scholarly journals Hypertensive Episode

2020 ◽  
Author(s):  
Keyword(s):  
Hypertensive Episode

Severe Hypertensive Episode Associated With Excess Licorice Consumption

2014 ◽  
Vol 29 (2) ◽  
pp. 158-164 ◽  
Author(s):  
Shelly Rachman-Elbaum ◽  
Teresa Johnson
Keyword(s):  
Hypertensive Episode

scholarly journals Neuropathology of Heart Transplantation

1989 ◽  
Vol 16 (3) ◽  
pp. 291-298 ◽  
Author(s):  
L.C. Ang ◽  
J.M. Gillett ◽  
J.C.E. Kaufmann
Keyword(s):  
Intracranial Hemorrhage ◽  
Systemic Infection ◽  
Clinical Findings ◽  
Cns Lymphoma ◽  
Cardiac Transplant ◽  
Transplant Recipients ◽  
Metabolic Encephalopathy ◽  
Causative Factors ◽  
The Central Nervous System ◽  
Hypertensive Episode

ABSTRACT:The neuropathology of 18 cardiac transplant recipients was reviewed with the clinical findings. Pathological changes were noted in the central nervous system (CNS) in 94% of the patients, the most frequent being cerebral vascular in origin (72%). Eight patients (44%) had multiple cerebral infarcts and morphologically, a large number of these antedated the transplantation. In addition 4 patients had acute focal ischemic changes which occurred after transplantation. Intracranial hemorrhage was noted in 5 patients (28%), including one case of fatal intracerebral hemorrhage following an acute hypertensive episode after the transplantation. While systemic infection was common (10 patients), there were only 5 cases of intracranial infection; including 3 cases of cytomegalovirus infection, one of candidiasis and one of aspergillosis. Post-transplant seizures, occurring in a third of the patients, were related to a variety of causative factors such as sepsis, intracranial hemorrhage, cerebral ischemia, metabolic encephalopathy and cyclosporin neurotoxicity. Of note in this series was the absence of CNS lymphoma or other systemic lymphoproliferative disorder.

scholarly journals A Rare Case of Acute Ocular Hypertensive Episode in a Patient with Chronic Infantile Neurological Cutaneous Articular Syndrome

2016 ◽  
Vol 30 (3) ◽  
pp. 237
Author(s):  
Karen Kar-Wun Chan ◽  
Jonathan Chun-Ho Ho ◽  
Irene Tak-Yee Yeung ◽  
Simon Tak-Chuen Ko
Keyword(s):  
Rare Case ◽  
Hypertensive Episode

Acute hypertensive episode induced by sulpiride - a case report

1989 ◽  
Vol 4 (2) ◽  
pp. 149-150 ◽  
Author(s):  
Robert D. Mayer ◽  
Stuart A. Montgomery
Keyword(s):  
Case Report ◽  
Hypertensive Episode

Mechanism of the acute pressor effect and bradycardia elicited by diaspirin crosslinked hemoglobin in anesthetized rats

1998 ◽  
Vol 76 (4) ◽  
pp. 434-442 ◽  
Author(s):  
Steve Moisan ◽  
Guy Drapeau ◽  
Kenneth E Burhop ◽  
Francis Rioux
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Heart Rate ◽  
Pressor Effect ◽  
Endogenous Nitric Oxide ◽  
Pressor Activity ◽  
Vagal Reflex ◽  
Generating System ◽  
Hypertensive Episode ◽  
Diaspirin Crosslinked Hemoglobin

Diaspirin crosslinked hemoglobin (DCLHb) is a chemically stabilizedhemoglobin (Hb) that induces an increase in blood pressure and a decrease of heart rate wheninjected intravenously in some animals. The mechanism by which DCLHb elicits thesehemodynamic effects was studied in pentobarbital-anesthetized, vagotomized rats using a varietyof drugs known for their inhibitory action towards endogenous hemodynamically active systems.The hypertensive episode elicited by DCLHb (100 or 400 mg·kg–1) was attenuatedin animals pretreated with NG-nitro-L-arginine (inhibitor of nitric oxidesynthases) throughout the 30-min period of observation, but it was not reduced in thosepretreated with a variety of sympatholytic drugs (e.g., prazosin), atropine, BIBP-3226(neuropeptide Y antagonist), indomethacin,[1-(Beta-mercapto-Beta,Beta-cyclopentanemethylene propionic acid), 2-(0-methyl)tyrosine]-Arg8 vasopressin (vasopressin antagonist), losartan (angiotensin antagonist),bosentan (endothelin antagonist), or L-arginine- (nitric oxide precursor), compared withcontrol animals. With the exception of propranolol and BIBP-3226, none of the aforenamedinhibitors reduced the amplitude of the bradycardia associated with the pressor effect of DCLHb.These results suggest that: (i) the acute (<30 min) pressor activity of DCLHb inour animal model requires the presence of an endogenous nitric oxide generating system to beexpressed; (ii) the bradycardia elicited by DCLHb might involve the participation ofneuropeptide Y and (or) its NPY-1 receptors, but it is unlikely to involve abaroreceptor-mediated vagal reflex, at least in our animal model.Key words: hemoglobin, nitric oxide, blood pressure, heart rate,DCLHb.

scholarly journals A Case Report of an Emergency Hypertensive Episode During Trastuzumab Therapy

Cureus ◽  
2021 ◽  
Author(s):  
Adriano Pacheco Mendes ◽  
Jerina Nogueira ◽  
André Mendes ◽  
Joana Cochicho ◽  
Isabel Lavadinho
Keyword(s):  
Case Report ◽  
Trastuzumab Therapy ◽  
Hypertensive Episode

scholarly journals Late onset postpartum eclampsia complicated with posterior reversible encephalopathy syndrome

2020 ◽  
Vol 24 (2) ◽  
pp. 234-236
Author(s):  
Ejaz Khan ◽  
Rovnat Babazade
Keyword(s):  
Acute Kidney Injury ◽  
Posterior Reversible Encephalopathy Syndrome ◽  
Late Onset ◽  
Kidney Injury ◽  
Resonance Imaging ◽  
Unique Case ◽  
Posterior Reversible Encephalopathy ◽  
Reversible Encephalopathy ◽  
Hypertensive Episode ◽  
Visual Disturbances

Posterior reversible encephalopathy syndrome (PRES) is a rare entity characterized by headache, vomiting, visual disturbances, seizure and unconsciousness with characteristic magnetic resonance imaging. Late onset postpartum eclampsia complicated by PRES has been reported in the literature, We report a unique case of 23-year-old patient who developed late onset postpartum eclampsia complicated by PRES and acute kidney injury requiring renal replacement therapy. The case report emphasizes the need to continue antihypertensive medication in hypertensive parturients during postpartum period for at least 6 weeks as frequency of late postpartum eclampsia is high. The medication should be tapered off slowly to avoid reactive hypertensive episode which might trigger PRES and acute kidney injury.

scholarly journals Bilateral Symmetrical Brain Lesions in Tricyclic Antidepressant Overdosage

1980 ◽  
Vol 7 (4) ◽  
pp. 305-307 ◽  
Author(s):  
Laila M. Sabet
Keyword(s):  
Tricyclic Antidepressant ◽  
Brain Lesions ◽  
Review Of The Literature ◽  
Cerebral Lesions ◽  
Antidepressant Overdose ◽  
Tricyclic Antidepressant Overdose ◽  
Cerebral Pathology ◽  
Hypertensive Episode

SUMMARY:The cerebral pathology in a case of tricyclic antidepressant overdose by amitriptyline is described. A review of the literature of similar cases is summarized. The relation of the cerebral lesions to the development of a hypertensive episode possibly induced by amitriptyline is discussed.

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