scholarly journals The laboratory-assessed performance predictors of elite cross-country marathon mountain bikers

Kinesiology ◽  
2021 ◽  
Vol 53 (2) ◽  
pp. 262-270
Author(s):  
Everton Crivoi do Carmo ◽  
Renato Barroso ◽  
Danilo Leite Prado ◽  
Allan Inoue ◽  
Tatiane Machado ◽  
...  
Keyword(s):  
Power Output ◽  
Completion Time ◽  
Peak Power ◽  
Ventilatory Threshold ◽  
Lower Limbs ◽  
Peak Power Output ◽  
Mean Power ◽  
Jump Performance ◽  
Cross Country ◽  
Race Performance

The study aimed to investigate the relationship between laboratory-assessed variables and cross-country marathon mountain biking (XCM-MTB) performance to suggest a more practical approach to monitor performance during the competitive phase. Nine elite athletes performed a battery of tests during the competitive phase of the season, one week before the race. Correlations between the physiological and neuromuscular laboratory-assessed variables and mean race completion time were verified and a multiple regression model was calculated. Cycling economy (r=0.86), power output at the first (POVT1; r=-0.73) and second ventilatory threshold (POVT2; r=-0.94), VO2peak (r=-0.71), peak power output (r=-0.91); peak power (r=-0.80), and mean power (r=-0.85) were very strongly related to race performance. TMG-derivate factors as rectus femoris (r=-0.61) and biceps femoris (r=-0.59) contraction velocity, 30-cm drop jump performance (r=-0.59), mean propulsive power in jump squat at 40% (JS40%; r=-0.65) and 60% (JS60%; r=-0.62) of athlete’s body weight and lower limbs maximal isometric voluntary strength (r=-0.59) were strongly related to race completion time. In an isolated way, the POVT2 explained 87% of race performance. Although both physiological and neuromuscular variables are related to XCM performance, the POVT2 seems to be the main variable during the competitive phase and an easy-to-apply approach should be used to monitor athletes’ performance.

Acute High-Intensity Interval Training Improves Tvent and Peak Power Output in Highly Trained Males

2002 ◽  
Vol 27 (4) ◽  
pp. 336-348 ◽  
Author(s):  
Paul B. Laursen ◽  
Michelle A. Blanchard ◽  
David G. Jenkins
Keyword(s):  
Oxygen Uptake ◽  
Power Output ◽  
High Intensity ◽  
Peak Power ◽  
Ventilatory Threshold ◽  
Interval Training ◽  
Peak Power Output ◽  
Control Group ◽  
High Intensity Interval Training ◽  
High Intensity Interval

This study examined the effects of four high-intensity interval-training (HIT) sessions performed over 2 weeks on peak volume of oxygen uptake [Formula: see text] the first and second ventilatory thresholds (VT1, VT2) and peak power output (PPO) in highly trained cyclists. Fourteen highly trained male cyclists [Formula: see text] performed a ramped cycle test to determine [Formula: see text]VT1, VT2, and PPO. Subjects were divided equally into a HIT group and a control group. The HIT group performed four HIT sessions (20 × 60 s at PPO, 120 s recovery); the [Formula: see text] test was repeated < 1 wk after the HIT program. Control subjects maintained their regular training program and were reassessed under the same timeline. There was no change in [Formula: see text] for either group; however, the HIT group showed a significantly greater increase in VT1 (+22% vs. −3%), VT2 (+15% vs. −1%), and PPO (+4.3 vs. −4%) compared to controls (all P < .05). This study has demonstrated that HIT can improve VT1, VT2 and PPO, following only four HIT sessions in already highly trained cyclists. Key words: cycling, cyclists, heart rate, oxygen uptake, short-term training, ventilatory threshold

Power– and velocity–load relationships to improve resistance exercise performance

2018 ◽  
Vol 232 (4) ◽  
pp. 349-359 ◽  
Author(s):  
Manuel V Garnacho-Castaño ◽  
Arturo Muñoz-González ◽  
María A Garnacho-Castaño ◽  
José L Maté-Muñoz
Keyword(s):  
Power Output ◽  
Peak Power ◽  
Bench Press ◽  
Peak Velocity ◽  
Peak Power Output ◽  
Mean Power ◽  
Mean Velocity ◽  
Maximal Power Output ◽  
Power Load ◽  
The Military

Knowledge of the power– and velocity–load relationships is a key factor to guide loads during resistance training and optimize sports performance. This study compares mean velocity–, peak velocity– and power–load relationships, and determines the load which elicits maximal power output in the military press and bench press. Fifty-seven healthy, active men were randomly assigned to a bench press (n = 28) or military press (n = 29) group. In separate test sessions, concentric-only or eccentric-concentric sequences of each exercise were performed in random order as incremental isoinertial load tests. Both mean velocity and peak velocity were highly related with the load lifted (% 1RM) in both bench press and military press (mean velocity: R2 = 0.94 and 0.95; peak velocity: R2 = 0.93 and 0.93, respectively). The loads maximizing mean power and peak power output were similar for the eccentric-concentric versus concentric sequences in bench press and military press. The loads maximizing mean power and peak power were between 54% and 57.5% 1RM for the bench press and 59.8%–63.1% 1RM for the military press. For the bench press, no significant differences were observed in mean power from 30% to 80% 1RM and peak power from 30% to 95% 1RM. For the military press, no significant differences were observed in mean power from 40% to 80% 1RM and peak power from 30% to 90%/95% 1RM. The close relationship detected between mean velocity or peak velocity and load means that the % 1RM can be estimated according to mean velocity and peak velocity. In both exercises, a broad range of relative intensities could be used at which power output is not significantly different than that at maximized power output (mean = 30%/40%–80% 1RM; peak = 30%–90%/95%). Mean velocity lower than approximately 0.33 m s−1 for bench press and 0.4 m s−1 for military press, as well as peak velocity lower than approximately 0.4 m s−1 for bench press and 0.5 m s−1 for military press do not optimize power output responses. The eccentric action was a determining factor for increasing power output only in bench press.

scholarly journals No Effect Of 8 Days Of Heat Acclimation On VO 2peak , Peak Power Output, Or Ventilatory Threshold

2020 ◽  
Vol 52 (7S) ◽  
pp. 527-528
Author(s):  
Karleigh E. Bradbury ◽  
Katherine M. Mitchell ◽  
Beau R. Yurkevicius ◽  
Adam J. Luippold ◽  
Kirsten E. Coffman ◽  
...  
Keyword(s):  
Power Output ◽  
Peak Power ◽  
Ventilatory Threshold ◽  
Heat Acclimation ◽  
Peak Power Output

scholarly journals The Effect of Prior Upper Body Exercise on Subsequent Wingate Performance

2014 ◽  
Vol 2014 ◽  
pp. 1-7 ◽  
Author(s):  
Marie Clare Grant ◽  
Robert Robergs ◽  
Marianne Findlay Baird ◽  
Julien S. Baker
Keyword(s):  
Power Output ◽  
Peak Power ◽  
Detrimental Effect ◽  
Recovery Period ◽  
Upper Body ◽  
Peak Power Output ◽  
Mean Power ◽  
Upper Body Exercise ◽  
Mean Power Output ◽  
Trial Participants

It has been reported previously that the upper body musculature is continually active during high intensity cycle ergometry. The aim of this study was to examine the effects of prior upper body exercise on subsequent Wingate (WAnT) performance. Eleven recreationally active males (20.8 ± 2.2 yrs; 77.7 ± 12.0 kg; 1.79 ± 0.04 m) completed two trials in a randomised order. In one trial participants completed2×30 s WAnT tests (WAnT1 and WAnT2) with a 6 min recovery period; in the other trial, this protocol was preceded with 4 sets of biceps curls to induce localised arm fatigue. Prior upper body exercise was found to have a statistically significant detrimental effect on peak power output (PPO) during WAnT1(P<0.05)but no effect was observed for mean power output (MPO)(P>0.05). Handgrip (HG) strength was also found to be significantly lower following the upper body exercise. These results demonstrate that the upper body is meaningfully involved in the generation of leg power during intense cycling.

scholarly journals Beta2-agonist salbutamol augments hypertrophy in MHCIIa fibers and sprint power output but not muscle force during 11 weeks of resistance training in young men

2020 ◽  
Author(s):  
Søren Jessen ◽  
Søren Reitelseder ◽  
Anders Kalsen ◽  
Michael Kreiberg ◽  
Johan Onslev ◽  
...  
Keyword(s):  
Resistance Training ◽  
Sarcoplasmic Reticulum ◽  
Power Output ◽  
Peak Power ◽  
Contractile Function ◽  
Young Men ◽  
Maximal Activity ◽  
Peak Power Output ◽  
Mean Power ◽  
Mean Power Output

In this study, we examined the effect of beta2-agonist salbutamol at oral doses during a period of resistance training on sprint performance, quadriceps contractile function, skeletal muscle hypertrophy, fiber-type composition, maximal activity of enzymes of importance for anaerobic energy turnover, and sarcoplasmic reticulum Ca2+-handling in young men. Twenty-six men (23±2 years;mean±SD) were randomized to daily intake of oral salbutamol (16 mg/d;RES+SAL) or placebo (RES) during 11 weeks full-body resistance training 3 times/week. Mean power output during 10s maximal cycling increased more (P=0.027) in RES+SAL (+12%) than in RES (+7%), whereas peak power output increased similarly (RES+SAL:+8%;RES:+7%;P=0.400). Quadriceps dynamic peak torque and maximal voluntary isometric torque increased by 13 and 14% (P≤0.001) in RES+SAL and 13 and 13% (P≤0.001) in RES, respectively. Myosin heavy chain (MHC) isoform distribution transitioned from MHCI and MHCIIx towards MHCIIa in RES+SAL (P=0.002), but not in RES (P=0.323). MHCIIa cross-sectional-area increased more (P=0.040) in RES+SAL (+35%) than RES (+21%). Sarcoplasmic reticulum Ca2+-release rate increased in both groups (RES+SAL:+9%,P=0.048;RES:+13%,P=0.008), whereas Ca2+-uptake rate increased only in RES (+12%,P=0.022) but not different from the non-significant change in RES+SAL (+2%,P=0.484). Maximal activity of lactate dehydrogenase increased only in RES+SAL (+13%,P=0.008). Muscle content of the dihydropyridine receptor, ryanodine receptor 1, and sarcoplasmic reticulum Ca2+-ATPase isoform 1 and 2 did not change with the intervention in either group (P≥0.100). These observations suggest that salbutamol is a muscle anabolic drug, which induces greater sprint mean power output, without affecting peak power output and muscle strength when ingested during a period of resistance training.

Is the Functional Threshold Power a Valid Surrogate of the Lactate Threshold?

2018 ◽  
Vol 13 (10) ◽  
pp. 1293-1298 ◽  
Author(s):  
Pedro L. Valenzuela ◽  
Javier S. Morales ◽  
Carl Foster ◽  
Alejandro Lucia ◽  
Pedro de la Villa
Keyword(s):  
Effect Size ◽  
Power Output ◽  
Lactate Threshold ◽  
Peak Power ◽  
Time Trial ◽  
Peak Power Output ◽  
Threshold Power ◽  
Limits Of Agreement ◽  
Mean Power ◽  
Mean Power Output

Purpose: To analyze the relationship between functional threshold power (FTP) and the lactate threshold (LT). Methods: A total of 20 male cyclists performed an incremental test in which LT was determined. At least 48 h later, they performed a 20-min time trial, and 95% of the mean power output was defined as FTP. Participants were divided into recreational (peak power output < 4.5 W·kg−1; n = 11) or trained cyclists (peak power output > 4.5 W·kg−1; n = 9) according to their fitness status. Results: The FTP (240 [35] W) was overall not significantly different (effect size = 0.20; limits of agreement = −2.4% [11.5%]) from the LT (246 [24] W), and both markers were strongly correlated (r = .95; P < .0001). Accounting for the participants’ fitness status, no significant differences were found between FTP and LT (effect size = 0.22; limits of agreement =2.1% [7.8%]) in trained cyclists, but FTP was significantly lower than the LT (P = .0004, effect size = 0.81; limits of agreement =−6.5% [8.3%]) in recreational cyclists. A significant relationship was found between relative peak power output and the bias between FTP and the LT markers (r = .77; P < .0001). Conclusions: FTP is a valid field test-based marker for the assessment of endurance fitness. However, caution should be taken when using FTP interchangeably with LT, as the bias between markers seems to depend on the athlete’s fitness status. Whereas FTP provides a good estimate of LT in trained cyclists, in recreational cyclists, it may underestimate LT.

Abstract P134: Preliminary Data: Dietary Nitrate Supplementation Does Not Extend Dry Static Apnea Or Wingate Performance

Circulation ◽  
2021 ◽  
Vol 143 (Suppl_1) ◽  
Author(s):  
Colin Carriker ◽  
Phillip Armentrout ◽  
Sarah Levine ◽  
James Smoliga
Keyword(s):  
Power Output ◽  
Preliminary Data ◽  
Repeated Measures ◽  
Peak Power ◽  
Hold Time ◽  
Wingate Test ◽  
Peak Power Output ◽  
Breath Hold ◽  
Dietary Nitrate ◽  
Nitrate Supplementation

Introduction: Previous studies have examined dietary nitrate supplementation and its effects on dry static apnea, and peak power. Dietary nitrate supplementation has been found to increase maximal apnea and peak power output. The purpose of this study was to determine the effects of beetroot juice on dry static apnea and Wingate performance. Hypothesis: Dietary nitrate will improve maximal breath hold time and peak power output. Dietary nitrate will improve tolerance to CO2, thereby improving maximal breath hold time and anaerobic capacity. Methods: In a randomized, double-blind, counterbalanced study, five healthy males (20.4±0.89 years) visited the lab on 3 separate occasions each separated by one week. Visit 1 served as a Wingate and breath hold familiarization visit. Prior to visits 2 and 3 participants were instructed to drink a beverage either a placebo (negligible nitrate content, PL) or dietary nitrate rich beverage (12.4 mmol nitrate, NIT) during the 4 days leading up to their next visit. Visits 2 and 3 consisted of two submaximal breath holds (80% of maximal determined during visit 1), with 2 minutes of rest between and three minutes of rest preceding the final breath hold for maximal duration. Finally, participants completed a standardized 10-minute warmup on the cycle ergometer before completing a 30-second maximal effort Wingate test. Results: A linear mixed effects model was used to determine whether treatment (NIT vs. PL) was associated with differences in VCO2 or PetCO2. Time (0, 10, 20, 30 min post-breath hold) and Treatment both served as repeated measures. Models were developed using multiple repeated measures covariance matrix structures, and the model with the lowest AIC was chosen as the final model. The interaction between time and treatment was included in the original models, and was removed if it was not statistically significant. Time was a statistically significant factor for VCO2 and PetCO2 (p < 0.001). Treatment, and the Time x Treatment interaction was not significant for either variable. No differences between NIT and PL were observed during the Wingate test for either time to peak power (5.02±2.45 and 6.2±2.43 sec, respectively) or maximal power (9.73±1.01 and 9.72±1.03 watts/kg, respectively) and fatigue index (49.42±14.98 and 47.30±6.99 watts/sec, respectively). Conclusion: Preliminary data indicates that in a general population four days of dietary nitrate supplementation may not improve breath hold time, tolerance to carbon dioxide in the lungs, or Wingate performance.

Force-velocity and force-power properties of single muscle fibers from elite master runners and sedentary men

1996 ◽  
Vol 271 (2) ◽  
pp. C676-C683 ◽  
Author(s):  
J. J. Widrick ◽  
S. W. Trappe ◽  
D. L. Costill ◽  
R. H. Fitts
Keyword(s):  
Myosin Heavy Chain ◽  
Heavy Chain ◽  
Power Output ◽  
Peak Power ◽  
Isometric Force ◽  
Peak Force ◽  
Peak Power Output ◽  
Type I ◽  
Shortening Velocity ◽  
Force Velocity

Gastrocnemius muscle fiber bundles were obtained by needle biopsy from five middle-aged sedentary men (SED group) and six age-matched endurance-trained master runners (RUN group). A single chemically permeabilized fiber segment was mounted between a force transducer and a position motor, subjected to a series of isotonic contractions at maximal Ca2+ activation (15 degrees C), and subsequently run on a 5% polyacrylamide gel to determine myosin heavy chain composition. The Hill equation was fit to the data obtained for each individual fiber (r2 > or = 0.98). For the SED group, fiber force-velocity parameters varied (P < 0.05) with fiber myosin heavy chain expression as follows: peak force, no differences: peak tension (force/fiber cross-sectional area), type IIx > type IIa > type I; maximal shortening velocity (Vmax, defined as y-intercept of force-velocity relationship), type IIx = type IIa > type I; a/Pzero (where a is a constant with dimensions of force and Pzero is peak isometric force), type IIx > type IIa > type I. Consequently, type IIx fibers produced twice as much peak power as type IIa fibers, whereas type IIa fibers produced about five times more peak power than type I fibers. RUN type I and IIa fibers were smaller in diameter and produced less peak force than SED type I and IIa fibers. The absolute peak power output of RUN type I and IIa fibers was 13 and 27% less, respectively, than peak power of similarly typed SED fibers. However, type I and IIa Vmax and a/Pzero were not different between the SED and RUN groups, and RUN type I and IIa power deficits disappeared after power was normalized for differences in fiber diameter. Thus the reduced absolute peak power output of the type I and IIa fibers from the master runners was a result of the smaller diameter of these fibers and a corresponding reduction in their peak isometric force production. This impairment in absolute peak power production at the single fiber level may be in part responsible for the reduced in vivo power output previously observed for endurance-trained athletes.

Arm muscle sympathetic nerve activity during preparation for and initiation of leg-cycling exercise in humans

1994 ◽  
Vol 77 (3) ◽  
pp. 1403-1410 ◽  
Author(s):  
R. Callister ◽  
A. V. Ng ◽  
D. R. Seals
Keyword(s):  
Power Output ◽  
Peak Power ◽  
General Pattern ◽  
Peak Power Output ◽  
Movement Initiation ◽  
Nerve Activity ◽  
Cycling Exercise ◽  
Target Power ◽  
Leg Cycling ◽  
Exercise In Humans

We tested the hypothesis that sympathetic vasoconstrictor nerve activity to nonactive skeletal muscle (MSNA) decreases immediately before and remains suppressed during initiation of conventional large muscle upright dynamic exercise in humans. In 11 healthy young subjects, adequate recordings of MSNA from the radial nerve in the arm were obtained during upright seated rest (control) and throughout 1 min of leg-cycling exercise at one or more submaximal workloads (range 33–266 W; approximately 10–80% of peak power output). MSNA was analyzed during four consecutive time intervals; control, preparation for cycling (end of control to onset of pedal movement), initiation of cycling (onset of pedal movement to attainment of target power output), and the initial 60 s of cycling at target power output. MSNA decreased (P < 0.05) abruptly and markedly in all subjects [to 19 +/- 4% (SE) of control levels] during the preparation period before the 33-W load and remained suppressed throughout the period of initiation of cycling in 8 of 11 subjects; MSNA increased during the initiation period in three subjects in whom diastolic arterial pressure fell below control levels. This general pattern was observed at all loads. MSNA remained at or below control levels throughout the 1 min of cycling exercise at 33–166 W. MSNA increased above control levels during the latter portion of the 1 min of cycling only at loads > or = 60% of peak power output.(ABSTRACT TRUNCATED AT 250 WORDS)

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