scholarly journals Potential role of the OPG/RANK/RANKL axis in prostate cancer invasion and bone metastasis

2014 ◽  
Vol 32 (6) ◽  
pp. 2605-2611 ◽  
Author(s):  
XIAOMING LI ◽  
YAOMING LIU ◽  
BIN WU ◽  
ZHILONG DONG ◽  
YICHEN WANG ◽  
...  
Keyword(s):  
Prostate Cancer ◽  
Bone Metastasis ◽  
Potential Role ◽  
Cancer Invasion

Phosphoproteome analysis demonstrates the potential role of THRAP3 phosphorylation in androgen-independent prostate cancer cell growth

PROTEOMICS ◽  
2016 ◽  
Vol 16 (7) ◽  
pp. 1069-1078 ◽  
Author(s):  
Yoko Ino ◽  
Noriaki Arakawa ◽  
Hitoshi Ishiguro ◽  
Hiroji Uemura ◽  
Yoshinobu Kubota ◽  
...  
Keyword(s):  
Prostate Cancer ◽  
Cell Growth ◽  
Cancer Cell ◽  
Potential Role ◽  
Prostate Cancer Cell ◽  
Cancer Cell Growth ◽  
Androgen Independent

scholarly journals Role of gangliosides in prostate pathology

2016 ◽  
Vol 19 (1) ◽  
pp. 10-15
Author(s):  
Ilinca Nicolae ◽  
◽  
Corina-Daniela Ene ◽  
Simona Roxana Georgescu ◽  
◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Prostate Cancer ◽  
Differential Diagnosis ◽  
Benign Prostatic Hyperplasia ◽  
Molecular Pathology ◽  
Potential Role ◽  
Prostatic Hyperplasia ◽  
Immunologic Markers ◽  
Immunological Interactions

Molecular pathology of benign prostatic hyperplasia is multifactorial and involves endocrine, biochemical, immunological interactions. The mechanisms involved in the onset and progression of benign prostatic hyperplasia are: infections, 50 years of age, hormones and neurotransmitters imbalances, inflammation, oxidative stress. The potential role of glycosylation in the pathogenesis of prostate disease has been neglected. In this study we documented the profile of gangliosides in normal and pathological prostatic tissues together with the pathologic changes seen in the level of extracellular gangliosides in patients with prostate pathology. Analysis of the data in the literature suggests that gangliosides may represent immunologic markers useful in the differential diagnosis between prostate cancer and benign prostatic hyperplasia.

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