scholarly journals Norwalk Virus

2020 ◽  
Author(s):  
Keyword(s):  
Norwalk Virus

A Statewide Assessment of the Role of Norwalk Virus in Outbreaks of Food-Borne Gastroenteritis

1985 ◽  
Vol 151 (3) ◽  
pp. 568-568 ◽  
Author(s):  
J. N. Kuritsky ◽  
M. T. Osterholm ◽  
J. A. Korlath ◽  
K. E. White ◽  
J. E. Kaplan
Keyword(s):  
Norwalk Virus ◽  
Food Borne ◽  
Statewide Assessment

scholarly journals Measurement of IgA responses following Norwalk virus infection and other human caliciviruses using a recombinant Norwalk virus protein EIA

1994 ◽  
Vol 113 (1) ◽  
pp. 143-151 ◽  
Author(s):  
S. P. Parker ◽  
W. D. Cubitt
Keyword(s):  
Virus Infection ◽  
Enzyme Immunoassay ◽  
Capsid Protein ◽  
Causative Agent ◽  
Food Poisoning ◽  
Hospital Staff ◽  
Virus Protein ◽  
Norwalk Virus ◽  
Major Outbreak ◽  
Virus Capsid Protein

SUMMARYAn enzyme immunoassay employing recombinant Norwalk virus capsid protein was evaluated for the measurement of IgA responses. Tests on 23 volunteers and patients known to have been infected with Norwalk virus (NV) showed that 19 developed significant IgA responses, 2 had unchanging levels of IgA and 2 failed to respond. There was no evidence of IgA responses to NV following infection with Hawaii or Snow Mountain-like viruses.Tests on sera from patients involved in outbreaks associated with eating contaminated shellfish suggest that some patients may have been infected with more than one strain of calicivirus. The use of the rNV EIA for measuring IgA and IgG responses in patients involved in a major outbreak of food poisoning affecting hospital staff indicated that the causative agent was probably NV.

scholarly journals Seroprevalence of Norwalk Virus and Mexico Virus in Chilean Individuals: Assessment of Independent Risk Factors for Antibody Acquisition

1998 ◽  
Vol 27 (4) ◽  
pp. 789-795 ◽  
Author(s):  
Miguel L. O'Ryan ◽  
Pablo A. Vial ◽  
Nora Mamani ◽  
Xi Jiang ◽  
Mary K. Estes ◽  
...  
Keyword(s):  
Risk Factors ◽  
Norwalk Virus ◽  
Independent Risk Factors

scholarly journals Norwalk Virus Assembly and Stability Monitored by Mass Spectrometry

2010 ◽  
Vol 9 (8) ◽  
pp. 1742-1751 ◽  
Author(s):  
Glen K. Shoemaker ◽  
Esther van Duijn ◽  
Sue E. Crawford ◽  
Charlotte Uetrecht ◽  
Marian Baclayon ◽  
...  
Keyword(s):  
Mass Spectrometry ◽  
Virus Assembly ◽  
Norwalk Virus

Detection of Norwalk virus in stools by enzyme immunoassay

1985 ◽  
Vol 17 (2) ◽  
pp. 127-133 ◽  
Author(s):  
John E. Herrmann ◽  
Nancy A. Nowak ◽  
Neil R. Blacklow
Keyword(s):  
Enzyme Immunoassay ◽  
Norwalk Virus

scholarly journals Identification of Feline Interferon Regulatory Factor 1 as an Efficient Antiviral Factor against the Replication of Feline Calicivirus and Other Feline Viruses

2018 ◽  
Vol 2018 ◽  
pp. 1-10 ◽  
Author(s):  
Yongxiang Liu ◽  
Xiaoxiao Liu ◽  
Hongtao Kang ◽  
Xiaoliang Hu ◽  
Jiasen Liu ◽  
...  
Keyword(s):  
Antiviral Activity ◽  
Viral Infections ◽  
Interferon Regulatory Factor ◽  
Feline Calicivirus ◽  
Type I ◽  
Norwalk Virus ◽  
Regulatory Factor ◽  
Feline Panleukopenia Virus ◽  
Protein Levels ◽  
Interferon Regulatory Factor 1

Interferons (IFNs) can inhibit most, if not all, viral infections by eliciting the transcription of hundreds of interferon-stimulated genes (ISGs). Feline calicivirus (FCV) is a highly contagious pathogen of cats and a surrogate for Norwalk virus. Interferon efficiently inhibits the replication of FCV, but the mechanism of the antiviral activity is poorly understood. Here, we evaluated the anti-FCV activity of ten ISGs, whose antiviral activities were previously reported. The results showed that interferon regulatory factor 1 (IRF1) can significantly inhibit the replication of FCV, whereas the other ISGs tested in this study failed. Further, we found that IRF1 was localized in the nucleus and efficiently activated IFN-β and the ISRE promoter. IRF1 can trigger the production of endogenous interferon and the expression of ISGs, suggesting that IRF1 can positively regulate IFN signalling. Importantly, the mRNA and protein levels of IRF1 were reduced upon FCV infection, which may be a new strategy for FCV to evade the innate immune system. Finally, the antiviral activity of IRF1 against feline panleukopenia virus, feline herpesvirus, and feline infectious peritonitis virus was demonstrated. These data indicate that feline IRF1 plays an important role in regulating the host type I IFN response and inhibiting feline viral infections.

A hospital outbreak due to Norwalk virus

1994 ◽  
Vol 26 (4) ◽  
pp. 261-272 ◽  
Author(s):  
P. Stevenson ◽  
R. McCann ◽  
R. Duthie ◽  
E. Glew ◽  
L. Ganguli
Keyword(s):  
Norwalk Virus ◽  
Hospital Outbreak
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