scholarly journals Effect of yeast-derived products on systemic innate immune response of broiler chickens following a lipopolysaccharide challenge

2016 ◽  
Vol 95 (10) ◽  
pp. 2266-2273 ◽  
Author(s):  
M. Alizadeh ◽  
J.C. Rodriguez-Lecompte ◽  
A. Yitbarek ◽  
S. Sharif ◽  
G. Crow ◽  
...  
Keyword(s):  
Immune Response ◽  
Innate Immune Response ◽  
Broiler Chickens ◽  
Innate Immune ◽  
Lipopolysaccharide Challenge

scholarly journals Effects of Dietary Maltol on Innate Immunity, Gut Health, and Growth Performance of Broiler Chickens Challenged With Eimeria maxima

2021 ◽  
Vol 8 ◽  
Author(s):  
Inkyung Park ◽  
Doyun Goo ◽  
Hyoyoun Nam ◽  
Samiru S. Wickramasuriya ◽  
Kichoon Lee ◽  
...  
Keyword(s):  
Immune Response ◽  
Innate Immunity ◽  
Growth Performance ◽  
Innate Immune Response ◽  
Broiler Chickens ◽  
Muscle Cells ◽  
Innate Immune ◽  
Gut Health ◽  
Eimeria Maxima

Two studies were conducted to evaluate the effects of maltol as a postbiotic on innate immunity, gut health, and enteric infection. In the first study, an in vitro culture system was used to evaluate the effects of maltol on the innate immune response of chicken macrophage cells (CMC), gut integrity of chicken intestinal epithelial cells (IEC), anti-parasitic activity against Eimeria maxima, and differentiation of quail muscle cells (QMC) and primary chicken embryonic muscle cells (PMC). All cells seeded in the 24-well plates were treated with maltol at concentrations of 0.1, 1.0, and 10.0 μg. CMC and IEC were stimulated by lipopolysaccharide to induce an innate immune response, and QMC and PMC were treated with 0.5 and 2% fetal bovine serum, respectively. After 18 h of incubation, pro-inflammatory cytokines, tight junction proteins (TJPs), and muscle cell growth markers were measured. In the second study, the dietary effect of maltol was evaluated on disease parameters in broiler chickens infected with E. maxima. Eighty male 1-day-old broiler chickens were allocated into the following four treatment groups: (1) Control group without infection, (2) Basal diet with E. maxima, (3) High maltol (HI; 10.0 mg /kg feed) with E. maxima, and (4) Low maltol (LO; 1.0 mg/kg feed) with E. maxima. Body weights (BW) were measured on days 0, 7, 14, 20, and 22. All chickens except the CON group were orally infected with 104E. maxima per chicken on day 14. Jejunum samples were collected for gut lesion scoring, and the gene expression of cytokines and TJPs. Data was analyzed using PROC MIXED in SAS. In vitro, maltol not only increased TJPs in IEC and cytokines in the LPS-stimulated CMC but also showed direct cytotoxicity against sporozoites of E. maxima. In vivo, the HI group improved the BW, reduced the gut lesion scores and fecal oocyst shedding, and decreased jejunal TNFSF15 and IL-1β expression in E. maxima-infected chickens. In conclusion, these results demonstrate the beneficial effects of dietary maltol in the enhancement of growth performance, gut health, and coccidiosis resistance and the applicability of maltol as a postbiotic for the replacement of antibiotic growth promoters in commercial poultry production.

scholarly journals Innate immune response to yeast-derived carbohydrates in broiler chickens fed organic diets and challenged with Clostridium perfringens

2012 ◽  
Vol 91 (5) ◽  
pp. 1105-1112 ◽  
Author(s):  
A. Yitbarek ◽  
H. Echeverry ◽  
J. Brady ◽  
J. Hernandez-Doria ◽  
G. Camelo-Jaimes ◽  
...  
Keyword(s):  
Immune Response ◽  
Clostridium Perfringens ◽  
Innate Immune Response ◽  
Broiler Chickens ◽  
Innate Immune

Nile tilapia fed insect meal: Growth and innate immune response in different times under lipopolysaccharide challenge

2020 ◽  
Author(s):  
Angélica Priscila do Carmo Alves ◽  
Renan Rosa Paulino ◽  
Raquel Tatiane Pereira ◽  
Diego Vicente Costa ◽  
Priscila Vieira Rosa
Keyword(s):  
Immune Response ◽  
Innate Immune Response ◽  
Nile Tilapia ◽  
Innate Immune ◽  
Lipopolysaccharide Challenge

Oral Immune Activation by Disgust and Disease-Related Pictures

2015 ◽  
Vol 29 (3) ◽  
pp. 119-129 ◽  
Author(s):  
Richard J. Stevenson ◽  
Deborah Hodgson ◽  
Megan J. Oaten ◽  
Luba Sominsky ◽  
Mehmet Mahmut ◽  
...  
Keyword(s):  
Immune Response ◽  
Immune Responses ◽  
Innate Immune Response ◽  
Negative Control ◽  
Innate Immune ◽  
Innate Immune Responses ◽  
Immune Markers ◽  
Before And After ◽  
Secondary Analyses ◽  
Image Set

Abstract. Both disgust and disease-related images appear able to induce an innate immune response but it is unclear whether these effects are independent or rely upon a common shared factor (e.g., disgust or disease-related cognitions). In this study we directly compared these two inductions using specifically generated sets of images. One set was disease-related but evoked little disgust, while the other set was disgust evoking but with less disease-relatedness. These two image sets were then compared to a third set, a negative control condition. Using a wholly within-subject design, participants viewed one image set per week, and provided saliva samples, before and after each viewing occasion, which were later analyzed for innate immune markers. We found that both the disease related and disgust images, relative to the negative control images, were not able to generate an innate immune response. However, secondary analyses revealed innate immune responses in participants with greater propensity to feel disgust following exposure to disease-related and disgusting images. These findings suggest that disgust images relatively free of disease-related themes, and disease-related images relatively free of disgust may be suboptimal cues for generating an innate immune response. Not only may this explain why disgust propensity mediates these effects, it may also imply a common pathway.

IL-17C is a mediator of respiratory epithelial innate immune response

Pneumologie ◽  
2013 ◽  
Vol 67 (S 01) ◽  
Author(s):  
P Pfeifer ◽  
M Voss ◽  
B Wonnenberg ◽  
M Bischoff ◽  
F Langer ◽  
...  
Keyword(s):  
Immune Response ◽  
Innate Immune Response ◽  
Innate Immune ◽  
Respiratory Epithelial
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