On the Protective Effects of IMOD and Silymarin Combination in a Rat Model of Acute Hepatic Failure Through Anti Oxidative Stress Mechanisms

2011 ◽  
Vol 7 (1) ◽  
pp. 38-45 ◽  
Author(s):  
H.R. Rahimi ◽  
M. Gholami ◽  
H.R. Khorram-Kh ◽  
F. Gharibdoos ◽  
M. Abdollahi
Keyword(s):  
Oxidative Stress ◽  
Rat Model ◽  
Hepatic Failure ◽  
Acute Hepatic Failure ◽  
Protective Effects

scholarly journals The protective effects of K 76 in an experimental acute hepatic failure.

1988 ◽  
Vol 11 (3) ◽  
pp. 209-215
Author(s):  
Yasuhiro Mizoguchi ◽  
Hiroshi Kuboi ◽  
Yoshihide Sakagami ◽  
Kenzo Kobayashi ◽  
Sukeo Yamamoto ◽  
...  
Keyword(s):  
Hepatic Failure ◽  
Acute Hepatic Failure ◽  
Protective Effects

Protective Effects of Dibutyryl Cyclic AMP on Acute Hepatic Failure in Rats

1996 ◽  
Vol 66 (1) ◽  
pp. 75-80 ◽  
Author(s):  
Shinichi Kasai ◽  
Akitoshi Kakisaka ◽  
Koichi Kudo ◽  
Michio Mito
Keyword(s):  
Cyclic Amp ◽  
Hepatic Failure ◽  
Acute Hepatic Failure ◽  
Protective Effects ◽  
Dibutyryl Cyclic Amp

Protective Effects of Honey Bee Products against Oxidative Stress-Associated Carcinogenesis in a Rat Model of Colon Cancer

2019 ◽  
Vol 7 (2) ◽  
pp. 62-78
Author(s):  
Hajer Ibrahim Salim Al-Ajmi ◽  
◽  
Hassan Talib Al-Lawati ◽  
Mohammad Shafiur Rahman ◽  
Nejib Guizani
Keyword(s):  
Oxidative Stress ◽  
Colon Cancer ◽  
Honey Bee ◽  
Rat Model ◽  
Protective Effects

scholarly journals Protective effects of epifriedelinol in a rat model of traumatic brain injury assessed with histological and hematological markers

2018 ◽  
Vol 9 (1) ◽  
pp. 38-42 ◽  
Author(s):  
Shiping Li ◽  
Qiaoying Zhang ◽  
Peiwu Li
Keyword(s):  
Oxidative Stress ◽  
Traumatic Brain Injury ◽  
Brain Injury ◽  
Water Content ◽  
Rat Model ◽  
Protective Effects ◽  
Hematological Markers ◽  
Cytokine Levels ◽  
Factor Α ◽  
And Oxidative Stress

Abstract Background This study evaluated the protective effects of epifriedelinol (EFD) in a rat model of traumatic brain injury (TBI). Methodology TBI was induced by dropping a weight from a specific height. The animals were separated into control, TBI, and EFD 100 and 200 mg/kg groups. The latter received 100 and 200 mg/kg EFD, respectively, for 2 days beginning 30 min after inducing TBI. The neurological examination score, permeability of the blood–brain barrier (BBB), water content of the brain, cytokine levels, and oxidative stress parameters were measured in the rats. The effects of EFD on glial fibrillary acidic protein (GFAP)-positive cells were evaluated using immunohistochemistry. ResultThe EFD treatment significantly decreased the neurological score, permeability of the BBB, and water content of brain compared with the TBI group. The levels of interleukin-1β (IL-1β), tumor necrosis factor-α (TNF-α), and oxidative stress were significantly decreased in the EFD-treated groups. The number of GFAP-positive cells was also significantly reduced in the EFD-treated groups. ConclusionEFD attenuates the secondary injury in TBI rats by reducing the serum cytokine levels and oxidative stress.

scholarly journals MP67-07 THE PROTECTIVE EFFECTS OF PROPOLIS ON OXIDATIVE STRESS AND CRYSTAL DEPOSITION WITH HYPEROXALURIA INDUCED TUBULAR CELL INJURY IN RAT MODEL

2016 ◽  
Vol 195 (4S) ◽  
Author(s):  
Kamil Fehmi Narter ◽  
Alper Kafkasli ◽  
Oguz Ozturk ◽  
Ozgur Yazici ◽  
Bilal Hamarat ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Rat Model ◽  
Cell Injury ◽  
Tubular Cell ◽  
Protective Effects ◽  
Crystal Deposition
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