scholarly journals Heat stress impact on blood biochemical response and plasma aldosterone level in three different indigenous goat breeds

2020 ◽  
Vol 8 (4) ◽  
pp. 266-275
Author(s):  
Joy Aleena ◽  
Veerasamy Sejian ◽  
Govindan Krishnan ◽  
Madiajagan Bagath ◽  
Prathap Pragna ◽  
...  
Keyword(s):  
Heat Stress ◽  
Plasma Aldosterone ◽  
Biochemical Response ◽  
Aldosterone Level ◽  
Blood Biochemical ◽  
Indigenous Goat ◽  
Plasma Aldosterone Level

scholarly journals Changes in plasma aldosterone level after weight loss by bariatric surgery in morbidly obese patients

2020 ◽  
Vol 20 (1) ◽  
Author(s):  
Preaw Suwannasrisuk ◽  
Patchaya Boonchaya-anant ◽  
Natnicha Houngngam ◽  
Suthep Udomsawaengsup ◽  
Sarat Sunthornyothin
Keyword(s):  
Bariatric Surgery ◽  
Weight Loss ◽  
Plasma Aldosterone ◽  
Morbidly Obese ◽  
Obese Patients ◽  
Aldosterone Level ◽  
Plasma Aldosterone Level

scholarly journals Use of piretanide, a new loop diuretic, in cirrhosis with ascites: relationship between the diuretic response and the plasma aldosterone level.

Gut ◽  
1980 ◽  
Vol 21 (10) ◽  
pp. 855-859 ◽  
Author(s):  
V Arroyo ◽  
J Bosch ◽  
R Casamitjana ◽  
J Cabrera ◽  
F Rivera ◽  
...  
Keyword(s):  
Loop Diuretic ◽  
Plasma Aldosterone ◽  
Aldosterone Level ◽  
Diuretic Response ◽  
Plasma Aldosterone Level

INCREASED STEROID METABOLIC CLEARANCE RATE IN ANEPHRIC PATIENTS

1974 ◽  
Vol 75 (4) ◽  
pp. 756-762 ◽  
Author(s):  
P. Corvol ◽  
X. Bertagna ◽  
J. Bedrossian
Keyword(s):  
Cardiac Output ◽  
Clearance Rate ◽  
Plasma Aldosterone ◽  
Normal Male ◽  
Metabolic Clearance ◽  
Metabolic Clearance Rate ◽  
Aldosterone Level ◽  
High Cardiac Output ◽  
Plasma Aldosterone Level ◽  
Anephric Patients

ABSTRACT The control of plasma aldosterone level depends on the secretion rate and on the metabolic clearance rate (MCR) of the hormone. The rate of aldosterone metabolism was measured in 6 anephric patients in comparison with normal volunteers. Unexpected high aldosterone MCR's were found in the anephric patients: 2700 ± 265 1/24 h (M ± sem), as compared to the normals (1600 ± 57). Passive 45° tilting did not significantly change aldosterone MCR's in both the normal and anephric patients. This increase in aldosterone MCR might be explained by a high hepatic plasma flow due to a high cardiac output. The haemodynamic condition of these patients could also explain the high testosterone MCR found in 3 male anephric patients, although testosterone-oestradiol binding globulin was in the high normal male range.

Inhibition of aldosterone turn-off phenomenon following chronic adrenocorticotropin treatment with in vivo administration of antiglucocorticoid and antioxidants in rats

1995 ◽  
Vol 133 (5) ◽  
pp. 578-584 ◽  
Author(s):  
Huiping Ni ◽  
Tomoatsu Mune ◽  
Hiroyuki Morita ◽  
Hisashi Daidoh ◽  
Junko Hanafusa ◽  
...  
Keyword(s):  
Body Weight ◽  
Vitamin E ◽  
Plasma Aldosterone ◽  
Type Ii ◽  
Plasma Acth ◽  
Aldosterone Level ◽  
Aldosterone Production ◽  
Plasma Aldosterone Level ◽  
In Vivo Administration

Ni H, Mune T, Morita H, Daidoh H, Hanafusa J, Shibata T, Yamakita N, Yasuda K. Inhibition of aldosterone turn-off phenomenon following chronic adrenocorticotropin treatment with in vivo administration of antiglucocorticoid and antioxidants in rats. Eur J Endocrinol 1995;133:578–84. ISSN 0804–4643 Chronic adrenocorticotropin (ACTH) treatment in rats leads to a fall in aldosterone secretion (aldosterone turn-off or "aldosterone escape" phenomenon) with a concomitant rise in corticosterone. To elucidate whether ACTH-induced aldosterone suppression is mediated by steroid type II receptor or related to a free-radical effect by over-synthesized corticosterone, we examined the effects of a glucocorticoid antagonist, RU486, and antioxidants dimethyl sulfoxide (DMSO) and vitamin E, on the aldosterone turn-off phenomenon in rats. Each rat received daily for 5 days a different dose of ACTH-Z (5, 10, 20 or 40 μg/100 g body weight) 1 mg RU486/100 g body weight, 100 μl (1.3 mmol) DMSO/100 g body weight or 2 mg vitamin E/100 g body weight with subcutaneous injection. Plasma steroid levels and in vitro release of steroids from the adrenal capsule were measured. The ACTH-Z treatment caused a dose-dependent increase in corticosterone and a decrease in aldosterone in both plasma and adrenal capsule experiments, as well as an increase in adrenal weights. For the following study 5 μg/100 g body weight of ACTH-Z was used. Administration of RU486 alone caused no change in plasma aldosterone level compared to controls, even though the steroid type II receptor was blocked, as evidenced by significant increases in plasma ACTH and corticosterone levels. Concomitant administration of RU486 and ACTH-Z increased both plasma corticosterone and aldosterone levels (p< 0.01) but decreased adrenal capsule corticosterone production (p< 0.05) compared to the rats treated with ACTH-Z alone. Treatment with DMSO alone caused a significant increase in plasma ACTH and corticosterone level (p< 0.05) but no change in plasma aldosterone level or adrenal capsule corticosterone and aldosterone production. The ACTH-induced aldosterone decrease was completely prevented by DMSO administration in both plasma and adrenal capsule experiments (p< 0.01). Vitamin E administration resulted in the elevation of plasma levels of ACTH and corticosterone (p< 0.01 and < 0.05) but not aldosterone, and it also increased adrenal capsule corticosterone production (p< 0.01) but not aldosterone production. By vitamin E administration, the ACTH-induced aldosterone decrease was suppressed almost completely in plasma (p< 0.01) and partially in adrenal capsule experiments (p< 0.01) compared to rats treated with ACTH-Z alone. Our findings suggest that RU486, DMSO and vitamin E inhibit the ACTH-induced aldosterone turn-off phenomenon in plasma, possibly due to the increase in activity of P-450aldo through antioxidant action or a steroid type II receptor blocking action. Keigo Yasuda, Third Department of Internal Medicine, Gifu University School of Medicine, 40 Tsukasa-machi, Gifu MZ500, Japan

scholarly journals Effect of Captopril Rapid Uptitration to Plasma Aldosterone level in Patients with Acute Myocardial Infarction

2016 ◽  
Vol 01 (03) ◽  
pp. 160-168
Author(s):  
Verry Gunawan Sohan ◽  
◽  
Niniek Purwaningtyas ◽  
Ari Natalia Probandari ◽  
◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Plasma Aldosterone ◽  
Aldosterone Level ◽  
Plasma Aldosterone Level

scholarly journals Plasma Aldosterone Level in a Female Case of Pseudohyperaldosteronism (Liddle's Syndrome).

1989 ◽  
Vol 36 (1) ◽  
pp. 167-173 ◽  
Author(s):  
KAZUHISA TAKEUCHI ◽  
KEISHI ABE ◽  
MAKITO SATO ◽  
MINORU YASUJIMA ◽  
KEN OMATA ◽  
...  
Keyword(s):  
Plasma Aldosterone ◽  
Aldosterone Level ◽  
Liddle’S Syndrome ◽  
Female Case ◽  
Liddle's Syndrome ◽  
Plasma Aldosterone Level
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