scholarly journals Preventative Taping in Futsal: An Exploratory Analysis of Low-Dye Taping on Planter Force Distribution and Pain Sensitivity

2020 ◽  
Vol 10 (2) ◽  
pp. 540
Author(s):  
Sebastian Klich ◽  
Biye Wang ◽  
Aiguo Chen ◽  
Jun Yan ◽  
Adam Kawczyński

The purpose of the present study was to investigate the changes in plantar foot force distribution (i.e., the percentage of force and force distribution under the rearfoot and forefoot) and plantar pressure pain sensitivity maps in professional futsal players after long-term low-dye taping (LDT). The subjects (n = 25) were male futsal players (age 23.03 ± 1.15 years). During the experiment, a nonelastic tape was applied on the plantar foot surface according to the standards of LDP. The experimental protocol consisted of a 3-day cycle during which the plantar foot force distribution (FFD) and plantar pressure pain threshold (PPT) were measured: (1) before the tape was applied, (2) 24 h after application, and (3) 72 h after application. The results revealed a significant decrease in the force distribution under the rearfoot (p ≤ 0.001) and forefoot (p ≤ 0.001) on the right and left sides. Moreover, the results showed an increase in the plantar pressure pain threshold in all regions of the foot (p ≤ 0.001). The results of this study suggest that plantar fascial taping can be an effective method for normalizing the force distribution on the foot and reducing the plantar pain threshold. The findings provide useful information regarding the prevention of and physical therapy of lower extremity injuries in soccer and futsal.

2012 ◽  
Vol 3 (3) ◽  
pp. 165-169 ◽  
Author(s):  
Christine Mohn ◽  
Olav Vassend ◽  
Stein Knardahl

AbstractBackground and purposeThe psychophysiological responses to and modulation of pressure pain stimulation are relatively new areas of investigation. The aims of the present study were to characterize subjective and cardiovascular (CV) responses to pressure pain stimulation, and to examine the relationship between CV responding and pain pressure pain sensitivity.MethodsThirty-nine pain-free, normotensive women were included in the study and tested during the follicular phase of their menstrual cycles. Pain threshold and tolerance were recorded at the right masseter muscle and the sternum, and visual analogue scales (VAS) were used to rate both pain intensity (the sensory dimension) and discomfort (the affective dimension). Mean arterial pressure (MAP), heart rate (HR), and facial and digital skin blood flux (SBF) were registered continuously.ResultsThe pain threshold and tolerance were significantly higher at the sternum compared with the masseter, but the level of affective distress was higher at the masseter tolerance point. No associations emerged between pressure pain threshold and tolerance stimulation levels, and the corresponding VAS ratings. Pressure pain stimulation of the masseter induced significant increases in MAP, HR, and a decrease in digital SBF. During sternum pressure stimulation a significant change in HR and digital SBF was observed. There were no significant correlations between CV responding and pressure pain sensitivity.ConclusionHealthy women seem to display higher pressure pain sensitivity at the masseter region relative to the sternum. Pressure pain stimulation was associated with significant changes in MAP, HR, and SBF, but was not modulated by CV responses. The validity of these findings is strengthened by our control for menstrual cycle events, weekend-related changes in physiology, and CV changes during pain stimulation.ImplicationsThis study extends previous reports of SBF sensitivity to electrocutaneous pain into the field of pressure stimulation. Moreover, this study suggests that the often demonstrated association between high BP and low pain sensitivity may not apply to pressure pain specifically. Alternatively, this finding adds to the literature of gender differences in the relationship between CV responding and acute pain sensitivity in general.


Author(s):  
Diana Lehmann Urban ◽  
Elizabeth Lehmann ◽  
Leila Motlagh Scholle ◽  
Torsten Kraya

Background: In patients with neuromuscular disorder, only little data of myalgia frequency and characterization exists. To date, only a weak correlation between pain intensity and pressure pain threshold has been found, and it remains enigmatic whether high pain intensity levels are equivalent to high pain sensitivity levels in neuromuscular disorders. Methods: 30 sequential patients with suspected neuromuscular disorder and myalgia were analyzed with regard to myalgia characteristics and clinical findings, including symptoms of depression and anxiety and pain- threshold. Results: A neuromuscular disorder was diagnosed in 14/30 patients. Muscular pain fasciculation syndrome (MPFS) without evidence for myopathy or myositis was diagnosed in 10/30 patients and 6/30 patients were diagnosed with pure myalgia without evidence for a neuromuscular disorder (e.g., myopathy, myositis, MPFS, polymyalgia rheumatica). Highest median pain scores were found in patients with pure myalgia and polymyalgia rheumatica. Pressure pain threshold measurement showed a significant difference between patients and controls in the biceps brachii muscle. Conclusion: Only a weak correlation between pain intensity and pressure pain threshold has been suggested, which is concordant with our results. The hypothesis that high pain intensity levels are equivalent to high pain sensitivity levels was not demonstrated.


2019 ◽  
Vol 129 (2) ◽  
pp. 52-54
Author(s):  
Grzegorz Zieliński ◽  
Michał Baszczowski ◽  
Michał Ginszt ◽  
Aleksandra Byś ◽  
Piotr Gawda

Abstract Introduction. Pathological muscle tone can cause changes in the facial skeleton, including tension headaches, tinnitus, temporomandibular joint dysfunctions. The complexity of pathologies and their spectrum may also indicate changes beyond the facial skeleton. The aim of the study was to determine the possible correlation between the pressure pain threshold of the trapezius muscle and the bioelectrical tension on the masticatory muscles. Aim. The aim of the study was to determine the possible correlation between the pressure pain threshold of the trapezius muscle and the bioelectric tension on the masticatory muscles. Material and methods. The number of 36 women applied for the study. They were all examined using an algometer within a trapezius muscle to determine the pressure pain threshold. The electromyographic study was carried out in accordance with the SENIAM guidelines. The resting activity of selected masticatory muscles (temporal and masseter) was recorded for 10 sec. The study involved an 8-channel BioEMG IIITM surface electromyography apparatus with BioPak Measurement System (BioResearch Associates, Inc. Milwaukee, WI, USA). Statistical analysis was carried out using the r-Pearson test. The level of significance was set at 5%. Results. After comparing the bioelectric tension of the masticatory muscles and the pressure pain threshold, left-sided correlation was observed (p<0.05). Both in the right-hand and general comparison, the results did not reach the required level of statistical significance (p>0.05). Conclusions. The pressure pain threshold seems to be linked to the bioelectrical muscle tone of the masticatory muscles. To confirm this observation, further research into a larger and more diverse group of participants is recommended.


2013 ◽  
Vol 31 (15_suppl) ◽  
pp. 9615-9615
Author(s):  
Norah Lynn Henry ◽  
Steven Harte ◽  
Anna S.C. Conlon ◽  
Kent A. Griffith ◽  
Gabriela Ramirez ◽  
...  

9615 Background: AIAA affect up to half of AI-treated women with early stage breast cancer, and can lead to treatment discontinuation in 20-30%. The etiology is thought to be related to estrogen deprivation although the mechanism is unknown. In premenopausal women, lower estrogen levels have been associated with increased pain. Impaired descending pain inhibitory pathways, which may be a risk factor for developing chronic pain, have also been associated with lower estrogen levels. We prospectively tested whether AI-induced estrogen deprivation alters pain sensitivity, thereby increasing the risk of developing AIAA. Methods: Fifty postmenopausal women with early stage breast cancer initiating AI therapy were enrolled to the study. Subjects underwent experimental pressure pain testing and conditioned pain modulation (CPM) assessment and completed symptom questionnaires prior to AI initiation and after 3 months. Positive CPM values (>0) signify impaired descending pain inhibition. Serum estradiol concentrations were determined using an ultrasensitive assay. T-tests, Fisher’s exact test, and linear regression models were used to assess associations among baseline (BL) experimental pain measures, patient-reported pain, and clinical factors. P values <0.05 were considered statistically significant. Results: All subjects had decreased serum estradiol concentrations with AI therapy. No statistically significant change in pressure pain threshold or CPM with AI therapy was detected. In addition, no association between change in patient-reported pain with AI therapy and change in pain threshold or CPM was identified. Patients demonstrated impaired CPM at baseline (mean 8.0, SD 14.9), and this impairment was greater in patients previously treated with chemotherapy 14.4 vs 2.0, p=0.006), with non-significant trends towards this being more pronounced in those with more severe pain. Conclusions: AI therapy did not impact pressure pain threshold or CPM, suggesting that AIAA is not likely due to pain amplification from estrogen depletion. Studies examining chemotherapy-induced changes in pain processing are needed to better understand how these alterations might contribute to the pain that these patients often develop.


2016 ◽  
Vol 13 (1) ◽  
pp. 114-122 ◽  
Author(s):  
Robert Waller ◽  
Anne Julia Smith ◽  
Peter Bruce O’Sullivan ◽  
Helen Slater ◽  
Michele Sterling ◽  
...  

AbstractBackground and aimsCurrently there is a lack of large population studies that have investigated pain sensitivity distributions in healthy pain free people. The aims of this study were: (1) to provide sex-specific reference values of pressure and cold pain thresholds in young pain-free adults; (2) to examine the association of potential correlates of pain sensitivity with pain threshold values.MethodsThis study investigated sex specific pressure and cold pain threshold estimates for young pain free adults aged 21–24 years. A cross-sectional design was utilised using participants (n =617) from the Western Australian Pregnancy Cohort (Raine) Study at the 22-year follow-up. The association of site, sex, height, weight, smoking, health related quality oflife, psychological measures and activity with pain threshold values was examined. Pressure pain threshold (lumbar spine, tibialis anterior, neck and dorsal wrist) and cold pain threshold (dorsal wrist) were assessed using standardised quantitative sensory testing protocols.ResultsReference values for pressure pain threshold (four body sites) stratified by sex and site, and cold pain threshold (dorsal wrist) stratified by sex are provided. Statistically significant, independent correlates of increased pressure pain sensitivity measures were site (neck, dorsal wrist), sex (female), higher waist-hip ratio and poorer mental health. Statistically significant, independent correlates of increased cold pain sensitivity measures were, sex (female), poorer mental health and smoking.ConclusionsThese data provide the most comprehensive and robust sex specific reference values for pressure pain threshold specific to four body sites and cold pain threshold at the dorsal wrist for young adults aged 21–24 years. Establishing normative values in this young age group is important given that the transition from adolescence to adulthood is a critical temporal period during which trajectories for persistent pain can be established.ImplicationsThese data will provide an important research resource to enable more accurate profiling and interpretation of pain sensitivity in clinical pain disorders in young adults. The robust and comprehensive data can assist interpretation of future clinical pain studies and provide further insight into the complex associations of pain sensitivity that can be used in future research.


2018 ◽  
Vol 12 (4) ◽  
pp. 28-38
Author(s):  
Iwona Wilk ◽  
Tomasz Matuszewski ◽  
Magdalena Tarkowska ◽  
Krzysztof Kassolik ◽  
Waldemar Andrzejewski ◽  
...  

Background: The pressure pain threshold (PPT) differs by muscle, depends on sex, age, and health status, and alters under the influence of disease. It is interesting to ask how and whether tissue sensitivity changes after the application of therapy. Aim of the study: The purpose of this study was to assess in healthy individuals the pressure sensitivity of muscles in the torso following a classic massage. Material and methods: A measurement of the pressure sensitivity of certain muscles of the back (the latissimus dorsi, the trapezius, and the spinal erector) was performed using an algometer before classic back massage and immediately after the procedure. A classic back massage was given to 60 healthy volunteers aged 20–47 years. The massage lasted 20 minutes and the major techniques were performed on selected superficial and deep muscles of the back. Results: The pressure sensitivity of the muscles decreased after the classic massage. Statistically significant changes were observed in the right (p < 0.001) and left (p = 0.002) spinal erectors; in the transverse part on both sides (p < 0.001), and in the descending part on both sides (p < 0.001) of the trapezius muscle; and in the right (p = 0.008) and left (p = 0.004) of the latissimus dorsi muscle. Conclusions: Classic massage alleviates tissue sensitivity to pressure in healthy individuals. It can be effective in cases of increased resting muscle tone accompanied by increased tissue sensitivity.


2018 ◽  
Vol 11 (1) ◽  
pp. 41-51 ◽  
Author(s):  
Hanan El-Tumi ◽  
Mark I. Johnson ◽  
Osama A. Tashani

Background: Ageing is associated with alterations of the structure and function of somatosensory tissue that can impact on pain perception. The aim of this study was to investigate the relationship between age and pain sensitivity responses to noxious thermal and mechanical stimuli in healthy adults. Methods: 56 unpaid volunteers (28 women) aged between 20 and 55 years were categorised according to age into one of seven possible groups. The following measurements were taken: thermal detection thresholds, heat pain threshold and tolerance using a TSA-II NeuroSensory Analyzer; pressure pain threshold using a handheld electronic pressure algometer; and cold pressor pain threshold, tolerance, intensity and unpleasantness. Results: There was a positive correlation between heat pain tolerance and age (r = 0.228, P = 0.046), but no statistically significant differences between age groups for cold or warm detection thresholds, or heat pain threshold or tolerance. Forward regression found increasing age to be a predictor of increased pressure pain threshold (B = 0.378, P = 0.002), and sex/gender to be a predictor of cold pressor pain tolerance, with women having lower tolerance than men (B = -0.332, P = 0.006). Conclusion: The findings of this experimental study provide further evidence that pressure pain threshold increases with age and that women have lower thresholds and tolerances to innocuous and noxious thermal stimuli. Significance: The findings demonstrate that variations in pain sensitivity response to experimental stimuli in adults vary according to stimulus modality, age and sex and gender.


Cephalalgia ◽  
2007 ◽  
Vol 27 (2) ◽  
pp. 145-152 ◽  
Author(s):  
L Buchgreitz ◽  
AC Lyngberg ◽  
L Bendtsen ◽  
R Jensen

We recently reported an increase in prevalence and frequency of tension-type headache (TTH) over a 12-year period in the young Danish population. The aim of the present study was to analyse whether this increase was related to increased pain sensitivity. The study was a cross-sectional replicate of a large Danish population study. It compared 113 subjects aged 25-36 years in 2001, with 221 comparable subjects in 1989. Tenderness was considerably higher in 2001 than in 1989. When stratified according to presence of headache, the increase in tenderness was clinically and statistically significant only in women with frequent TTH. The pressure pain threshold was significantly lower in 2001 compared with 1989 in women with frequent TTH. The increase in tenderness in the population may predict an even higher prevalence of TTH in future. The changes support the hypothesis of central sensitization in TTH.


2021 ◽  
Vol 29 (1) ◽  
Author(s):  
Casper Glissmann Nim ◽  
Kenneth Arnold Weber ◽  
Gregory Neill Kawchuk ◽  
Søren O’Neill

Abstract Background Pain hypersensitivity can be assessed using Quantitative Sensory Testing (QST) and is associated with persistent low back pain. Spinal manipulation appears to modify pain hypersensitivity, and this could function as one mechanism leading to clinical improvements. In the current study, we applied a comprehensive QST battery to assess pain sensitivity in a cohort of low back pain patients before and after spinal manipulation to improve our understanding of the association between QST and clinical improvements. This study addresses two questions: Are clinical improvements following spinal manipulation in low back pain patients contingent on pain hypersensitivity, and does pain sensitivity change following spinal manipulation? Methods We performed a secondary analysis of data from a randomized clinical trial. One hundred and thirty-two participants with persistent LBP were treated with spinal manipulation four times over two weeks. Patient-reported outcomes and QST were assessed at baseline, after the fourth spinal manipulation session, and 14-days later. The clinical outcomes were changes in low back pain intensity and disability. Using latent profile analysis, we categorized the participants into clusters depending on their baseline QST scores. We used linear mixed models to examine the association between clusters and changes in patient-reported outcomes and QST. Results Two clusters emerged: a Sensitized and a Not sensitized. The former had significantly lower regional pressure and thermal pain thresholds, remote pressure pain tolerance, and lower inhibitory conditioned pain modulation than the Not sensitized group. However, we only found between-cluster differences for regional pressure pain threshold following spinal manipulation. Thus, the clusters were not associated with patient-reported pain and disability changes or the remaining QST outcomes. Conclusions We report that the baseline QST profile was not associated with clinical improvements following spinal manipulation. We did observe a substantial change for regional pressure pain threshold, which suggests that any effect of spinal manipulation on pain sensitivity is most likely to be observed as changes in regional, mechanical pain threshold. However, the mechanism that invokes clinical improvement and pain sensitivity changes appear distinct. Due to methodological caveats, we advise caution when interpreting the results. Trial registration Clinical.Trial.gov identifier: NCT04086667, registered 11 September 2019 – Retrospectively registered, https://clinicaltrials.gov/ct2/show/NCT04086667


2005 ◽  
Vol 6 (3) ◽  
pp. 22-29 ◽  
Author(s):  
Youssef S. Abou-Atme ◽  
Marcello Melis ◽  
Khalid H. Zawawi

Abstract Objectives The aim of this experiment was to detect pressure pain threshold (PPT) differences on intra-oral palpation of the lateral pterygoid muscle (LPM) between subjects diagnosed with temporomandibular disorders (TMD) and controls. Methods Thirty-one consecutive female TMD patients and 31 age and gender matched controls underwent palpation of the LPM using an algometer made with a queue-tip connected to a digital scale, and PPT was measured. Results Mean PPTs of the right and left LPM of the controls were respectively 191g (49KPa) and 200g (51KPa), and mean PPTs of the right and left LPM of TMD patients were respectively 245g (62KPa) and 256g (63KPa). ANOVA between the four PPT measurements showed significant difference only between the PPT readings of the right LPM of the controls and the left LPM of the patients (p<0.05). Conclusions The findings of this study suggest that PPT measured by means of the described algometer is not decreased in TMD patients as compared to control subjects. Citation Abou-Atme YS, Melis M, Zawawi KH. Pressure Pain Threshold of the Lateral Pterygoid Muscles. J Contemp Dent Pract 2005 August;(6)3:022-029.


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