation of the sympathetic system and uncoupling and downregulation of β-adrenergic receptors. Recent studies suggest that β blocker therapy in PH is safe, well tolerated, and preserves right ventricle function and cardiac output by reducing right ventricular glycolysis. Fibrosis, an evolutionary conserved process in host defense and wound healing, is dysregulated in maladaptive cardiac tissue contributing directly to right ventricle failure. Despite several mechanisms having been suggested in right heart disease, the causes of maladaptive cardiac remodeling remain unknown and require further research.

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