scholarly journals Pediatric Neurologist

2020 ◽  
Author(s):  
PEDIATRICS ◽  
1987 ◽  
Vol 79 (6) ◽  
pp. 935-938
Author(s):  
Paul J. Hudson ◽  
Richard L. Vogt ◽  
Jack Brondum ◽  
Linden Witherell ◽  
Gary Myers ◽  
...  

Because evidence of mercury exposure was found among workers of a mercury thermometer-manufacturing plant in March 1984, the Vermont Department of Health studied the workers' children for both exposure to mercury and evidence of mercury toxicity. The median urine mercury level of 23 workers' children was 25 µg/L. This was significantly higher than the level (5 µg/L) among 39 children randomly selected from nonworkers' households in the same community (P < .001). Mercury-in-air levels measured in workers' homes were higher than those measured in control homes. A significant correlation was found between the urine mercury levels of the workers' children and the urine mercury levels of their working parents. No child had frank mercury toxicity. No evidence of neurologic toxicity among exposed children was discovered by a pediatric neurologist who examined these and unexposed children without knowledge of their exposure status. This is the first report demonstrating mercury exposure in children of mercury workers. Although toxic effects of mercury were not demonstrated at these levels of exposure, children of mercury workers are at risk for mercury exposure and potential mercury toxicity.


2007 ◽  
Vol 13 (1) ◽  
pp. 17-20
Author(s):  
Raquel Rego ◽  
Paulo Breno Noronha Liberalesso ◽  
Mônica Jaques Spinosa ◽  
Simone Carreiro Vieira ◽  
Alaídes S. Fojo Olmos ◽  
...  

INTRODUCTION: It is currently estimated that more than 10 million children all over the world have epilepsy and the EEG is the most commonly used diagnostic test in the investigation of these patients. The aim of this study was to analyze the sensibility of the EEG in revealing abnormalities in children with the clinical hypothesis of an epileptic seizure. METHODS: Out of 970 EEGs obtained between April 2005 and August 2006 at Pequeno Príncipe Children's Hospital, Curitiba, PR, Brazil, 692 fit the criteria proposed (clinical hypothesis of an epileptic seizure after the evaluation of a pediatric neurologist). All EEGs were recorded digitally, with minimal duration of 20 minutes and electrodes positioned according to the International System 10-20. Neonates were excluded. RESULTS: Age ranged from 30 days to 16.5 years (mean of 6.4 years and median of 4.1 years), 403 were female (58.2%). Out of the 692 EEG included in the study, 281 (40.6%) yielded abnormal results, 96 (34.2%) with abnormalities of the background activity (disorganization and/or asymmetry) and 185 (65.8%) with epileptiform paroxysms. The sensibility of the EEG was 40.6%. Sharpe wave occurred in 77 cases (41.6%), spike in 21 (11.4%), polispike in 14 (7.6%), spike-wave in 17 (9.2%), polispike-wave in 24 (13.0%) and exams with discharges of more than one morphology in 32 (17.3%). CONCLUSION: Our data strengthen the concept that even though the diagnosis of epilepsy is clinical and based on the semiological description of the epileptic crisis, the EEG has a good sensibility when properly indicated.


2016 ◽  
Vol 11 (3) ◽  
pp. 57-61
Author(s):  
E. V. Shishkina ◽  
M. V. Barkhatov ◽  
E. V. Borisova ◽  
S. O. Falalaleeva ◽  
N. A. Shchekaleva ◽  
...  

1992 ◽  
Vol 7 (1_suppl) ◽  
pp. S112-S131 ◽  
Author(s):  
Jan Brismar

The incidence of many autosomal recessive neurometabolic disorders is very high in Saudi Arabia, probably as a result of the frequency of consanguineous marriages. Because our hospital is the main referral center for the entire Kingdom, we examine a large number of patients who have a wide spectrum of neurometabolic disorders. We add our experience and review the world literature. Though a specific diagnosis is radiologically possible in a few disorders, the diagnosis must always be verified biochemically. When the patient is referred from a pediatric neurologist with the diagnosis of neurometabolic disorder, the aim of the neuroradiologist is to determine the amount of brain damage present and to follow the response to given therapy. When the patient is referred with a nonspecific diagnosis, such as delayed development, the aim is to suggest the possibility of a neurometabolic disorder and to initiate further evaluation including possible therapy and genetic counseling. (J Child Neurol 1992;7(Suppl):S112-S131.)


PEDIATRICS ◽  
1963 ◽  
Vol 31 (6) ◽  
pp. 902-902

Elsewhere in this issue of Pediatrics the problem of the physically abused child is considered in papers and in a Commentary. This small pamphlet, a joint meeting at the Annual Forum of the National Conference of Social Welfare, held in New York, May 31, 1962, is a useful source of well-considered information for those who wish a further reference. The authors: an executive of the Massachusetts Society for the Prevention of Cruelty to Children, a pediatric neurologist, a psychiatrist, and a counsel of the Boston Legal Aid Society, know what they are writing about and present what they know with useful simplicity and brevity.


PEDIATRICS ◽  
1972 ◽  
Vol 49 (2) ◽  
pp. 308-309
Author(s):  
Gerald Erenberg

Dr. Ralph Olsen's recent letter to the Editor,1 questioning the validity of prescribing medication for all children with school behavior problems, is more acceptable to me than the opinions stated in the response of Dr. L. Eugene Arnold.2 As a pediatric neurologist, I have first-hand knowledge of the frequency with which this request is made and have had many opportunities to evaluate the results of pharmacotherapy. The initial problem is that of identifying the child whose hyperactive behavior is due to minimal brain dysfunction (by definition an "organic" process).


2020 ◽  
Vol 108 ◽  
pp. 47-53 ◽  
Author(s):  
Mary E. Hartman ◽  
Cydni N. Williams ◽  
Trevor A. Hall ◽  
Christopher C. Bosworth ◽  
Juan A. Piantino

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