scholarly journals Disseminated nocardiosis in a patient with nephrotic syndrome following HIV infection

2014 ◽  
Vol 8 (4) ◽  
pp. 1142-1144 ◽  
Author(s):  
LETIAN ZHOU ◽  
HONG LIU ◽  
FANG YUAN ◽  
SHUGUANG YUAN
Keyword(s):  
Nephrotic Syndrome ◽  
Hiv Infection ◽  
Disseminated Nocardiosis

Erratum

1996 ◽  
Vol 17 (12) ◽  
pp. 425-425
Keyword(s):  
Renal Failure ◽  
Nephrotic Syndrome ◽  
Sickle Cell Disease ◽  
Hiv Infection ◽  
Sickle Cell ◽  
Pneumococcal Vaccine ◽  
Csf Leak ◽  
Cell Disease

The callout on page 339 of the October issue of Pediatrics in Review should read: "Pneumococcal vaccine is recommended for children older than 2 years of age who have sickle cell disease, asplenia, nephrotic syndrome or renal failure, immunosuppression, CSF leak, or HIV infection."

scholarly journals Protease inhibitor therapy for HIV infection: the effect on HIV-associated nephrotic syndrome

1999 ◽  
Vol 14 (3) ◽  
pp. 744-747 ◽  
Author(s):  
E. Dellow ◽  
R. Unwin ◽  
R. Miller ◽  
I. Williams ◽  
M. Griffiths
Keyword(s):  
Nephrotic Syndrome ◽  
Hiv Infection ◽  
Protease Inhibitor ◽  
Inhibitor Therapy ◽  
Protease Inhibitor Therapy

scholarly journals Disseminated nocardiosis with psoas abscess in a patient with AIDS: first reported case

2008 ◽  
Vol 50 (2) ◽  
pp. 131-133 ◽  
Author(s):  
Marcelo Corti ◽  
Rubén Solari ◽  
Luis De Carolis ◽  
Diana Cangelos ◽  
Mario Bianchi ◽  
...  
Keyword(s):  
Human Immunodeficiency Virus ◽  
Virus Infection ◽  
Human Immunodeficiency Virus Infection ◽  
Hiv Infection ◽  
Psoas Abscess ◽  
Psoas Muscle ◽  
Sensu Stricto ◽  
Nocardia Asteroides ◽  
Immunodeficiency Virus ◽  
Disseminated Nocardiosis

Psoas muscle abscess is an uncommon infection that have been diagnosed increasingly in the last years. We present a case of a patient with advanced human immunodeficiency virus infection who developed a disseminated infection due to Nocardia asteroides sensu stricto type VI with psoas abscess. To our knowledge no other cases of Nocardia psoas abscess in the setting of HIV infection have been reported in the literature.

HIV-associated nephropathy in Caucasians: case report and review of literature

2008 ◽  
Vol 19 (11) ◽  
pp. 789-790 ◽  
Author(s):  
R Yalavarthy ◽  
M L Smith ◽  
C L Edelstein
Keyword(s):  
Renal Failure ◽  
Nephrotic Syndrome ◽  
Case Report ◽  
African Americans ◽  
Hiv Infection ◽  
Transgenic Mouse ◽  
Genetic Background ◽  
Vital Role ◽  
Review Of Literature ◽  
Caucasian Patients

HIV-associated nephropathy (HIVAN) is almost exclusively seen in African-Americans (AA) and is rare in Caucasians. The mechanisms responsible for the predilection of HIVAN in AA are not well understood. In transgenic mouse studies, genetic background plays a vital role in the development of the HIVAN phenotype. Larger studies in humans have been initiated to study genetic polymorphisms responsible for HIVAN. As our case illustrates, HIVAN should be considered in Caucasian patients with HIV infection complicated by nephrotic syndrome and renal failure.

scholarly journals Tubuloreticular Inclusions in the Absence of Systemic Lupus Erythematosus and HIV Infection: A Report of Three Pediatric Cases

2017 ◽  
Vol 7 (2) ◽  
pp. 91-101 ◽  
Author(s):  
Ayah Elmaghrabi ◽  
Elizabeth Brown ◽  
Ei Khin ◽  
Jared Hassler ◽  
Allen R. Hendricks
Keyword(s):  
Systemic Lupus Erythematosus ◽  
Nephrotic Syndrome ◽  
Hiv Infection ◽  
Renal Biopsy ◽  
Lupus Erythematosus ◽  
Viral Infections ◽  
Case Reports ◽  
Systemic Lupus ◽  
Extensive Evaluation ◽  
Tubuloreticular Inclusions

Tubuloreticular inclusions (TRIs) are subcellular structures located within the cisternae of endoplasmic reticulum. Formation of TRIs has been linked to the exposure of excess interferon (IFN), either from endogenous or exogenous sources. In renal disease, TRIs have been most commonly associated with systemic lupus erythematosus (SLE), and human immunodeficiency virus-associated nephropathy (HIVAN). Case reports of patients with renal biopsies showing TRIs without underlying SLE or HIV are infrequent in adults, and to our knowledge none have been reported in children. We report 3 pediatric cases in which the renal biopsy showed TRIs on electron microscopy without underlying SLE or HIV infection. The first patient presented at 2 years of age with nephrotic syndrome and renal failure. His renal biopsy revealed focal segmental glomerulosclerosis and TRIs. The second patient presented at 6 months of age with infantile nephrotic syndrome, and his renal biopsy revealed membranous glomerulopathy and TRIs. The last patient presented at 4 years of age with acute kidney injury of unclear etiology leading to chronic kidney disease. Her biopsy revealed acute and chronic tubulointerstitial nephritis with TRIs. Despite extensive evaluation in all 3 patients, including testing for HIV infection and SLE, we could not identify an underlying etiology to explain the presence of TRIs. In conclusion, renal biopsy with TRIs in the absence of underling SLE and HIV remains obscure. We propose a possible role for excess IFN triggered by an abnormal immune response to common viral infections in the formation of TRIs and renal injury.

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