Posterior fossa subdural hematoma due to ruptured arteriovenous malformation

2000 ◽  
Vol 8 (6) ◽  
pp. 1-4 ◽  
Author(s):  
Narendra N. Datta ◽  
Kwong Y. Chan ◽  
John C. K. Kwok ◽  
Christopher Y. F. Poon

Patients with cerebellar arteriovenous malformations (AVM) commonly present to the neurosurgical department after having suffered hemorrhages. The subarachnoid space is the usual location for these often repeating episodes of bleedings. In addition, these patients can present with parenchymal hemorrhage. Acute subdural hematoma caused by a ruptured cerebellar AVM is a rare entity and is not generally recognized. The authors present a case of acute posterior fossa subdural hematoma resulting from a ruptured cerebellar AVM.

1977 ◽  
Vol 47 (1) ◽  
pp. 50-56 ◽  
Author(s):  
Hiroshi Matsumura ◽  
Yasumasa Makita ◽  
Kuniyuki Someda ◽  
Akinori Kondo

✓ We have operated on 12 of 14 cases of arteriovenous malformation (AVM) in the posterior fossa since 1968, with one death. The lesions were in the cerebellum in 10 cases (three anteromedial, one central, three lateral, and three posteromedial), and in the cerebellopontine angle in two; in two cases the lesions were directly related to the brain stem. The AVM's in the anterior part of the cerebellum were operated on through a transtentorial occipital approach.


2019 ◽  
Vol 80 (05) ◽  
pp. 359-364 ◽  
Author(s):  
Stefanie Kaestner ◽  
Marina van den Boom ◽  
Wolfgang Deinsberger

Background In an aging society, traumatic head injuries, such as acute subdural hematomas (aSDHs), are increasingly common because the elderly are prone to falls and are often undergoing anticoagulation treatment. Especially in advanced age, cranial surgery such as craniotomies may put patients in further jeopardy. But if treatment is conservative, a chronic subdural hematoma (cSDH) may develop, requiring surgical evacuation. Existing studies have reported a correlation between several risk factors contributing to the frequency of chronification. To improve the prediction of the course of disease and to aid counseling patients and relatives, this study aimed to determine the frequency and the main risk factors influencing the process of chronification of an aSDH following conservative treatment. Methods We identified patients presenting between January 2012 and September 2017 at our neurosurgical department with an aSDH. All patients treated conservatively were selected retrospectively, and the following parameters were documented: age, sex, chronification status, Glasgow Coma Scale score on admission and discharge, hematoma thickness and density, the degree of midline shift (MLS), prior anticoagulants and administration of procoagulants, thrombosis management, other coagulopathies, initial length of hospital stay, interval between discharge and readmission, and interval between initial injury and date of surgery and last follow-up. The cohort was divided into patients with complete resolution of their aSDH, and patients who needed surgery due to chronification. Results A total of 75 conservatively treated patients with aSDH were included. A chronification was observed in 24 cases (32%). The process of chronification takes an average of 18 days (range: 10–98 days). The following factors were significantly associated with the process of chronification: age (p = 0.001), anticoagulant medication (acetylsalicylic acid [ASA], Coumadin, and novel anticoagulants [NOACs]) before injury (p = 0.026), administration of procoagulants (p = 0.001), presence of other coagulopathies such as thrombocytopenia (p = 0.002), low hematoma density at discharge (p = 0.001), hematoma thickness on admission and discharge (p = 0.001), and the degree of MLS (p = 0.044). Conclusion Chronification occurred in a third of all patients with conservatively treated aSDH, on average within 3 weeks. The probability of developing a cSDH is 0.96 times higher with every yearly increase in age, resulting in 56% chronification in patients ≥ 70 years. Hematoma thickness and impairment of the coagulation system such as anticoagulant medication (ASA, Coumadin, and NOACs) or thrombocytopenia are further risk factors for chronification.


2004 ◽  
Vol 21 (3) ◽  
pp. 234-236 ◽  
Author(s):  
Hirosuke Fujisawa ◽  
Hiroyasu Yonaha ◽  
Katsuki Okumoto ◽  
Hidekatsu Uehara ◽  
Tomotsugu Ie ◽  
...  

2016 ◽  
Vol 26 (2) ◽  
pp. 154-157
Author(s):  
Alfio Spina ◽  
Nicola Boari ◽  
Filippo Gagliardi ◽  
Pietro Mortini

1990 ◽  
Vol 92 (1) ◽  
pp. 57-62 ◽  
Author(s):  
Chr. Raftopoulos ◽  
Ch. Reuse ◽  
C. Chaskis ◽  
J. Brotchi

Cephalalgia ◽  
1991 ◽  
Vol 11 (11_suppl) ◽  
pp. 345-345
Author(s):  
Pereira Monteiro ◽  
M.J. Rosas ◽  
C. Correia ◽  
Arthur Vaz

The authors studied the prevalence of headaches in patients with cerebral arteriovenous malformations followed in the Neurosurgical Department of the Hospital Santo Antonio, Porto, Portugal. The methods used were headache questionnaire and clinic interview. The IHS criteria and classification were used for the diagnostic classification of headaches. From all the patients studied the relative frequency of the different types of headaches was calculated and compared with the general population data. A correlative study of the headache characteristics with the type and localization of the arteriovenous malformation was performed and the results are presented and discussed. Some selected cases of migraine type headaches associated with AVM are presented and the effect of surgery is commented.


2021 ◽  
Vol 4 (1) ◽  
pp. V7
Author(s):  
Brian M. Howard ◽  
Daniel L. Barrow

Many brain arteriovenous malformations (AVMs) derive dural blood supply, while 10%–15% of dural arteriovenous fistulas (dAVFs) have pial arterial input. To differentiate between the two is critical, as treatment of these entities is diametrically opposed. To treat dAVFs, the draining vein(s) is disconnected from feeding arteries, which portends hemorrhagic complications for AVMs. The authors present an operative video of a subtle cerebellar AVM initially treated as a dAVF by attempted embolization through dural vessels. The lesion was subsequently microsurgically extirpated. The authors show a comparison case of an AVM mistaken for a dAVF and transvenous embolization that resulted in a fatal hemorrhage.The video can be found here: https://youtu.be/eDeiMrGoE0Q


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