Human Truncated Tau Induces Mature Neurofibrillary Pathology in a Mouse Model of Human Tauopathy

2016 ◽  
Vol 54 (2) ◽  
pp. 831-843 ◽  
Author(s):  
Ivana Zimova ◽  
Veronika Brezovakova ◽  
Tomas Hromadka ◽  
Petronela Weisova ◽  
Veronika Cubinkova ◽  
...  
2009 ◽  
Vol 23 (8) ◽  
pp. 2595-2604 ◽  
Author(s):  
Emmanuel Planel ◽  
Alexis Bretteville ◽  
Li Liu ◽  
Laszlo Virag ◽  
Angela L. Du ◽  
...  

2006 ◽  
Vol 168 (5) ◽  
pp. 1598-1607 ◽  
Author(s):  
Tara L. Spires ◽  
Jennifer D. Orne ◽  
Karen SantaCruz ◽  
Rose Pitstick ◽  
George A. Carlson ◽  
...  

Author(s):  
H. D. Geissinge ◽  
L.D. Rhodes

A recently discovered mouse model (‘mdx’) for muscular dystrophy in man may be of considerable interest, since the disease in ‘mdx’ mice is inherited by the same mode of inheritance (X-linked) as the human Duchenne (DMD) muscular dystrophy. Unlike DMD, which results in a situation in which the continual muscle destruction cannot keep up with abortive regenerative attempts of the musculature, and the sufferers of the disease die early, the disease in ‘mdx’ mice appears to be transient, and the mice do not die as a result of it. In fact, it has been reported that the severely damaged Tibialis anterior (TA) muscles of ‘mdx’ mice seem to display exceptionally good regenerative powers at 4-6 weeks, so much so, that these muscles are able to regenerate spontaneously up to their previous levels of physiological activity.


1998 ◽  
Vol 13 (11-s4) ◽  
pp. S178-S184 ◽  
Author(s):  
PETER KONTUREK ◽  
TOMASZ BRZOZOWSKI ◽  
STANISLAW KONTUREK ◽  
ELZBIETA KARCZEWSKA ◽  
ROBERT PAJDO ◽  
...  

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