scholarly journals Chronic inflammatory demyelinating neuropathy

2016 ◽  
Vol 17 (1) ◽  
pp. 16-21 ◽  
Author(s):  
Josef Bednařík
Keyword(s):  
Demyelinating Neuropathy ◽  
Chronic Inflammatory Demyelinating Neuropathy

scholarly journals Hypertrophy of multiple cranial nerves and spinal roots in chronic inflammatory demyelinating neuropathy

1999 ◽  
Vol 67 (5) ◽  
pp. 685-687 ◽  
Author(s):  
J Duarte ◽  
A C. Martinez ◽  
F Rodriguez ◽  
A Mendoza ◽  
A P Sempere ◽  
...  
Keyword(s):  
Cranial Nerves ◽  
Demyelinating Neuropathy ◽  
Chronic Inflammatory Demyelinating Neuropathy ◽  
Spinal Roots

An ambispective cohort study of chronic inflammatory demyelinating neuropathy, and its variants

2021 ◽  
Vol 429 ◽  
pp. 118819
Author(s):  
Mohit Anand ◽  
V.Y. Vishnu ◽  
Mv Padma Srivastava ◽  
Rohit Bhatia ◽  
Mamta Singh ◽  
...  
Keyword(s):  
Cohort Study ◽  
Demyelinating Neuropathy ◽  
Chronic Inflammatory Demyelinating Neuropathy

scholarly journals Heterogeneity of Polyneuropathy Associated with Anti-MAG Antibodies

2015 ◽  
Vol 2015 ◽  
pp. 1-9 ◽  
Author(s):  
Laurent Magy ◽  
Raphaël Kaboré ◽  
Stéphane Mathis ◽  
Prisca Lebeau ◽  
Karima Ghorab ◽  
...  
Keyword(s):  
Peripheral Nerves ◽  
Monoclonal Gammopathy ◽  
Demyelinating Neuropathy ◽  
Laboratory Findings ◽  
Electrodiagnostic Testing ◽  
Immune Mediated ◽  
Chronic Inflammatory Demyelinating Neuropathy ◽  
Myelin Associated Glycoprotein ◽  
Demyelinating Peripheral Neuropathy ◽  
Self Antigen

Polyneuropathy associated with IgM monoclonal gammopathy and anti-myelin associated glycoprotein (MAG) antibodies is an immune-mediated demyelinating neuropathy. The pathophysiology of this condition is likely to involve anti-MAG antibody deposition on myelin sheaths of the peripheral nerves and it is supposed to be distinct from chronic inflammatory demyelinating neuropathy (CIDP), another immune-mediated demyelinating peripheral neuropathy. In this series, we have retrospectively reviewed clinical and laboratory findings from 60 patients with polyneuropathy, IgM gammopathy, and anti-MAG antibodies. We found that the clinical picture in these patients is highly variable suggesting a direct link between the monoclonal gammopathy and the neuropathy. Conversely, one-third of patients had a CIDP-like phenotype on electrodiagnostic testing and this was correlated with a low titer of anti-MAG antibodies and the absence of widening of myelin lamellae. Our data suggest that polyneuropathy associated with anti-MAG antibodies is less homogeneous than previously said and that the pathophysiology of the condition is likely to be heterogeneous as well with the self-antigen being MAG in most of the patients but possibly being another component of myelin in the others.

Anesthesitic management for insulinoma resection in a patient having chronic inflammatory demyelinating neuropathy

2020 ◽  
Vol 49 (4) ◽  
pp. 500-563
Author(s):  
Raham Hasan Mostafa ◽  
Amr Amin Rashwan ◽  
Mohamed Mohamed AboKamar
Keyword(s):  
Female Patient ◽  
Clinical Characteristics ◽  
Chronic Inflammatory Demyelinating Polyneuropathy ◽  
Anesthetic Management ◽  
Young Female ◽  
Demyelinating Polyneuropathy ◽  
Demyelinating Neuropathy ◽  
Chronic Inflammatory Demyelinating Neuropathy ◽  
Inflammatory Demyelinating Polyneuropathy

Chronic Inflammatory Demyelinating Polyneuropathy associated with hypoglycemia 2 to insulinoma is unusual, and to our knowledge, very few patients have been reported in literature. Despite varying presentations in these patients, the clinical characteristics are usually the same. The syndrome usually occurs after several episodes of protracted hypoglycemia. The neuropathy is nearly always symmetrical. We report anesthetic management for a young female patient presenting with CIDP & repeated hypoglycemic episodes during a 2-year period scheduled for insulinoma enucleation.

Sign in / Sign up

Export Citation Format

Share Document