scholarly journals Analisis asupan karbohidrat dan lemak serta aktivitas fisik terhadap profil lipid darah pada penderita penyakit jantung koroner

2020 ◽  
Vol 5 (2) ◽  
pp. 179
Author(s):  
Novi Dwi Yanti ◽  
Suryana Suryana ◽  
Yulia Fitri

World Health Organization has estimated that the main cause of death in degenerative diseases is coronary heart disease (CHD). Coronary heart disease can be caused by narrowing of the blood vessels due to high levels of fat in the blood. This is due to excessive intake of fat and carbohydrates, and not doing physical activity or the body is not moving. This study aims to determine the blood lipid profile of outpatients with coronary heart disease. This descriptive-analytic study was used as a case-control study design, with a sample of 64 outpatients. The study was conducted at the Meuraxa Hospital Banda Aceh in March 2019. Intake data were collected using the SQ-FFQ method and physical activity data using the PAL method, while HDL, LDL, and triglyceride data were obtained through secondary data from hospital medical records. The statistical test was used by the Chi-Square test. The results showed that there was an effect of fat intake on blood lipid profiles in coronary heart disease patients (p= 0,006 with OR= 4,80), and the effect of carbohydrate intake (p= 0,032 and OR= 2,96), on the contrary, physical activity (p= 0,130) with a lipid profile showed no association. In conclusion, the intake of fat and carbohydrates has an effect on blood lipid profiles, but physical activity does not affect the blood lipid profiles of patients with coronary heart disease. Suggestions, it is necessary to maintain a balance of consumption of foods that are high in fat, and it is necessary to do consultations regarding low-fat diets

2012 ◽  
Vol 9 (6) ◽  
pp. 867-874 ◽  
Author(s):  
Peter Pagels ◽  
Anders Raustorp ◽  
Trevor Archer ◽  
Ulf Lidman ◽  
Marie Alricsson

Background:Health organizations suggest that adults ought to engage in at least 30 minutes of moderate-intensity daily physical activity. This study investigated the effects of a 30-minute single daily bout of brisk walking upon risk factors for coronary heart disease with blood lipid profile in particular.Methods:Thirty-three (25–45 y) adults, were randomly assigned into an exercise group (EG; n = 16, 9w) and a control group (CG; n = 17, 6w). The EG walked briskly 30 minutes daily during the 3-week test period. Compliance/adherence was maximal throughout the 3-week intervention due to stringent daily monitoring.Results:The EG showed a significant decrease in concentrations of low density lipoprotein cholesterol (LDL-C) and total cholesterol (TC) during the intervention period. A significant inverse correlation between Δ energy expenditure/day and Δ LDL-C (r = –0.39, P < .05) and an improvement in weight and BMI in the EG was found. Average steps during 30 minutes brisk walking bout was 3669 steps/bout generating a mean energy expenditure of 191 kcal/ bout.Conclusions:The most unique findings were that daily single bouts of moderate-intensity physical activity for 30 minutes, during 3 weeks, induced favorable effects upon body weight, BMI, and blood concentration of LDL-C and TC in healthy adults.


2007 ◽  
Vol 39 (Supplement) ◽  
pp. S233
Author(s):  
Shannon Duff ◽  
Hassanali Vatanparast ◽  
Jonathan Little ◽  
Denise Cornish ◽  
Stephen Cornish ◽  
...  

2001 ◽  
Vol 28 (12) ◽  
pp. 197-204
Author(s):  
Eriko SEKI ◽  
Yoshiro WATANABE ◽  
Satoshi SUNAYAMA ◽  
Yoshitaka IWAMA ◽  
Kazunori SHIMADA ◽  
...  

2017 ◽  
Vol 4 (1) ◽  
pp. 39-44
Author(s):  
Ni Made Restina Juliani ◽  
I Putu Oka Dharmawan ◽  
Putu Ayu Parwati

Introduction: Low Density Lipoprotein (LDL) is a type of low-density lipoprotein and the most widely transported cholesterol in the body. Increased levels of LDL in the body can be affected by genetics, age, gender, obesity, physical activity, lifestyle, drug consumption and smoking. Substances in a cigarette can cause an increase of LDL levels. Increased of LDL cholesterol levels can cause Coronary Heart Disease (CHD). The purpose of this research is to know the description of Low Density Lipoprotein (LDL) levels on smoker and non-smoker adolescent in Buyan Hamlet, Pancasari Village, Sukasada District, Buleleng Bali. Method: The type of this research is descriptive. This research was conducted in April-May 2017, which used fasting blood samples of 42 respondents. Result: From the average result of LDL level in smoker adolescent that is 134,91 mg/dL higher than the average of LDL level in non-smoker adolescent that is 74,90 mg/dL. The result of LDL cholesterol levels was determined by 21 smoker adolescent respondents with the close to optimal category (100-129 mg/dL) as many as 9 people (42,8%), and 12 people (57,3%) with worry category (130-159 mg/dL). Whereas in 21 non-smoker adolescent respondents obtained  result of LDL cholesterol level test with optimal category (<100 mg/dL) counted 18 people (87,71%) and 3 person (14,30%) with close to optimal category (100-129 mg/dL). Discussion: Based on the results of this research can be concluded that in smoker adolescent obtained LDL levels with close to optimal category and worrying whereas in non-smoker adolescents obtained LDL levels in the optimal category and close to optimal.


2020 ◽  
Vol 35 (Supplement_3) ◽  
Author(s):  
Natalia Stepanova ◽  
Ganna Tolstanova ◽  
Iryna Akulenko ◽  
Olena Savchenko ◽  
Larysa Lebid ◽  
...  

Abstract Background and Aims Cardiovascular disease (CVD) remains the leading cause of morbidity and death in end-stage renal disease (ESRD) patients. Thus, the accumulation of oxalate due to ESRD can increase the risk of CVD. Also, the ability of fecal oxalate-degrading activity (ODA) to reduce plasma oxalate levels has been discussed. On the other hand, despite a key role of atherogenic dyslipidemia in the development of CVD, only a few studies have shown a potential role of microbiota in the regulation of blood lipid profiles in the general population. However, at present, there is a general lack of research on this topic in dialysis patients.   The present study aimed to investigate the association between oxalate-degrading activity (ODA) in fecal microbiota and blood lipid profiles in ESRD patients. Method We represented the data of a cross-sectional pilot study examining ODA in fecal microbiota, plasma oxalate concentration (POx) and blood lipid profile markers in 32 ESRD patients. Among the patients, there were 21 hemodialysis (HD) patients and 11 peritoneal dialysis (PD) patients. The average age of the patients was 52.5 [39; 65] years. The redoximetric titration with KMnO4 was adopted to evaluate total ODA in fecal microbiota. The results were expressed in % oxalate degradation per 0.01 g of feces. The blood lipid spectrum was determined in all patients: total cholesterol level (TC), high (HDL), low (LDL) and very low (VLDL) density cholesterol, triglycerides (TG) and atherogenic index of plasma (AIP). The median (Me) and interquartile ranges [Q25; Q75] were calculated using the nonparametric Kruskal-Wallis test. The Spearman test was used for the correlation analysis. Chi-square tests were used for comparison of 2 proportions. All statistical analyses were performed using MedCalc. Results Dyslipidemia defined as an increase in atherogenic lipoprotein fractions and inhibition of HDL cholesterol was identified in 13/32 (40.6%) ESRD patients. Negative ODA in fecal microbiota (≤ 0 % /0.01 g) was observed in 14/32 (43.7%) patients. Among them, there were 5/21 (23.8%) HD patients and 7/11 (63.6%) PD patients (χ2=3.9, p=0.04). Significantly lower ODA in fecal microbiota was observed in the patients with atherogenic dyslipidemia in comparison with dyslipidemia-free patients (-5.0 [-8.7; 3.7] vs 3.0 [0.5; 9] %/0.01 g of feces, p=0.02) (Fig. 1). Less ODA in fecal microbiota was, higher levels of TG (r=-0.5, p=0.004) (Fig. 2), VLDL (r=-0.34, p=0.05) (Fig. 3) and, accordingly, AIP (r=-0.38, p=0.03) (Fig. 4) occurred. Conclusion The results of our study have provided preliminary evidence on the fact that a decrease in total ODA in fecal microbiota is associated with atherogenic dyslipidemia. Further studies are needed to determine the role of fecal ODA in the formation of dyslipidemia.


Sign in / Sign up

Export Citation Format

Share Document