scholarly journals Paradoxical response to propofol and electroencephalogram (EEG) findings leads to early diagnosis of drug induced nonconvulsive status epilepticus: a case report

2019 ◽  
Vol 26 (1) ◽  
pp. 37-38
Author(s):  
Mariko Aizawa ◽  
Satoshi Ishihara ◽  
Ryo Nishisako ◽  
Takako Akimoto ◽  
Ryousuke Kamimura ◽  
...  
Keyword(s):  
Case Report ◽  
Status Epilepticus ◽  
Early Diagnosis ◽  
Nonconvulsive Status Epilepticus ◽  
Drug Induced ◽  
Paradoxical Response ◽  
Electroencephalogram Eeg

scholarly journals Catatonia, beyond a psychiatric syndrome

2017 ◽  
Vol 11 (2) ◽  
pp. 209-212
Author(s):  
Nathália Stela Visoná de Figueiredo ◽  
Débora Bartzen Moraes Angst ◽  
Antônio de Matos Lima Neto ◽  
Michel Ferreira Machado ◽  
Maria Sheila Guimarães Rocha ◽  
...  
Keyword(s):  
Case Report ◽  
Status Epilepticus ◽  
Clinical Manifestation ◽  
Sinus Thrombosis ◽  
Cerebral Venous Sinus Thrombosis ◽  
Venous Sinus ◽  
Nonconvulsive Status Epilepticus ◽  
Diagnostic Investigation ◽  
Venous Sinus Thrombosis ◽  
Psychiatric Syndrome

ABSTRACT Although catatonia is a well-known psychiatric syndrome, there are many possible systemic and neurological etiologies. The aim of this case report was to present a case of a patient with cerebral venous sinus thrombosis and infarction in which catatonia was the clinical manifestation of a possible nonconvulsive status epilepticus. To our knowledge, only one such case has been reported in the literature, which had a simplified diagnostic investigation. It is important to correctly recognize the organic cause underlying catatonia in order to treat the patient as soon as possible thereby improving outcome. Therefore, physicians need to update their knowledge on catatonia, recognizing that it can be part of a psychiatric or neurologic condition.

Nonconvulsive Status Epilepticus on Electroencephalography in a Case With Subacute Sclerosing Panencephalitis

2006 ◽  
Vol 21 (3) ◽  
pp. 256-260
Author(s):  
Ömer Faruk Aydin ◽  
Nesrin Şenbil ◽  
Y. K. Yavuz Gürer
Keyword(s):  
Status Epilepticus ◽  
Subacute Sclerosing Panencephalitis ◽  
Closed Head Injury ◽  
High Amplitude ◽  
Nonconvulsive Status Epilepticus ◽  
Computed Tomographic ◽  
Cognitive Difficulties ◽  
Intravenous Diazepam ◽  
Electroencephalogram Eeg ◽  
The Brain

Subacute sclerosing panencephalitis is a neurodegenerative disease with a poor prognosis. We report a case of a 5Z\x-year-old boy who had emotional lability, cognitive difficulties, and myoclonia after a mild closed head injury. The magnetic resonance image of the brain and computed tomographic scan of the head were normal. His electroencephalogram (EEG) showed continuous nonconvulsive status epilepticus activity, which could not be suppressed with intravenous diazepam. After treatment with phenytoin for 2 days, an EEG showed periodic high-amplitude sharp-and-slow-wave complexes, which were also not suppressed with intravenous diazepam. Since the patient had measles at 5 months of age, subacute sclerosing panencephalitis was considered, and the diagnosis was confirmed by the presence of measles antibodies in cerebrospinal fluid. ( J Child Neurol 2006;21:256—260; DOI 10.2310/7010.2006.00056).

Pediatric Metronidazole-Induced Encephalopathy: A Case Report and Review of the Literature

2020 ◽  
Author(s):  
Alberto M. Cappellari ◽  
Donata Rossetti ◽  
Sabrina Avignone ◽  
Elisa Scola ◽  
Antonio Di Cesare
Keyword(s):  
Case Report ◽  
Status Epilepticus ◽  
Antimicrobial Drug ◽  
Adult Patients ◽  
Nonconvulsive Status Epilepticus ◽  
Toxic Encephalopathy ◽  
Review Of The Literature ◽  
The Common

AbstractMetronidazole-induced encephalopathy is a rare toxic encephalopathy secondary to the common use to this antimicrobial drug. It has been reported mainly in adult patients but only rarely in children. Owing to possible devastating complication of this disease, clinicians should have a higher index of suspicion for encephalopathic patients on metronidazole therapy. Here, we report a 5-month-old infant with metronidazole-induced encephalopathy presenting with nonconvulsive status epilepticus. A review of the literature in pediatric, as well as adult metronidazole-induced encephalopathy, is also provided.

scholarly journals Topiramate overdose: A case report of a patient with extremely high topiramate serum concentrations and nonconvulsive status epilepticus

Epilepsia ◽  
2009 ◽  
Vol 51 (6) ◽  
pp. 1090-1093 ◽  
Author(s):  
Christian Brandt ◽  
Heike Elsner ◽  
Nora Füratsch ◽  
Matthias Hoppe ◽  
Esther Nieder ◽  
...  
Keyword(s):  
Case Report ◽  
Status Epilepticus ◽  
Nonconvulsive Status Epilepticus ◽  
Serum Concentrations

scholarly journals Cefepime-Induced Encephalopathy and Nonconvulsive Status Epilepticus: Dispelling an Artificial Dichotomy

2018 ◽  
Vol 9 (2) ◽  
pp. 100-104 ◽  
Author(s):  
Dmitry Tchapyjnikov ◽  
Matthew W. Luedke
Keyword(s):  
Status Epilepticus ◽  
Mental Status ◽  
Case Series ◽  
Generation Cephalosporin ◽  
Nonconvulsive Status Epilepticus ◽  
Fourth Generation ◽  
Definitive Treatment ◽  
Transient Improvement ◽  
Cephalosporin Antibiotic ◽  
Electroencephalogram Eeg

Cefepime is a fourth-generation cephalosporin antibiotic known to have neurotoxic side effects. Recent reports have described patients on cefepime presenting with altered mentation and concurrent triphasic wave discharges on electroencephalogram (EEG). Some have described this clinical presentation as cefepime-induced encephalopathy, while others have termed it as cefepime-induced nonconvulsive status epilepticus (NCSE). We report on 4 patients who developed cefepime-associated altered mentation with triphasic discharges on EEG. A benzodiazepine trial was attempted in 3 of the patients, all of whom had improvement in the frequency of the triphasic discharges, but only 2 of whom demonstrated a concurrent partial and transient improvement in mental status. All 4 patients had normalization of mental status upon discontinuation of cefepime. We provide a literature review of prior cases and propose that these reports, including those labeled as NCSE, are best described as a cefepime-induced encephalopathy with triphasic discharges as opposed to an ictal phenomenon. We contend that aggressive treatment with anti-seizure medications is not warranted and that cefepime discontinuation is the definitive treatment. This case series and review of the literature clarifies a long-standing terminological ambiguity in a unique clinical picture that can be encountered by the neurohospitalist or other providers.

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