scholarly journals A Case of Umbilical Hernia in which Internal Hernia Occurred in the Hernia sac, Causing a Colon Skin Fistula

2016 ◽  
Vol 77 (8) ◽  
pp. 2106-2110
Author(s):  
Kazutaka KIMURA ◽  
Teruhiko MINAGAWA ◽  
Mari MORITA ◽  
Yukitake HASEBE
1988 ◽  
Vol 30 (1) ◽  
pp. 71-75 ◽  
Author(s):  
Beverly W. Baron ◽  
Wolfgang H. Schraut ◽  
Freidoon Azizi ◽  
A. Talerman
Keyword(s):  

2018 ◽  
Vol 2018 ◽  
pp. 1-4
Author(s):  
Deepti M. Reddi ◽  
Kathryn P. Scherpelz ◽  
Angelica Lerma ◽  
Jabi Shriki ◽  
Jeffrey Virgin

Hernia sacs are a common anatomic pathology specimen, which rarely contain malignancy. We present a case of rapidly growing pancreatic adenocarcinoma, which initially presented as metastasis to an umbilical hernia sac. The patient was a 55-year-old male with a two-year history of umbilical hernia. Two months prior to herniorrhaphy, the hernia became painful and the patient experienced nausea and weight loss. The gross examination did not reveal distinct lesions. Microscopically, the hernia sac was diffusely infiltrated by moderately differentiated adenocarcinoma, which was positive for CK7 and pancytokeratin and negative for TTF-1, CK20, PSA, and CDX2. Clinical laboratory tests found elevated levels of CA 19-9 and CEA. Computed tomography scan with intravenous contrast showed a 5 cm ill-defined and hypoattenuating mass involving the pancreatic tail and body, as well as numerous ill-defined lesions in the liver and peritoneal carcinomatosis. The patient had an earlier noncontrast computed tomography scan four months prior to the surgery, which did not detect any lesions in the abdomen. This case highlights the importance of intravenous contrast with computed tomography for the evaluation of pancreatic lesions and also emphasizes the importance of thorough histologic evaluation of hernia sacs for the detection of occult malignancy.


2016 ◽  
Vol 5 (3) ◽  
pp. 30 ◽  
Author(s):  
Yogender Singh Kadian ◽  
Anjali Verma ◽  
Kamal Nain Rattan ◽  
Pardeep Kajal

Background: Vitellointestinal duct (VID) or omphalomesenteric duct anomalies are secondary to the persistence of the embryonic vitelline duct, which normally obliterates by weeks 5–9 of intrauterine life.Methods: This is a retrospective analysis of a total of 16 patients of symptomatic remnants of vitellointestinal duct from period of Jan 2009 to May 2013.Results: Male to female ratio (M:F) was 4.3:1 and mean age of presentation was 2 months and their mode of presentation was: patent VID in 9 (56.25%) patients, umbilical cyst in 2(12.25%), umbilical granuloma in 2 (12.25%), and Meckel diverticulum as content of hernia sac in obstructed umbilical hernia in 1 (6.25%) patient. Two patients with umbilical fistula had severe electrolyte disturbance and died without surgical intervention.Conclusion: Persistent VID may have varied presentations in infancy. High output umbilical fistula and excessive bowel prolapse demand urgent surgical intervention to avoid morbidity and mortality.


2017 ◽  
Vol 4 (4) ◽  
pp. 1481 ◽  
Author(s):  
Prakash Kumar Sahoo ◽  
Suman Saurav Rout

Littré’s hernia is caused by the protrusion of Meckel’s diverticulum through an orifice in the abdominal wall. Meckel’s diverticulum is the most common congenital anomaly of the gastrointestinal tract that is generally asymptomatic and only manifests in a specific way when complications exist. An unusual complication of Meckel’s diverticulum is known as Littre’s hernia. It comprises less than 1% of all Meckel’s diverticulum. Usual sites of Littre hernia are right inguinal (50% of cases), umbilical hernia (20%), and femoral hernia (20%). We present a case of Littré’s hernia where we found a strangulated Meckel’s diverticulum in an inguinal hernia sac.


2021 ◽  
Vol 9 (1) ◽  
pp. 236
Author(s):  
Venu Bharagava Malpuri ◽  
Prasanth Gurijala ◽  
Bhaskar Reddy Yerrola ◽  
Krishna Ramavath ◽  
Gopisingh Lavudya

Internal hernias have the potential to cause small bowel obstruction. Congenital internal hernias are impossible to diagnose clinically and radiologically in asymptomatic patients. We presented a case of 36 years male with complaints of pain abdomen abdominal distension and vomiting, contrast-enhanced CT showed an internal hernia with small bowel obstruction. On exploration, small bowel loops were identified near the lesser curvature and they are congested an edema was present, a defect of 5×1 cm was identified in the transverse mesocolon and was managed by reducing the hernia sac and closure of the defect in the mesentery of the transverse colon. If the intervention was delayed internal hernia might lead to ischemia, gangrene increasing morbidity and mortality. Early intervention is the key to decrease morbidity and mortality. 


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