scholarly journals General Transcription Factor IIF Subunit 1

2020 ◽  
Author(s):  
2017 ◽  
Vol 89 (4) ◽  
pp. 730-745 ◽  
Author(s):  
Elena Babiychuk ◽  
Khai Trinh Hoang ◽  
Klaas Vandepoele ◽  
Eveline Van De Slijke ◽  
Danny Geelen ◽  
...  

BMC Genetics ◽  
2013 ◽  
Vol 14 (1) ◽  
pp. 41 ◽  
Author(s):  
Shinji Sasaki ◽  
Takayuki Ibi ◽  
Toshio Watanabe ◽  
Tamako Matsuhashi ◽  
Shogo Ikeda ◽  
...  

Cell Research ◽  
2003 ◽  
Vol 13 (2) ◽  
pp. 111-120 ◽  
Author(s):  
Wenxiang WEI ◽  
Jun Xia GU ◽  
Cui Qing ZHU ◽  
Feng Yan SUN ◽  
Dorjbal DORJSUREN ◽  
...  

Genetics ◽  
1999 ◽  
Vol 153 (4) ◽  
pp. 1573-1581 ◽  
Author(s):  
Susanna Chou ◽  
Sukalyan Chatterjee ◽  
Mark Lee ◽  
Kevin Struhl

Abstract The general transcription factor IIA (TFIIA) forms a complex with TFIID at the TATA promoter element, and it inhibits the function of several negative regulators of the TATA-binding protein (TBP) subunit of TFIID. Biochemical experiments suggest that TFIIA is important in the response to transcriptional activators because activation domains can interact with TFIIA, increase recruitment of TFIID and TFIIA to the promoter, and promote isomerization of the TFIID-TFIIA-TATA complex. Here, we describe a double-shut-off approach to deplete yeast cells of Toa1, the large subunit of TFIIA, to <1% of the wild-type level. Interestingly, such TFIIA-depleted cells are essentially unaffected for activation by heat shock factor, Ace1, and Gal4-VP16. However, depletion of TFIIA causes a general two- to threefold decrease of transcription from most yeast promoters and a specific cell-cycle arrest at the G2-M boundary. These results indicate that transcriptional activation in vivo can occur in the absence of TFIIA.


1991 ◽  
Vol 266 (31) ◽  
pp. 20940-20945
Author(s):  
M. Gerard ◽  
L. Fischer ◽  
V. Moncollin ◽  
J.M. Chipoulet ◽  
P. Chambon ◽  
...  

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