scholarly journals Frost Survival Mechanism of Vegetative Buds in Temperate Trees: Deep Supercooling and Extraorgan Freezing vs. Ice Tolerance

2019 ◽  
Vol 10 ◽  
Author(s):  
Gilbert Neuner ◽  
Kristina Monitzer ◽  
Dominik Kaplenig ◽  
Julia Ingruber
Keyword(s):  
Survival Mechanism ◽  
Temperate Trees ◽  
Vegetative Buds ◽  
Deep Supercooling

Deep supercooling of water and preservation of cells at deep supercooled temperatures

Cryobiology ◽  
2020 ◽  
Vol 97 ◽  
pp. 255
Author(s):  
Osman Berk Usta ◽  
Haishui Huang ◽  
Camilo Rey-Bedon ◽  
Martin Yarmush
Keyword(s):  
Deep Supercooling

scholarly journals Molecular Dynamics Simulations of Crystal Nucleation near Interfaces in Incompatible Polymer Blends

Polymers ◽  
2021 ◽  
Vol 13 (3) ◽  
pp. 347
Author(s):  
Wenlin Zhang ◽  
Lingyi Zou
Keyword(s):  
Molecular Dynamics ◽  
Polymer Blends ◽  
Md Simulations ◽  
Nucleation And Growth ◽  
Crystal Nucleation ◽  
Crystalline Order ◽  
Mobile Species ◽  
Deep Supercooling ◽  
Crystallizable Polymer ◽  
Dynamics Simulations

We apply molecular dynamics (MD) simulations to investigate crystal nucleation in incompatible polymer blends under deep supercooling conditions. Simulations of isothermal nucleation are performed for phase-separated blends with different degrees of incompatibility. In weakly segregated blends, slow and incompatible chains in crystallizable polymer domains can significantly hinder the crystal nucleation and growth. When a crystallizable polymer is blended with a more mobile species in interfacial regions, enhanced molecular mobility leads to the fast growth of crystalline order. However, the incubation time remains the same as that in pure samples. By inducing anisotropic alignment near the interfaces of strongly segregated blends, phase separation also promotes crystalline order to grow near interfaces between different polymer domains.

Role of autophagy as trophoblast survival mechanism in preeclampsia

Placenta ◽  
2017 ◽  
Vol 57 ◽  
pp. 278-279
Author(s):  
Martina Hutabarat ◽  
Noroyono Wibowo ◽  
Berthold Huppertz
Keyword(s):  
Survival Mechanism

scholarly journals A Sacrifice-for-Survival Mechanism Protects Root Stem Cell Niche from Chilling Stress

Cell ◽  
2017 ◽  
Vol 170 (1) ◽  
pp. 102-113.e14 ◽  
Author(s):  
Jing Han Hong ◽  
Maria Savina ◽  
Jing Du ◽  
Ajay Devendran ◽  
Karthikbabu Kannivadi Ramakanth ◽  
...  
Keyword(s):  
Stem Cell ◽  
Stem Cell Niche ◽  
Chilling Stress ◽  
Survival Mechanism ◽  
Root Stem Cell ◽  
Cell Niche

Position and Abundance of Pear Thrips Eggs in Sugar Maple Flowering and Vegetative Buds

1995 ◽  
pp. 93-95
Author(s):  
Tracy C. Leskey ◽  
David A. J. Teulon ◽  
E. Alan Cameron
Keyword(s):  
Sugar Maple ◽  
Vegetative Buds

scholarly journals Predicted Effects of Climate Change on Winter Chill Accumulation by Temperate Trees in South Korea

2018 ◽  
Vol 87 (2) ◽  
pp. 166-173 ◽  
Author(s):  
YoSup Park ◽  
ByulHaNa Lee ◽  
Hee-Seung Park
Keyword(s):  
Climate Change ◽  
South Korea ◽  
Winter Chill ◽  
Temperate Trees

Seasonal variations in the properties of ribosome assemblies from vegetative buds of Scots pine

1987 ◽  
Vol 71 (1) ◽  
pp. 55-60 ◽  
Author(s):  
Sirkka Kupila-Ahvenniemi ◽  
Aija Lindfors ◽  
Hely Haggman
Keyword(s):  
Scots Pine ◽  
Seasonal Variations ◽  
Vegetative Buds

scholarly journals Upregulated Autophagy in Calcific Aortic Valve Stenosis Confers Protection of Valvular Interstitial Cells

2019 ◽  
Vol 20 (6) ◽  
pp. 1486 ◽  
Author(s):  
Miguel Carracedo ◽  
Oscar Persson ◽  
Peter Saliba-Gustafsson ◽  
Gonzalo Artiach ◽  
Ewa Ehrenborg ◽  
...  
Keyword(s):  
Aortic Valve ◽  
Aortic Valve Stenosis ◽  
Interstitial Cells ◽  
Left Ventricular ◽  
Autophagic Flux ◽  
Valvular Interstitial Cells ◽  
Survival Mechanism ◽  
Calcified Tissue ◽  
Left Ventricular Outflow

Autophagy serves as a cell survival mechanism which becomes dysregulated under pathological conditions and aging. Aortic valve thickening and calcification causing left ventricular outflow obstruction is known as calcific aortic valve stenosis (CAVS). CAVS is a chronic and progressive disease which increases in incidence and severity with age. Currently, no medical treatment exists for CAVS, and the role of autophagy in the disease remains largely unexplored. To further understand the role of autophagy in the progression of CAVS, we analyzed expression of key autophagy genes in healthy, thickened, and calcified valve tissue from 55 patients, and compared them with nine patients without significant CAVS, undergoing surgery for aortic regurgitation (AR). This revealed a upregulation in autophagy exclusively in the calcified tissue of CAVS patients. This difference in autophagy between CAVS and AR was explored by LC3 lipidation in valvular interstitial cells (VICs), revealing an upregulation in autophagic flux in CAVS patients. Inhibition of autophagy by bafilomycin-A1 led to a decrease in VIC survival. Finally, treatment of VICs with high phosphate led to an increase in autophagic activity. In conclusion, our data suggests that autophagy is upregulated in the calcified tissue of CAVS, serving as a compensatory and pro-survival mechanism.

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