scholarly journals THREE CASES OF FITZ-HUGH-CURTIS SYNDROME DUE TO CHLAMYDIA TRACHOMATIS INFECTION-ANALYSIS OF US AND CT FINDINGS-

2005 ◽  
Vol 66 (1) ◽  
pp. 135-139
Author(s):  
Nagato SHIMADA ◽  
Tomoaki KANEKO ◽  
Toshihide ITO ◽  
Taro NAKANO ◽  
Kazuo UEDA ◽  
...  
Keyword(s):  
Chlamydia Trachomatis ◽  
Chlamydia Trachomatis Infection ◽  
Ct Findings ◽  
Fitz Hugh Curtis Syndrome

scholarly journals A Operated Case of Ileus Caused by Perihepatitis (Fitz-Hugh-Curtis Syndrome) Supposedly due to Chlamydia Trachomatis Infection.

2001 ◽  
Vol 34 (8) ◽  
pp. 1331-1335
Author(s):  
Hiroki Hayakawa ◽  
Masahiro Suenaga ◽  
Jyunichi Tobinaga ◽  
Yuuki Takeuchi ◽  
Masashi Uchimura ◽  
...  
Keyword(s):  
Chlamydia Trachomatis ◽  
Chlamydia Trachomatis Infection ◽  
Fitz Hugh Curtis Syndrome

Chlamydia trachomatis infection in Fitz-Hugh-Curtis syndrome

1980 ◽  
Vol 138 (7) ◽  
pp. 1034-1038 ◽  
Author(s):  
San-Pin Wang ◽  
David A. Eschenbach ◽  
King K. Holmes ◽  
Gael Wager ◽  
J.Thomas Grayston
Keyword(s):  
Chlamydia Trachomatis ◽  
Chlamydia Trachomatis Infection ◽  
Fitz Hugh Curtis Syndrome

Fitz-Hugh–Curtis Syndrome Caused by Chlamydia trachomatis: Atypical CT Findings

2004 ◽  
Vol 182 (3) ◽  
pp. 822-824 ◽  
Author(s):  
Benoît Mesurolle ◽  
François Mignon ◽  
Jean H. Gagnon
Keyword(s):  
Chlamydia Trachomatis ◽  
Ct Findings ◽  
Fitz Hugh Curtis Syndrome

scholarly journals A Case of Fitz-Hugh-Curtis Syndrome Developed Ileus due to Chlamydia Trachomatis Infection.

1998 ◽  
Vol 31 (12) ◽  
pp. 2374-2378
Author(s):  
Isao Matsumoto ◽  
Ichiro Takahashi ◽  
Makoto Shinagawa ◽  
Masayuki Yoshida ◽  
Shiro Yamazaki ◽  
...  
Keyword(s):  
Chlamydia Trachomatis ◽  
Chlamydia Trachomatis Infection ◽  
Fitz Hugh Curtis Syndrome

scholarly journals P3.026 Low Prevalence of Chlamydia Trachomatis Infection: Fact or Missed Diagnosis?

2013 ◽  
Vol 89 (Suppl 1) ◽  
pp. A156.1-A156
Author(s):  
T Bharara ◽  
D Rawat ◽  
P Bhalla ◽  
V K Garg ◽  
K Sardana ◽  
...  
Keyword(s):  
Chlamydia Trachomatis ◽  
Chlamydia Trachomatis Infection ◽  
Missed Diagnosis ◽  
Low Prevalence

scholarly journals Differential Regulation of Inflammatory Cytokine Secretion by Human Dendritic Cells upon Chlamydia trachomatis Infection

2004 ◽  
Vol 72 (12) ◽  
pp. 7231-7239 ◽  
Author(s):  
Ana Gervassi ◽  
Mark R. Alderson ◽  
Robert Suchland ◽  
Jean François Maisonneuve ◽  
Kenneth H. Grabstein ◽  
...  
Keyword(s):  
Dendritic Cells ◽  
Chlamydia Trachomatis ◽  
Wide Spectrum ◽  
Cytokine Secretion ◽  
Host Cells ◽  
Microbial Pathogens ◽  
Potential Contribution ◽  
Necrosis Factor Alpha ◽  
Chlamydia Trachomatis Infection ◽  
Tnf Α

ABSTRACT Chlamydia trachomatis is an obligate intracellular gram-negative bacterium responsible for a wide spectrum of diseases in humans. Both genital and ocular C. trachomatis infections are associated with tissue inflammation and pathology. Dendritic cells (DC) play an important role in both innate and adaptive immune responses to microbial pathogens and are a source of inflammatory cytokines. To determine the potential contribution of DC to the inflammatory process, human DC were infected with C. trachomatis serovar E or L2. Both C. trachomatis serovars were found to infect and replicate in DC. Upon infection, DC up-regulated the expression of costimulatory (B7-1) and cell adhesion (ICAM-1) molecules. Furthermore, chlamydial infection induced the secretion of interleukin-1β (IL-1β), IL-6, IL-8, IL-12p70, IL-18, and tumor necrosis factor alpha (TNF-α). The mechanisms involved in Chlamydia-induced IL-1β and IL-18 secretion differed from those of the other cytokines. Chlamydia-induced IL-1β and IL-18 secretion required infection with viable bacteria and was associated with the Chlamydia-induced activation of caspase-1 in infected host cells. In contrast, TNF-α and IL-6 secretion did not require that the Chlamydia be viable, suggesting that there are at least two mechanisms involved in the Chlamydia-induced cytokine secretion in DC. Interestingly, an antibody to Toll-like receptor 4 inhibited Chlamydia-induced IL-1β, IL-6, and TNF-α secretion. The data herein demonstrate that DC can be infected by human C. trachomatis serovars and that chlamydial components regulate the secretion of various cytokines in DC. Collectively, these data suggest that DC play a role in the inflammatory processes caused by chlamydial infections.

Scrotal mass with fever and generalized lymphadenopathy in a young man secondary to Chlamydia trachomatis infection

2005 ◽  
Vol 51 (3) ◽  
pp. e107-e108 ◽  
Author(s):  
N.E. Jenkins ◽  
L. Radcliffe ◽  
Mike Beadsworth ◽  
H. Mallinson ◽  
Fred Nye ◽  
...  
Keyword(s):  
Chlamydia Trachomatis ◽  
Scrotal Mass ◽  
Chlamydia Trachomatis Infection ◽  
Generalized Lymphadenopathy
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