The Applicability of Behavioral Activation Therapy in College Students

2019 ◽  
Vol 1 (8) ◽  
pp. 373-386
Author(s):  
Shen Ziwei ◽  
Xue Ying
Keyword(s):  
College Students ◽  
Behavioral Activation ◽  
Behavioral Activation Therapy ◽  
Activation Therapy

Sudden Gains in Depressed Cancer Patients Treated With Behavioral Activation Therapy

2009 ◽  
Vol 40 (4) ◽  
pp. 346-356 ◽  
Author(s):  
Derek R. Hopko ◽  
Sarah M.C. Robertson ◽  
John P. Carvalho
Keyword(s):  
Cancer Patients ◽  
Behavioral Activation ◽  
Sudden Gains ◽  
Behavioral Activation Therapy ◽  
Activation Therapy

scholarly journals A comprehensive review of behavioral activation treatment for depression

2020 ◽  
Author(s):  
Xiaoxia Wang
Keyword(s):  
Mental Health ◽  
Behavioral Activation ◽  
Behavioral Therapy ◽  
Third Wave ◽  
Self Help ◽  
Effective Components ◽  
System P ◽  
Behavioral Activation Therapy ◽  
Activation Therapy ◽  
Evidenced Based

Behavioral activation (BA) treatment has evolved from a component of cognitive behavioral therapy (CBT) and has become standalone psychotherapy for depression. The rapid increasing application of tele-mental health approaches such as telephone-, internet-, and smartphone-based interventions with BA were emphasized. With its efficacy comparable to traditional CBT, and its evidenced-based cost-effectiveness, BA is promising to be developed into a guided self-help intervention. The efficacy across diagnoses and effective components of BA treatment were reviewed. With the rise of the third wave of psychotherapy, therapeutic components across diagnoses will be incorporated into behavioral activation therapy. However, extensive studies are required to examine the neural and modulatory mechanism of BA for depression, and to explore the feasibility and necessity of tele-mental health BA application into the healthcare system.

scholarly journals Behavioral Activation Therapy on Reward Seeking Behaviors in Depressed People: An Experimental study

2020 ◽  
Vol 9 (4) ◽  
pp. 195-202
Author(s):  
Alireza Karimpour-Vazifehkhorani ◽  
Abbas Bakhshipour Rudsari ◽  
Akram Rezvanizadeh ◽  
Leila Kehtary- Harzang ◽  
Kamyar Hasanzadeh
Keyword(s):  
Positive Reinforcement ◽  
Behavioral Activation ◽  
Intervention Group ◽  
Analysis Of Covariance ◽  
Behavioral Inhibition System ◽  
Reward Seeking ◽  
Behavioral Activation Therapy ◽  
Activation Therapy ◽  
And Control

Introduction: Behavioral activation therapy (BAT) is designed to help individuals’ approach and access sources of positive reinforcement in their life, which can serve a natural antidepressant function and efforts to help depressed people reengage in their life through focused activation strategies. Methods: In this study, 60 individuals were selected and randomly assigned to intervention and control groups. The intervention group received behavioral activation treatment, including eight treatment sessions and 5 weeks later, a follow-up study was conducted. The data were collected, using a Beck Depression Inventory-II and behavioral activation system (BIS)/ behavioral inhibition system (BAS) Carver and White questionnaires, before the intervention and after the intervention and five weeks after the intervention. SPSS 23 and analysis of covariance (ANCOVA) was used for data analysis. Results: Results showed a significant increase in the two components of the BAS including reward seeking and response to reward in the intervention group, which indicates an increase in positive affect and appetitive motivation for reward seeking and decreases the risk of depression. Also, the results showed a significant decrease in the BIS and depression in the intervention group, which indicates a decline in experiencing negative emotions. Conclusion: The implementation of BAT will cause depressed people to try to maximize future rewards and it’s effective in improving the reward seeking and reward response in depressed people because this treatment will increase the positive reinforcement and lead to learning cues that predict possible rewards in environments.

Behavioral activation therapy for depression is associated with a reduction in the concentration of circulating quinolinic acid

2020 ◽  
pp. 1-10
Author(s):  
Jonathan Savitz ◽  
Bart N. Ford ◽  
Hung-Wen Yeh ◽  
Elisabeth Akeman ◽  
Kelly Cosgrove ◽  
...  
Keyword(s):  
Quinolinic Acid ◽  
Behavioral Activation ◽  
Depression Scale ◽  
Kynurenine Pathway ◽  
Serum Samples ◽  
Mixed Effect ◽  
Logistic Regression Models ◽  
Behavioral Activation Therapy ◽  
Activation Therapy ◽  
Post Therapy

Abstract Background An inflammation-induced imbalance in the kynurenine pathway (KP) has been reported in major depressive disorder but the utility of these metabolites as predictive or therapeutic biomarkers of behavioral activation (BA) therapy is unknown. Methods Serum samples were provided by 56 depressed individuals before BA therapy and 29 of these individuals also provided samples after 10 weeks of therapy to measure cytokines and KP metabolites. The PROMIS Depression Scale (PROMIS-D) and the Sheehan Disability Scale were administered weekly and the Beck depression inventory was administered pre- and post-therapy. Data were analyzed with linear mixed-effect, general linear, and logistic regression models. The primary outcome for the biomarker analyses was the ratio of kynurenic acid to quinolinic acid (KynA/QA). Results BA decreased depression and disability scores (p's < 0.001, Cohen's d's > 0.5). KynA/QA significantly increased at post-therapy relative to baseline (p < 0.001, d = 2.2), an effect driven by a decrease in QA post-therapy (p < 0.001, uncorrected, d = 3.39). A trend towards a decrease in the ratio of kynurenine to tryptophan (KYN/TRP) was also observed (p = 0.054, uncorrected, d = 0.78). The change in KynA/QA was nominally associated with the magnitude of change in PROMIS-D scores (p = 0.074, Cohen's f2 = 0.054). Baseline KynA/QA did not predict response to BA therapy. Conclusion The current findings together with previous research show that electronconvulsive therapy, escitalopram, and ketamine decrease concentrations of the neurotoxin, QA, raise the possibility that a common therapeutic mechanism underlies diverse forms of anti-depressant treatment but future controlled studies are needed to test this hypothesis.

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