scholarly journals Perioperative Management of a Patient with Factor XI Deficiency Undergoing Cardiac Surgery

2018 ◽  
Vol 3 (1) ◽  
Author(s):  
Mungan İbrahim ◽  
Yamanyar Serdar ◽  
Songur Çetin Murat ◽  
Turan Sema
2013 ◽  
Vol 28 (4) ◽  
pp. 618-620 ◽  
Author(s):  
Yohei Yamada ◽  
Kayoko Fujimoto-Ibusuki ◽  
Keiko Morikawa-Kubota

2016 ◽  
Vol 16 (1) ◽  
pp. 143-144
Author(s):  
Yoko Edahiro ◽  
Kunimoto Ichikawa ◽  
Hiroharu Suzuki ◽  
Hajime Yasuda ◽  
Michiaki Koike ◽  
...  

Haemophilia ◽  
2017 ◽  
Vol 23 (6) ◽  
pp. e512-e514 ◽  
Author(s):  
Nathan D. Gay ◽  
Sharl Azar ◽  
Ophira Salomon ◽  
Jason A. Taylor

2017 ◽  
Vol 32 (6) ◽  
pp. 429-436 ◽  
Author(s):  
Mohamed S. Alsammak ◽  
Aneel A. Ashrani ◽  
Jeffrey L. Winters ◽  
Rajiv K. Pruthi

2018 ◽  
pp. bcr-2017-222434
Author(s):  
Margaret L McCarthy ◽  
Sarah M Ordway ◽  
Ryan M Jones ◽  
Jeremy G Perkins

Author(s):  
Legrand Antoine ◽  
Bouquerel Remi ◽  
Strzelecki Antoine ◽  
Gaillard Benjamin Le ◽  
Pelletier Christophe ◽  
...  

2018 ◽  
Vol 33 (2) ◽  
pp. 86-89 ◽  
Author(s):  
Michael G. Fitzsimons ◽  
Rebecca Karp Leaf ◽  
Johnathan Mack ◽  
Pavan K. Bendapudi ◽  
Tao Shen ◽  
...  

1984 ◽  
Vol 51 (03) ◽  
pp. 371-375 ◽  
Author(s):  
Kangathevy Morgan ◽  
Sandra Schiffman ◽  
Donald Feinstein

SummaryTwo patients with hereditary factor XI deficiency developed inhibitors following plasma transfusions. Neither had severe spontaneous bleeding. The patients’ plasmas neutralized both factor XI in plasma, purified factor XI, and purified factor XIa. The inhibitor in both patients’ plasmas adsorbed to Protein A- Sepharose. The inhibitors eluted from Protein A-Sepharose were partially neutralized by kappa and lambda light chain antisera indicating that they were polyclonal IgG antibodies. Both inhibitors markedly decreased adsorption of factor XI to glass surfaces. The cleavage of factor XI by trypsin was unaffected by the inhibitors. The lack of severe spontaneous bleeding in both of these patients strongly suggests that an alternate coagulation mechanism bypassing factor XI must compensate for this severe defect.


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