scholarly journals Partitioning Turkey Blood Plasma and Egg Yolk Lipoproteins Using Agarose Gel

1974 ◽  
Vol 53 (3) ◽  
pp. 1167-1173 ◽  
Author(s):  
Wayne L. Bacon ◽  
Margery A. Musser
Keyword(s):  
Blood Plasma ◽  
Egg Yolk ◽  
Agarose Gel ◽  
Egg Yolk Lipoproteins

scholarly journals The Influence of Crude Cottonseed Oil in the Feed On the Blood and Egg Yolk Lipoproteins of Laying Hens

1977 ◽  
Vol 56 (2) ◽  
pp. 468-479 ◽  
Author(s):  
Robert John Evans ◽  
Cal J. Flegal ◽  
Charles A. Foerder ◽  
Doris H. Bauer ◽  
Michael La Vigne
Keyword(s):  
Laying Hens ◽  
Egg Yolk ◽  
Cottonseed Oil ◽  
Crude Cottonseed Oil ◽  
Egg Yolk Lipoproteins

LIPID EXTRACTION AND DISTRIBUTION STUDIES OF EGG YOLK LIPOPROTEINS

1963 ◽  
Vol 41 (1) ◽  
pp. 657-666 ◽  
Author(s):  
W. G. Martin ◽  
N. H. Tattrie ◽  
W. H. Cook
Keyword(s):  
Fatty Acid ◽  
Neutral Lipids ◽  
Lipid Extraction ◽  
Egg Yolk ◽  
Low Density ◽  
Cholesterol Esters ◽  
Density Fraction ◽  
Distribution Studies ◽  
Egg Yolk Lipoproteins ◽  
Fatty Acid Compositions

The three lipoproteins of egg yolk, α- and β-lipovitellin and the low-density fraction (LDF), have been isolated and their lipid compositions determined. α- and β-lipovitellin comprise 22 to 26% lipid, of which 61% is phospholipid, 35% is triglyceride, and 4% is cholesterol and its esters. LDF contains about 89% lipid having 27% phospholipid, 69% triglyceride, and 4% cholesterol and cholesterol esters. The phospholipids of the three lipoproteins are similar, i.e., 74% lecithins, 18% cephalins, and 8% minor phospholipids. The fatty acid compositions of the neutral lipids, lecithins, and cephalins of the α- and β-lipovitellins were also similar, with only minor differences.Gentle extraction of the LDF solutions with ethyl ether readily removes about 85% of the total lipid and 55% of the phospholipid, while subsequent changes are slow. The lipoprotein residue contains 52% lipid which is mostly phospholipid; when the residual ether is removed, five sedimenting components are observed in the ultracentrifuge.

Contribution of egg yolk lipoproteins to cake structure

1973 ◽  
Vol 24 (1) ◽  
pp. 77-88 ◽  
Author(s):  
V. B. Kamat ◽  
G. A. Lawrence ◽  
C. J. Hart ◽  
R. Yoell
Keyword(s):  
Egg Yolk ◽  
Egg Yolk Lipoproteins

scholarly journals Antioxidative Activity of Egg Yolk Lipoproteins

1994 ◽  
Vol 58 (9) ◽  
pp. 1711-1713 ◽  
Author(s):  
Yukiko Yamamoto ◽  
Miki Omori
Keyword(s):  
Antioxidative Activity ◽  
Egg Yolk ◽  
Egg Yolk Lipoproteins

scholarly journals Slight Disruption in Intestinal Environment by Dextran Sodium Sulfate Reduces Egg Yolk Size Through Disfunction of Ovarian Follicle Growth

2021 ◽  
Vol 11 ◽  
Author(s):  
Takahiro Nii ◽  
Takashi Bungo ◽  
Naoki Isobe ◽  
Yukinori Yoshimura
Keyword(s):  
Gene Expression ◽  
Oral Administration ◽  
Blood Plasma ◽  
Sodium Sulfate ◽  
Egg Production ◽  
Laying Hens ◽  
Ovarian Follicle ◽  
Egg Yolk ◽  
Dextran Sodium Sulfate ◽  
Intestinal Environment

Intestinal environments such as microbiota, mucosal barrier function, and cytokine production affect egg production in laying hens. Dextran sodium sulfate (DSS) is an agent that disrupts the intestinal environment. Previously, we reported that the oral administration of dextran sodium sulfate (DSS: 0.9 g/kg BW) for 5 days caused severe intestinal inflammation in laying hens. However, the DSS concentration in the previous study was much higher to induce a milder disruption of the intestinal environment without heavy symptoms. Thus, the goal of this study was to determine the effects of a lower dose of DSS on the intestinal environment and egg production in laying hens. White Leghorn laying hens (330-day old) were oral administered with or without 0.225 g DSS/kg BW for 28 days (DSS and control group: n = 7 and 8, respectively). Weekly we collected all laid eggs and blood plasma samples. Intestinal tissues, liver, ovarian follicles, and the anterior pituitary gland were collected 1 day after the final treatment. Lower concentrations of orally administered DSS caused (1) a decrease in the ratio of villus height/crypt depth, occludin gene expressions in large intestine and cecal microbiota diversity, (2) a decrease in egg yolk weight, (3) an increase in VLDLy in blood plasma, (4), and enhanced the egg yolk precursor accumulation in the gene expression pattern in the follicular granulosa layer, (5) an increase in FSH and IL-1β gene expression in the pituitary gland, and (6) an increase in concentration of plasma lipopolysaccharide binding protein. These results suggested that the administration of the lower concentration of DSS caused a slight disruption in the intestinal environment. This disruption included poor intestinal morphology and decreased cecal microbiome diversity. The change in the intestinal environment decreases egg yolk size without decreasing the VLDLy supply from the liver. The decrease in egg yolk size is likely to be caused by the dysfunction of egg-yolk precursor uptake in ovarian follicles. In conclusion, the oral administration of a lower dose of DSS is an useful method to cause slight disruptions of intestinal environment, and the intestinal condition decreases egg yolk size through disfunction of ovarian follicle.

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