scholarly journals LEUKOGRAM INTEGRAL INDICATORS IN EVALUATION OF HEALTH PIGLETS’ WITH HYPOPLASTIC ANEMIA

2018 ◽  
pp. 39-42
Author(s):  
G.S. Kiiko ◽  
Keyword(s):  
Hypoplastic Anemia

Transient severe hypoplastic anemia in q fever

Pathology ◽  
1986 ◽  
Vol 18 (2) ◽  
pp. 254-255 ◽  
Author(s):  
Robert Hitchins ◽  
R.G. Cobcroft ◽  
G. Hocker
Keyword(s):  
Q Fever ◽  
Hypoplastic Anemia

Hypoplastic Anemia, Friedreichʼs Ataxia and Chromosomal Breakage

1973 ◽  
Vol 66 (1) ◽  
pp. 135-140 ◽  
Author(s):  
FASIH U. SAMAD ◽  
ERIC ENGEL ◽  
ROBERT C. HARTMANN
Keyword(s):  
Chromosomal Breakage ◽  
Hypoplastic Anemia

scholarly journals Complexities of genetic diagnosis illustrated by an atypical case of congenital hypoplastic anemia

2018 ◽  
Vol 4 (6) ◽  
pp. a003384 ◽  
Author(s):  
David Claassen ◽  
Michelle Boals ◽  
Kevin M. Bowling ◽  
Gregory M. Cooper ◽  
Jennifer Cox ◽  
...  
Keyword(s):  
Genetic Diagnosis ◽  
Atypical Case ◽  
Hypoplastic Anemia

scholarly journals CHRONIC CONGENITAL AREGENERATIVE ANEMIA (PURE RED-CELL ANEMIA) ASSOCIATED WITH ISO-IMMUNIZATION BY THE BLOOD GROUP FACTOR "A"

Blood ◽  
1949 ◽  
Vol 4 (6) ◽  
pp. 697-705 ◽  
Author(s):  
CARL H. SMITH
Keyword(s):  
Bone Marrow ◽  
Blood Group ◽  
Neonatal Period ◽  
Blood Levels ◽  
Fetal Life ◽  
Red Cell ◽  
Serum Titer ◽  
Persistent Depression ◽  
Blood Group Factor ◽  
Hypoplastic Anemia

Abstract Chronic congenital aregenerative anemia describes a "pure red-cell" anemia in which the failure of hematopoiesis is restricted entirely to the erythrocytes without simultaneous impairment of leukocyte or platelet production. The separation of this entity from the category of the increasing number of cases designated as hypoplastic anemia will facilitate a more direct examination of the factors involved in its pathogenesis. In the case described in this paper illustrating this condition, the onset of the anemia dated to the newborn period with the clinical and hematologic features of a mild type of erythroblastosis fetalis. The mother’s blood group was O, Rh positive and that of the infant and father A, Rh positive. The anti-A serum titer in the mother reached a maximum of 1:128,000. The infant was shown to be a non-secretor. The patient, now 17 months of age, requires repeated transfusions to maintain normal blood levels. The bone marrow reveals a persistent depression of erythropoiesis but the platelet and granulocyte levels are entirely unaffected. It is postulated that prolonged depression in red blood cell production may result from an antibody directed solely against the red cells in fetal life or from the early neonatal period. This concept finds substantiation in other cases of erythroblastosis in which temporary failure of erythropoiesis as confirmed by bone marrow studies is reflected in a state of protracted anemia.

HYPOPLASTIC ANEMIA WITH MULTIPLE CONGENITAL DEFECTS (FANCONI SYNDROME)

PEDIATRICS ◽  
1952 ◽  
Vol 10 (3) ◽  
pp. 286-292
Author(s):  
HANS W. KUNZ
Keyword(s):  
Bone Marrow ◽  
Fanconi Syndrome ◽  
Skin Pigmentation ◽  
Young Age ◽  
Adrenocortical Function ◽  
Congenital Defects ◽  
Hypoplastic Anemia

A case of Fanconi syndrome at an unusually young age presenting anemia, leukopenia, thrombocytopenia, microcephaly, hypogenitalism, skin pigmentation, and anomalies of thumbs, kidneys and heart is described. The patient's younger brother with multiple anomalies of the extremities, hydrocephalus, abnormal ears and genitalia died at the age of 17 months without pigmentation and hematologic symptoms, changes which usually do not occur before the age of 5 years. The literature is reviewed and a follow-up on previously reported cases is given. It seems that in addition to a hypoplasia of the bone marrow an extracorpuscular hemolytic mechanism plays a role in the pathogenesis of the anemia. It is suggested that some manifestations may be due to impaired adrenocortical function.

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