scholarly journals Successful treatment of anorexia nervosa and alleviation of chronic Guillain Barré syndrome

2011 ◽  
Vol 3 (1) ◽  
pp. 1-2
Author(s):  
Anneke Aden ◽  
Thomas Stegemann ◽  
Andreas Richterich

Eating disorders do not typically occur in conjunction with specific neurological disorders. Only very few cases of Guillain-Barré-Syndrome (GBS) associated with eating disorders have been reported. The objective of this paper is to describe and discuss a case of anorexia nervosa and concomittant chronic GBS. We report on a course of medical management for a 15 year old female patient, who presented with acute neurological syndrome (GBS) which was followed by the onset of a severe eating disorder. The patient was diagnosed to have two different entities, with the association between the two remaining unclear. The mainstay of management was focused on the eating disorder. Using an integrative psychiatric therapy a significant improvement of the eating disorder was achieved. The patient's body weight was stabilised and the locomotor deficits improved. Though a significant somatic disorder was evident, it proved to be advantageous to primarily focus on the eating disorder, until it was under control. The possible correlations between the two distinct disorders are discussed.

2009 ◽  
Vol 405 (1-2) ◽  
pp. 143-147 ◽  
Author(s):  
Han-Lin Chiang ◽  
Rong-Kuo Lyu ◽  
Mu-Yun Tseng ◽  
Kuo-Hsuan Chang ◽  
Hong-Shiu Chang ◽  
...  

Considered a respiratory infection, COVID-19 can lead to neurological pathologies, including encephalitis, stroke, acute disseminated encephalomyelitis and peripheral neuropathies, such as Guillain-Barré syndrome. Guillain-Barré syndrome, also known as acute polyradiculoneuropathy, is an autoimmune disease of the nervous system. Its main manifestation is the acute inflammation of the nerves and nerve roots, attacking the myelin sheath. Bearing in mind the relationship between neurological disorders and COVID-19, the doctor can adopt the best approach for each case, knowing that the neurological manifestations of COVID-19 are varied and that the data on them continue to evolve as the pandemic progresses.


1980 ◽  
Vol 7 (2) ◽  
pp. 108-112 ◽  
Author(s):  
Barbra Goldschmidt ◽  
Joseph Menonna ◽  
Julei Fortunato ◽  
Peter Dowling ◽  
Stuart Cook

2021 ◽  
Vol 14 (1) ◽  
pp. 34-48
Author(s):  
Abdullah Ahmad Tawakul ◽  
Amal Waleed Al-Doboke ◽  
Shahad Ali Altayyar ◽  
Seham Abdulhafith Alsulami ◽  
Ahlam Musallam Alfahmi ◽  
...  

There have been several reported cases of severe acute respiratory syndrome (SARS-CoV-2) infection that were associated with an increased incidence of neurological manifestations, including Guillain–Barré syndrome (GBS). This review aims to present information on the reports of GBS associated with coronavirus disease 2019 (COVID-19) infection. Our review is retrospective work examining articles published from the 1 April 2020 to the 8 May 2021 in the English language. We used the diagnostic criteria and classification published by the National Institute of Neurological Disorders and Stroke and Brighton Collaboration. GBS is usually a postinfectious syndrome, but GBS in the COVID-19 pandemic also takes on a para-infectious profile. In the reports, the genetic factor has a role in developing GBS in some patients. In conclusion, the association between COVID-19 and GBS is not very clear. Still, one mechanism is strongly associated with COVID-19 and immune-mediated neurological complications, which is molecular mimicry between SARS-CoV-2 and human autoantigens.


1985 ◽  
Vol 31 (11) ◽  
pp. 1820-1823 ◽  
Author(s):  
O C Fagnart ◽  
C L Cambiaso ◽  
C J Sindic ◽  
P L Masson

Abstract A fetuin-like antigen was detected (smallest concentration detectable: 5 micrograms/L) by particle-counting immunoassay in 2% (13/641) of consecutive patients' sera but not in sera from 80 healthy blood donors, 40 neonates, or 40 pregnant women. The relation of the presence of detectable antigen to patients' diagnosis is not yet clear. However, in the group with cancer (154), it was found only in two of four patients with nephroblastoma and in three of five with tumors of tissue derived from the neurological crest: retinoblastoma (1/1), neuroblastoma (1/3), and medulloblastoma (1/1). Serum specimens from 422 patients with neurological disorders showed the antigen at a concentration greater than 5 micrograms/L in cases of neurosyphilis (5/11), peripheral neuropathy (12/38), Guillain-Barré syndrome (7/27), and multiple sclerosis (74/184). When we assayed 232 specimens of cerebrospinal fluid from the same neurological patients, we found the antigen in two cases of multiple sclerosis (6 and 15 micrograms/L) and in one case of Guillain-Barré syndrome (54 micrograms/L).


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