scholarly journals Different Stimulation Frequencies Alter Synchronous Fluctuations in Motor Evoked Potential Amplitude of Intrinsic Hand Muscles—a TMS Study

2016 ◽  
Vol 10 ◽  
Author(s):  
Martin V. Sale ◽  
Nigel C. Rogasch ◽  
Michael A. Nordstrom
Keyword(s):  
Evoked Potential ◽  
Motor Evoked Potential ◽  
Hand Muscles ◽  
Potential Amplitude ◽  
Intrinsic Hand Muscles

scholarly journals Correction to: Preservation of motor maps with increased motor evoked potential amplitude threshold in RMT determination

2018 ◽  
Vol 160 (8) ◽  
pp. 1519-1519
Author(s):  
Giuseppe Lucente ◽  
Steven Lam ◽  
Heike Schneider ◽  
Thomas Picht
Keyword(s):  
Evoked Potential ◽  
Motor Evoked Potential ◽  
Potential Amplitude ◽  
Motor Maps

Proceedings #41: HD-tDCS Effect on Motor Evoked Potential Amplitude Elicited by Neuronavigated TMS

2019 ◽  
Vol 12 (2) ◽  
pp. e110-e112
Author(s):  
Egas Caparelli-Dáquer ◽  
Sidário R. Malheiros-Junior ◽  
Dylan J. Edwards ◽  
Talita Reis ◽  
Claudia F. Machado ◽  
...  
Keyword(s):  
Evoked Potential ◽  
Motor Evoked Potential ◽  
Potential Amplitude

Preservation of motor maps with increased motor evoked potential amplitude threshold in RMT determination

2017 ◽  
Vol 160 (2) ◽  
pp. 325-330 ◽  
Author(s):  
Giuseppe Lucente ◽  
Steven Lam ◽  
Heike Schneider ◽  
Thomas Picht
Keyword(s):  
Evoked Potential ◽  
Motor Evoked Potential ◽  
Potential Amplitude ◽  
Motor Maps

EEG and the Variance of Motor Evoked Potential Amplitude

2006 ◽  
Vol 37 (3) ◽  
pp. 247-251 ◽  
Author(s):  
P. Zarkowski ◽  
C.J. Shin ◽  
T. Dang ◽  
J. Russo ◽  
D. Avery
Keyword(s):  
Evoked Potential ◽  
Motor Evoked Potential ◽  
Potential Amplitude

scholarly journals Posture-Dependent Corticomotor Excitability Differs Between the Transferred Biceps in Individuals With Tetraplegia and the Biceps of Nonimpaired Individuals

2016 ◽  
Vol 31 (4) ◽  
pp. 354-363
Author(s):  
Carrie L. Peterson ◽  
Lynn M. Rogers ◽  
Michael S. Bednar ◽  
Anne M. Bryden ◽  
Michael W. Keith ◽  
...  
Keyword(s):  
Transcranial Magnetic Stimulation ◽  
Magnetic Stimulation ◽  
Evoked Potential ◽  
Muscle Length ◽  
Motor Evoked Potential ◽  
Biomechanical Model ◽  
Single Pulse ◽  
Elbow Extension ◽  
Corticomotor Excitability ◽  
Potential Amplitude

Background. Following biceps transfer to enable elbow extension in individuals with tetraplegia, motor re-education may be facilitated by greater corticomotor excitability. Arm posture modulates corticomotor excitability of the nonimpaired biceps. If arm posture also modulates excitability of the transferred biceps, posture may aid in motor re-education. Objective. Our objective was to determine whether multi-joint arm posture affects corticomotor excitability of the transferred biceps similar to the nonimpaired biceps. We also aimed to determine whether corticomotor excitability of the transferred biceps is related to elbow extension strength and muscle length. Methods. Corticomotor excitability was assessed in 7 arms of individuals with tetraplegia and biceps transfer using transcranial magnetic stimulation and compared to biceps excitability of nonimpaired individuals. Single-pulse transcranial magnetic stimulation was delivered to the motor cortex with the arm in functional postures at rest. Motor-evoked potential amplitude was recorded via surface electromyography. Elbow moment was recorded during maximum isometric extension trials, and muscle length was estimated using a biomechanical model. Results. Arm posture modulated corticomotor excitability of the transferred biceps differently than the nonimpaired biceps. Elbow extension strength was positively related and muscle length was unrelated, respectively, to motor-evoked potential amplitude across the arms with biceps transfer. Conclusions. Corticomotor excitability of the transferred biceps is modulated by arm posture and may contribute to strength outcomes after tendon transfer. Future work should determine whether modulating corticomotor excitability via posture promotes motor re-education during the rehabilitative period following surgery.

scholarly journals Effect of Therapy on Motor Cortical Excitability in Parkinson’s Disease

2008 ◽  
Vol 35 (2) ◽  
pp. 166-172 ◽  
Author(s):  
Aysun Soysal ◽  
Ismail Sobe ◽  
Turan Atay ◽  
Aysu Sen ◽  
Baki Arpaci
Keyword(s):  
Parkinson’S Disease ◽  
Late Stage ◽  
Evoked Potential ◽  
Cortical Excitability ◽  
Early Stage ◽  
Motor Evoked Potential ◽  
Motor Threshold ◽  
Potential Amplitude ◽  
Motor Cortical Excitability ◽  
Motor Cortical

Objective:To assess the impact of the disease stage and therapy on motor cortical excitability in Parkinson’s disease (PD).Methods:Twenty newly diagnosed and medication-free, early stage patients, 20 late stage patients under antiparkinsonian therapy and 20 normal healthy controls were included. Motor threshold (MT), amplitudes of motor evoked potential (MEP), motor evoked potential amplitude/compound muscle action potential amplitude (MEP/CMAP) ratio, central motor conduction time (CMCT) and cortical silent period (CSP) were measured by stimulation of the motor cortex using a 13.5 cm circular coil and recordings from abductor digiti minimi muscle. Following the first study protocol, early stage patients were given therapy and the same protocol was repeated three months later.Results:Motor threshold was lower; and the MEP/CMAP ratio was higher in early and late stage patients than normals. In early stage patients after proper therapy, the MTs became higher than before therapy, but still remained lower than normals. In late stage patients, the CMCTs were shorter than the early stage patients before therapy and normals, but there was no difference between the early stage patients and normals. In early stage patients after therapy, the CMCT became longer than before therapy and this difference was significant in both late stage patients and normals. Although more prominent in late stage patients, the CSP duration in both PD groups was found shorter than normals. In early stage patients, after therapy, the CSP durations became significantly longer compared with before therapy.Conclusion:These findings suggest that the motor cortical excitability increases in PD because of the impairment of the corticomotoneuronal inhibitory system.

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