scholarly journals Autocrine VEGF Isoforms Differentially Regulate Endothelial Cell Behavior

2016 ◽  
Vol 4 ◽  
Author(s):  
Hideki Yamamoto ◽  
Helene Rundqvist ◽  
Cristina Branco ◽  
Randall S. Johnson
Keyword(s):  
Endothelial Cell ◽  
Cell Behavior ◽  
Vegf Isoforms

Endothelial Cell Behavior on Vascular Prosthetic Grafts

ASAIO Journal ◽  
1999 ◽  
Vol 45 (4) ◽  
pp. 272-280 ◽  
Author(s):  
YVES MAROIS ◽  
MARIE-FRANCOISE ◽  
SUGOT-LUIZARD ◽  
ROBERT GUIDOIN
Keyword(s):  
Endothelial Cell ◽  
Cell Behavior

scholarly journals Effects of macro- versus nanoporous silicon substrates on human aortic endothelial cell behavior

2014 ◽  
Vol 9 (1) ◽  
pp. 421 ◽  
Author(s):  
Pilar Formentín ◽  
María Alba ◽  
Úrsula Catalán ◽  
Sara Fernández-Castillejo ◽  
Josep Pallarès ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Cell Behavior ◽  
Silicon Substrates ◽  
Human Aortic Endothelial Cell ◽  
Nanoporous Silicon ◽  
Aortic Endothelial Cell ◽  
Aortic Endothelial

scholarly journals Lysosomal Pathways and Autophagy Distinctively Control Endothelial Cell Behavior to Affect Tumor Vasculature

2019 ◽  
Vol 9 ◽  
Author(s):  
Marco B. Schaaf ◽  
Diede Houbaert ◽  
Odeta Meçe ◽  
San Kit To ◽  
Maarten Ganne ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Tumor Vasculature ◽  
Cell Behavior

Fibrinogen nanofibers for guiding endothelial cell behavior

2013 ◽  
Vol 1 (10) ◽  
pp. 1065 ◽  
Author(s):  
Dencho Gugutkov ◽  
Johan Gustavsson ◽  
Maria Pau Ginebra ◽  
George Altankov
Keyword(s):  
Endothelial Cell ◽  
Cell Behavior

Dirofilaria immitis: heartworm infection alters pulmonary artery endothelial cell behavior

1997 ◽  
Vol 82 (2) ◽  
pp. 389-398 ◽  
Author(s):  
Maria Mupanomunda ◽  
Jeffrey F. Williams ◽  
Charles D. Mackenzie ◽  
Lana Kaiser
Keyword(s):  
Nitric Oxide ◽  
Smooth Muscle ◽  
Endothelial Cell ◽  
Pulmonary Artery ◽  
Dirofilaria Immitis ◽  
Cell Behavior ◽  
A 23187 ◽  
Heartworm Infection ◽  
Endothelium Dependent Relaxation ◽  
Pulmonary Artery Endothelial Cell

Mupanomunda, Maria, Jeffrey F. Williams, Charles D. Mackenzie, and Lana Kaiser. Dirofilaria immitis:heartworm infection alters pulmonary artery endothelial cell behavior. J. Appl. Physiol. 82(2): 389–398, 1997.—The pathogenesis of filariasis has generally been attributed to either physical presence of the adult parasites or the host’s immune response to the parasites. However, the spectrum of filariasis cannot be entirely explained by these causes, and other mechanisms must be operative. It is now evident that factors released by filarial parasites likely contribute to the pathogenesis of filarial diseases. Adult heartworms ( Dirofilaria immitis) reside in the right heart and pulmonary artery, so the pulmonary artery should be exposed to the highest concentration of filarial factors. We tested the hypothesis that endothelium-dependent relaxation is altered in the in vitro pulmonary artery from heartworm-infected dogs. Relaxation responses to endothelium-dependent vasodilators (methacholine, bradykinin, substance P, and A-23187) and the non-endothelium-dependent vasodilator nitroglycerin and contractile responses were measured in rings of pulmonary artery from control and heartworm-infected dogs. Endothelium-dependent relaxation was assessed in the presence and absence of inhibitors of nitric oxide synthase, cyclooxygenase, and guanylate cyclase. Responses to methacholine, substance P, and A-23187, but not to bradykinin, nitroglycerin, norepinephrine, or KCl, were depressed in pulmonary artery from heartworm-infected dogs when compared with control, suggesting that changes in endothelial cell and not vascular smooth muscle behavior are involved in altered relaxation. The mechanism of endothelium-dependent relaxation in control pulmonary artery appears to involve nitric oxide in the case of methacholine and both nitric oxide and a cyclooxygenase product in the case of bradykinin and A-23187. The mechanism of endothelium-dependent relaxation in pulmonary artery from heartworm-infected dogs was not clearly elucidated. These data provide no evidence that heartworm infection globally influences either endothelial cell receptor function or the vascular smooth muscle guanylate cyclase guanosine 3′,5′-cyclic monophosphate system, making it likely that changes in intracellular signaling are primarily responsible for the observed alteration of endothelium-mediated relaxation. Alteration of endothelial cell function by filarial parasites may be an important component in the pathology associated with filariasis.

scholarly journals Complex interactions between the laminin  4 subunit and integrins regulate endothelial cell behavior in vitro and angiogenesis in vivo

2002 ◽  
Vol 99 (25) ◽  
pp. 16075-16080 ◽  
Author(s):  
A. M. Gonzalez ◽  
M. Gonzales ◽  
G. S. Herron ◽  
U. Nagavarapu ◽  
S. B. Hopkinson ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Cell Behavior ◽  
Complex Interactions

Exogenous and endogenous force regulation of endothelial cell behavior

2010 ◽  
Vol 43 (1) ◽  
pp. 79-86 ◽  
Author(s):  
Joseph P. Califano ◽  
Cynthia A. Reinhart-King
Keyword(s):  
Endothelial Cell ◽  
Cell Behavior

Effect of crosslinking chemistry of albumin/heparin multilayers on FGF-2 adsorption and endothelial cell behavior

2017 ◽  
Vol 411 ◽  
pp. 240-250 ◽  
Author(s):  
Marta Kumorek ◽  
Olga Janoušková ◽  
Anita Höcherl ◽  
Milan Houska ◽  
Eliška Mázl-Chánová ◽  
...  
Keyword(s):  
Endothelial Cell ◽  
Cell Behavior

scholarly journals Biological assays and genomic analysis reveal lipoic acid modulation of endothelial cell behavior and gene expression

2006 ◽  
Vol 28 (5) ◽  
pp. 1008-1020 ◽  
Author(s):  
P. Larghero ◽  
R. Vene ◽  
S. Minghelli ◽  
G. Travaini ◽  
M. Morini ◽  
...  
Keyword(s):  
Gene Expression ◽  
Endothelial Cell ◽  
Lipoic Acid ◽  
Genomic Analysis ◽  
Cell Behavior ◽  
Biological Assays
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