scholarly journals Biomechanics of Cell Membrane

2020 ◽  
Vol 21 (15) ◽  
pp. 5413
Author(s):  
Stefano Leporatti ◽  
José L. Toca-Herrera
Keyword(s):  
Cell Membrane ◽  
Special Issue ◽  
Adhesive Properties

This Special Issue is focused on measuring and characterizing the mechanical and adhesive properties of cells and membranes [...]

scholarly journals Special Issue on Adhesion, surface preparation and adhesive properties

2020 ◽  
Vol 96 (1-4) ◽  
pp. 1-1 ◽  
Author(s):  
Lucas F. M. da Silva ◽  
Robert D Adams
Keyword(s):  
Surface Preparation ◽  
Special Issue ◽  
Adhesive Properties

Effect of He?Ne laser radiation on adhesive properties of the cell membrane

1993 ◽  
Vol 115 (6) ◽  
pp. 695-697
Author(s):  
T. I. Karu ◽  
L. V. Pyatibrat ◽  
G. S. Kalendo
Keyword(s):  
Laser Radiation ◽  
Cell Membrane ◽  
Adhesive Properties

Special Issue on Adhesion, Surface Preparation and Adhesive Properties

2018 ◽  
Vol 94 (9) ◽  
pp. 667-667
Author(s):  
Lucas F. M. da Silva ◽  
Robert D. Adams
Keyword(s):  
Surface Preparation ◽  
Special Issue ◽  
Adhesive Properties

Dynamic adhesion of eryptotic erythrocytes to immobilized platelets via platelet phosphatidylserine receptors

2014 ◽  
Vol 306 (3) ◽  
pp. C291-C297 ◽  
Author(s):  
Britta Walker ◽  
Syeda T. Towhid ◽  
Evi Schmid ◽  
Sascha M. Hoffmann ◽  
Majed Abed ◽  
...  
Keyword(s):  
Cell Membrane ◽  
Annexin V ◽  
Vascular Occlusion ◽  
Adenosine Diphosphate ◽  
Flow Chamber ◽  
Adhesive Properties ◽  
Glucose Depletion ◽  
Phosphatidylserine Exposure ◽  
Erythrocyte Surface ◽  
Erythrocyte Adhesion

Glucose depletion of erythrocytes triggers suicidal erythrocyte death or eryptosis, which leads to cell membrane scrambling with phosphatidylserine exposure at the cell surface. Eryptotic erythrocytes adhere to endothelial cells by a mechanism involving phosphatidylserine at the erythrocyte surface and CXCL16 as well as CD36 at the endothelial cell membrane. Nothing has hitherto been known about an interaction between eryptotic erythrocytes and platelets, the decisive cells in primary hemostasis and major players in thrombotic vascular occlusion. The present study thus explored whether and how glucose-depleted erythrocytes adhere to platelets. To this end, adhesion of phosphatidylserine-exposing erythrocytes to platelets under flow conditions was examined in a flow chamber model at arterial shear rates. Platelets were immobilized on collagen and further stimulated with adenosine diphosphate (ADP, 10 μM) or thrombin (0.1 U/ml). As a result, a 48-h glucose depletion triggered phosphatidylserine translocation to the erythrocyte surface and augmented the adhesion of erythrocytes to immobilized platelets, an effect significantly increased upon platelet stimulation. Adherence of erythrocytes to platelets was blunted by coating of erythrocytic phosphatidylserine with annexin V or by neutralization of platelet phosphatidylserine receptors CXCL16 and CD36 with respective antibodies. In conclusion, glucose-depleted erythrocytes adhere to platelets. The adhesive properties of platelets are augmented by platelet activation. Erythrocyte adhesion to immobilized platelets requires phosphatidylserine at the erythrocyte surface and CXCL16 as well as CD36 expression on platelets. Thus platelet-mediated erythrocyte adhesion may foster thromboocclusive complications in diseases with stimulated phosphatidylserine exposure of erythrocytes.

scholarly journals Editorial to the Special Issue “Electrophysiology”

2021 ◽  
Vol 22 (6) ◽  
pp. 2956
Author(s):  
Sheng-Nan Wu ◽  
Chin-Wei Huang
Keyword(s):  
Ion Channels ◽  
Cell Membrane ◽  
Special Issue ◽  
Pass Through

Ion channels are well recognized to select ions to pass through the cell membrane in a wide variety of cells [...]

Sarcolemmal permeability changes during early myocardial anoxia

1978 ◽  
Vol 36 (2) ◽  
pp. 642-643
Author(s):  
M. Ashraf ◽  
L. Landa ◽  
L. Nimmo ◽  
C. M. Bloor
Keyword(s):  
Cell Membrane ◽  
Membrane Permeability ◽  
Coronary Artery Occlusion ◽  
Artery Occlusion ◽  
Cell Membrane Permeability ◽  
Enzyme Release ◽  
Sprague Dawley ◽  
Sprague Dawley Rats ◽  
Sarcolemmal Permeability ◽  
Membrane Changes

Following coronary artery occlusion, the myocardial cells lose intracellular enzymes that appear in the serum 3 hrs later. By this time the cells in the ischemic zone have already undergone irreversible changes, and the cell membrane permeability is variably altered in the ischemic cells. At certain stages or intervals the cell membrane changes, allowing release of cytoplasmic enzymes. To correlate the changes in cell membrane permeability with the enzyme release, we used colloidal lanthanum (La+++) as a histological permeability marker in the isolated perfused hearts. The hearts removed from sprague-Dawley rats were perfused with standard Krebs-Henseleit medium gassed with 95% O2 + 5% CO2. The hypoxic medium contained mannitol instead of dextrose and was bubbled with 95% N2 + 5% CO2. The final osmolarity of the medium was 295 M osmol, pH 7. 4.

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