Effect of vasopressors on fetal umbilical arterial blood after spinal anaesthesia during caesarean section

2021 ◽  
Vol 18 (3) ◽  
pp. 117-121
Author(s):  
Divya V ◽  
2021 ◽  
pp. 25-28
Author(s):  
Anant Prakash ◽  
Rahul Kumar ◽  
Chandeshwar Choudhary ◽  
Debarshi Jana

Introduction: Spinal anaesthesia has become a popular technique for caesarean section. It however has the potential to cause rapid onset of maternal hypotension which may have detrimental maternal and neonatal effects. Thus, a number of strategies for treating hypotension have been investigated. Careful positioning and volume preloading with crystalloid or colloids have been used to prevent it, but these are not complete measures and vasopressor is required to correct hypotension quickly . Methodology: 100 parturients ASA I and II scheduled for elective as well as emergency Caesarean section under sub arachnoid block (SAB) were studied. All parturients were at term, had uncomplicated singleton pregnancy with cephalic presentation. Results: Phenylephrine and Ephedrine are effective given prophylactically IV bolus followed by infusion in maintenance of arterial pressure within 15% limit of baseline. Phenylephrine has quicker and shorter duration of action effect in comparison to Ephedrine. Conclusion: Vasopressor drugs Phenylephreine and Ephedrine, effectively maintained arterial blood pressure during spinal anaesthesia for caesarean section


2012 ◽  
Vol 8 (4) ◽  
pp. 415-419
Author(s):  
J K Mitra

Hypotension during spinal anaesthesia for caesarean section remains a common scenario in our clinical practice. Certain risk factors play a role in altering the incidence of hypotension. Aortocaval compression counteraction does not help to prevent hypotension. Intravenous crystalloid prehydration has poor efficacy; thus, the focus has changed toward co-hydration and use of colloids. Phenylephrine is established as a first- line vasopressor, although there are limited data from high-risk patients. Ephedrine crosses the placenta more than phenylephrine and cause possible alterations in the foetal physiology.http://dx.doi.org/10.3126/kumj.v8i4.6242 Kathmandu Univ Med J 2010;8(4):415-19   


The Lancet ◽  
1930 ◽  
Vol 215 (5567) ◽  
pp. 1012-1014 ◽  
Author(s):  
John William Burns

1996 ◽  
Vol 77 (2) ◽  
pp. 301
Author(s):  
R N Foster ◽  
C M Rogers

1996 ◽  
Vol 76 (2) ◽  
pp. 335-336 ◽  
Author(s):  
A Mallick ◽  
A Samaan ◽  
P Braithwaite

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