scholarly journals Celiac Ganglion

2020 ◽  
Author(s):  
Keyword(s):  
Celiac Ganglion

Distribution of peptidergic terminals of enteric origin in the rat celiac ganglion

1989 ◽  
Vol 102 (2-3) ◽  
pp. 121-124 ◽  
Author(s):  
Masayasu Hamaji ◽  
Yoshinori Kawai ◽  
Yasunaru Kawashima ◽  
Masaya Tohyama
Keyword(s):  
Celiac Ganglion

EUS-guided celiac ganglion irradiation with iodine-125 seeds for pain control in pancreatic carcinoma: a prospective pilot study

2012 ◽  
Vol 76 (5) ◽  
pp. 945-952 ◽  
Author(s):  
Kai-Xuan Wang ◽  
Zhen-Dong Jin ◽  
Yi-Qi Du ◽  
Xian-Bao Zhan ◽  
Duo-Wu Zou ◽  
...  
Keyword(s):  
Pilot Study ◽  
Pancreatic Carcinoma ◽  
Pain Control ◽  
Celiac Ganglion ◽  
Prospective Pilot Study ◽  
Iodine 125

Gastric mucosal hyperemia due to acid backdiffusion depends on splanchnic nerve activity

1992 ◽  
Vol 262 (3) ◽  
pp. G505-G509 ◽  
Author(s):  
P. Holzer ◽  
I. T. Lippe
Keyword(s):  
Nicotinic Acetylcholine Receptors ◽  
Mucosal Blood Flow ◽  
Gastric Mucosal Blood Flow ◽  
Hydrogen Gas ◽  
Mucosal Barrier ◽  
Neural Pathways ◽  
Hydrogen Gas Clearance ◽  
Celiac Ganglion ◽  
Arterial Blood ◽  
Splanchnic Nerves

Acid backdiffusion through a disrupted gastric mucosal barrier leads to an increase in gastric mucosal blood flow (MBF). This response involves afferent neurons that pass through the celiac ganglion. The present study examined the neural pathways that underlie the rise in MBF caused by gastric perfusion with 15% ethanol in 0.15 N HCl. MBF was measured by the hydrogen gas clearance technique in urethan-anesthetized rats. Mucosal hyperemia due to acid backdiffusion was not changed by acute bilateral subdiaphragmatic vagotomy but was blocked by acute removal of the celiac-superior mesenteric ganglion complex or acute bilateral transection of the greater splanchnic nerves. Hexamethonium (85 mumol/kg iv) also attenuated the rise in MBF due to acid backdiffusion, whereas guanethidine (0.225 mmol/kg sc) had no effect. None of the procedures and drug treatments altered basal MBF to a significant extent. Transection of the splanchnic nerves, hexamethonium, and guanethidine lowered mean arterial blood pressure, but hypotension as such did not significantly influence the hyperemic response under study. Taken together, the previous and present data indicate that the rise in MBF caused by acid backdiffusion depends on the integrity of afferent and efferent neural pathways that run in the splanchnic nerves and through the celiac ganglion. The efferent pathway involves ganglionic transmission through nicotinic acetylcholine receptors but is independent of noradrenergic neurons.

scholarly journals Pregnancy in rats is modulated by ganglionic cholinergic action

Reproduction ◽  
2006 ◽  
Vol 131 (6) ◽  
pp. 1151-1158 ◽  
Author(s):  
M Casais ◽  
S M Delgado ◽  
Z Sosa ◽  
A M Rastrilla
Keyword(s):  
Ex Vivo ◽  
Integrated System ◽  
Sympathetic System ◽  
Buffer Solution ◽  
Celiac Ganglion ◽  
Ovarian Steroidogenesis ◽  
Superior Ovarian Nerve ◽  
Cholinergic Agents ◽  
Neural Factors

The control of ovarian steroidogenesis during pregnancy is mainly of endocrine origin. At present, there is little information about the influence of neural factors on the gestation physiology. The purpose of this work was to study the action of cholinergic agents in celiac ganglion upon the liberation of progesterone and ovarian androstenedione in the second half of pregnancy in rats. We used the ex vivo celiac ganglion–superior ovarian nerve–ovary integrated system (celiac ganglion–SON–ovary) that was incubated in buffer solution for 180 min, with the celiac ganglion and the ovary located in different compartments and linked by the SON. The results obtained indicate that the control values of ovarian androstenedione vary according to the pregnancy day analyzed. The addition of acetylcholine in ganglion decreased the liberation of both steroids on Day 15 whereas at the end of pregnancy it decreased the liberation of androstenedione without modifying progesterone. Due to the effect observed with atropine and hexametonium, acetylcholine action might occur through unspecific ganglionic pathways (Days 15 and 21) or through muscarinic ganglionic receptors (Days 19 and 20). Thus, we conclude that the cholinergic sympathetic system from the celiac ganglion might be a fine modulator of the pregnancy physiology.

scholarly journals Immunohistochemical analysis of the mouse celiac ganglion: An integrative relay station of the peripheral nervous system

2019 ◽  
Vol 527 (16) ◽  
pp. 2742-2760 ◽  
Author(s):  
Charlotte L. Kaestner ◽  
Elizabeth H. Smith ◽  
Stanley G. Peirce ◽  
Donald B. Hoover
Keyword(s):  
Nervous System ◽  
Peripheral Nervous System ◽  
Immunohistochemical Analysis ◽  
Relay Station ◽  
Celiac Ganglion

scholarly journals Differential Expression of Functionally Identified and Immunohistochemically Identified NK1 Receptors on Sympathetic Neurons

2001 ◽  
Vol 85 (5) ◽  
pp. 1888-1898 ◽  
Author(s):  
Phillip Jobling ◽  
Jennifer P. Messenger ◽  
Ian L. Gibbins
Keyword(s):  
Substance P ◽  
Receptor Antagonist ◽  
Sympathetic Neurons ◽  
Cell Diameter ◽  
Celiac Ganglion ◽  
C Terminus ◽  
Current Voltage ◽  
Transduction Mechanisms ◽  
Inward Currents ◽  
Pkc Inhibitors

We have used multiple-labeling immunohistochemistry, intracellular dye-filling, and intracellular microelectrode recordings to characterize the distribution of tachykinin receptors and substance P boutons on subpopulations of neurons within the guinea pig celiac ganglion. Superfusion of substance P (SP, 1 μM for 1 min) depolarized 42% of tonic neurons and inhibited afterhyperpolarizations in 66% of long afterhyperpolarizing (LAH) neurons without significant desensitization. Twenty-one percent of tonic neurons and 24% of LAH neurons responded to the NK3 agonist senktide but did not respond to SP, indicating SP did not activate NK3 receptors at this concentration. All effects of SP were abolished by the selective NK1 receptor antagonist, SR140333, but not by the selective NK3 receptor antagonist, SR142801, suggesting that exogenous SP activated a receptor with NK1 pharmacology. No dye-filled LAH neuron and only 50% of tonic neurons responding to SP expressed NK1 receptor immunoreactivity (NK1-IR). All neurons responding to SP had SP immunoreactive fibers within one cell diameter, indicating good spatial matching between SP release sites and target neurons. These results indicate that SP may act via a receptor with NK1-like pharmacology that has a C terminus not recognized by antibodies to the intracellular domain of the conventional NK1 receptor. Inward currents evoked by SP acting on this NK1-like receptor or senktide acting through NK3 receptors had identical current-voltage relationships. In LAH neurons, both agonists suppressed I sAHP without reducing I AHP. Responses evoked by SP and senktide were resistant to PKC inhibitors, suggesting that the transduction mechanisms for the NK1-like receptor and the NK3 receptor may be similar.

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