scholarly journals Positional differences in some physiological parameters obtained by the incremental field endurance test among elite handball players

Kinesiology ◽  
2021 ◽  
Vol 53 (1) ◽  
pp. 3-11
Author(s):  
Uroš Mohorič ◽  
Marko Šibila ◽  
Boro Štrumbelj

The purpose of the study was to assess assumed differences in some physiological parameters, obtained by an incremental intermittent running field test 30–15IFT, among elite handball players to get an insight into the specifics of aerobic capacity profiles of players in different playing positions. Twenty-four elite male handball players were tested using the Cosmed K4 portable telemetry system. The following parameters were analysed: running velocity, heart rate, oxygen uptake, relative oxygen uptake, pulmonary ventilation breath-by-breath, at the three points—lactate threshold (LT), onset of blood lactate accumulation (OBLA), and at the peak velocity achieved on the test (v30–15IFT). Additionally, blood lactate concentration was analysed at v30–15IFT. The players were divided in three groups based on their playing positions: eight backcourt players, eight wing players and eight pivot players. In terms of both the statistically significant and non-significant differences, the wings achieved slightly different results in comparison to the backcourt players and pivots. The wings reached a statistically significant higher velocity at the LT than the players of the other two groups and a significantly higher velocity than the pivots at the OBLA. At all the three points, wings presented the highest HR values, meaning they can operate at higher intensities still within the aerobic work zone. This would probably allow wing players to longer persist in handball game.

2006 ◽  
Vol 38 (Supplement) ◽  
pp. S514
Author(s):  
Matthew J. Garver ◽  
Leland J. Nielsen ◽  
Jared M. Dickinson ◽  
Derek S. Campbell ◽  
Charilaos Papadopoulos ◽  
...  

1996 ◽  
Vol 4 (3) ◽  
pp. 286-296
Author(s):  
Fiona Iredale ◽  
Frank Bell ◽  
Myra Nimmo

Fourteen sedentary 50- to 55-year-old men were exercised to exhaustion using an incremental treadmill protocol. Mean (±SEM) peak oxygen uptake (V̇O2peak) was 40.5 ± 1.19 ml · kg1· min−1, and maximum heart rate was 161 ± 4 beats · min−1. Blood lactate concentration was measured regularly to identify the lactate threshold (oxygen consumption at which blood lactate concentration begins to systematically increase). Threshold occurred at 84 ± 2% of V̇O2peak. The absolute lactate value at threshold was 2.9 ± 0.2 mmol · L−1. On a separate occasion, 6 subjects exercised continuously just below their individual lactate thresholds for 25 min without significantly raising their blood lactate levels from the 10th minute to the 25th. The absolute blood lactate level over the last 20 min of the steady-state test averaged 3.7 ± 1.2 mmol · L−1. This value is higher than that elicited at the threshold in the incremental test because of the differing nature of the protocols. It was concluded that although the lactate threshold occurs at a high percentage of V̇O2peak, subjects are still able to sustain exercise at that intensity for 25 min.


1993 ◽  
Vol 84 (4) ◽  
pp. 441-447 ◽  
Author(s):  
Peter Báaráany ◽  
Ulla Freyschuss ◽  
Erna Pettersson ◽  
Jonas Bergström

1. The effects of correcting anaemia on exercise capacity were evaluated in 21 haemodialysis patients (aged 39 ± 12 years) before starting treatment with recombinant human erythropoietin (Hb concentration, 73 ± 10 g/l; total Hb, 59 ± 12% of expected), after correction of the anaemia to a Hb concentration of 108 ± 7 g/l and a total Hb 82 ± 10% of expected, and in 13 of the patients after 12 months on maintenance recombinant human erythropoietin treatment (Hb concentration 104 ± 14 g/l, total Hb 79 ± 17% of expected). Fifteen healthy subjects (aged 41 ± 9 years), who took no regular exercise, constituted the control group. Maximal exercise capacity was determined on a bicycle ergometer. Oxygen uptake, respiratory quotient, blood lactate concentration, heart rate and blood pressure were measured at rest and at maximal workload. 2. After 6 ± 3 months on recombinant human erythropoietin, maximal exercise capacity increased from 108 ± 27 W to 130 ± 36 W (P < 0.001) and the maximal oxygen uptake increased from 1.24 ± 0.39 litres/min to 1.50 ± 0.45 litres/min (P < 0.001). No significant changes in respiratory quotient (1.16 ± 0.13 versus 1.18 ± 0.13) and blood lactate concentration (4.0 ± 1.8 versus 3.6 ± 1.1 mmol/l) at maximal workload were observed, but the blood lactate concentration in the patients was significantly lower than that in the control subjects (6.7 ± 2.3 mmol/l, P < 0.01). After the correction of anaemia, the aerobic power was still 38% lower in the patients than in the control subjects and 17% lower than the reference values. 3. After 12 months on maintenance recombinant human erythropoietin treatment (17 ± 3 months from the start of the study), no further significant changes were observed in maximal exercise capacity (before start, 112 ± 31 W, 6 ± 3 months, 134 ± 42 W, 17 ± 3 months, 134 ± 50 W), maximal oxygen uptake (before start, 1.33 ± 0.45 litres/min; 6 ± 3 months, 1.59 ± 0.54 litres/min; 17 ± 3 months, 1.75 ± 0.78 litres/min) or blood lactate concentration (before start, 4.4 ± 1.9 mmol/l; 6 ± 3 months, 4.0 ± 1.0 mmol/l; 17 ± 3 months, 4.7 ± 2.0 mmol/l). 4. Thus, in haemodialysis patients the improvement in maximal aerobic power after the correction of anaemia persists without marked changes during long-term treatment with recombinant human erythropoietin. We did not observe any effects on exercise capacity that could be attributed to a spontaneous increase in physical activity after treatment of anaemia.


2015 ◽  
Vol 45 (1) ◽  
pp. 217-224 ◽  
Author(s):  
José Luiz Dantas ◽  
Christian Doria

Abstract Incremental tests on a treadmill are used to evaluate endurance athletes; however, no criterion exists to determine the intensity at which to start the test, potentially causing the loss of the first lactate threshold. This study aimed to determine the ideal speed for runners to start incremental treadmill tests. The study consisted of 94 runners who self-reported the average speed from their last competitive race (10-42.195 km) and performed an incremental test on a treadmill. The speeds used during the first three test stages were normalised in percentages of average competition speed and blood lactate concentration was analysed at the end of each stage. The relationship between speed in each stage and blood lactate concentration was analysed. In the first stage, at an intensity corresponding to 70% of the reported average race speed, only one volunteer had blood lactate concentration equal to 2 mmol·L-1, and in the third stage (90% of the average race speed) the majority of the volunteers had blood lactate concentration ≥2 mmol·L-1. Our results demonstrated that 70% of the average speed from the subject’s last competitive race - from 10 to 42.195 km - was the best option for obtaining blood lactate concentration <2 mmol·L-1 in the first stage, however, 80% of the average speed in marathons may be a possibility. Evaluators can use 70% of the average speed in competitive races as a strategy to ensure that the aerobic threshold intensity is not achieved during the first stage of incremental treadmill tests.


1997 ◽  
Vol 5 (1) ◽  
pp. 39-49 ◽  
Author(s):  
K. Fiona Iredale ◽  
Myra A. Nimmo

Thirty-three men (age 26–55 years) who did not exercise regularly were exercised to exhaustion using an incremental treadmill protocol. Blood lactate concentration was measured to identify lactate threshold (LT, oxygen consumption at which blood lactate concentration begins to systematically increase). The correlation coefficient for LT (ml · kg−1 · min−1) with age was not significant, but when LT was expressed as a percentage of peak oxygen consumption (VO2 peak), the correlation was r = +.69 (p < .01). This was despite a lack of significant correlation between age and VO2 peak (r = −.33). The correlation between reserve capacity (the difference between VO2 peak and LT) and age was r = −.73 (p < .01 ), and reserve capacity decreased at a rate of 3.1 ml · kg−1 · min−1 per decade. It was concluded that the percentage of VO2 peak at which LT occurs increases progressively with age, with a resultant decrease in reserve capacity.


1985 ◽  
Vol 59 (3) ◽  
pp. 853-859 ◽  
Author(s):  
E. F. Coyle ◽  
W. H. Martin ◽  
S. A. Bloomfield ◽  
O. H. Lowry ◽  
J. O. Holloszy

Seven endurance-trained subjects were studied 12, 21, 56, and 84 days after cessation of training. Heart rate, ventilation, respiratory exchange ratio, and blood lactate concentration during submaximal exercise of the same absolute intensity increased (P less than 0.05) progressively during the first 56 days of detraining, after which a stabilization occurred. These changes paralleled a 40% decline (P less than 0.001) in mitochondrial enzyme activity levels and a 21% increase in total lactate dehydrogenase (LDH) activity (P less than 0.05) in trained skeletal muscle. After 84 days of detraining, the experimental subjects' muscle mitochondrial enzyme levels were still 50% above, and LDH activity was 22% below, sedentary control levels. The blood lactate threshold of the detrained subjects occurred at higher absolute and relative (i.e., 75 +/- 2% vs. 62 +/- 3% of maximal O2 uptake) exercise intensities in the subjects after 84 days of detraining than in untrained controls (P less than 0.05). Thus it appears that a portion of the adaptation to prolonged and intense endurance training that is responsible for the higher lactate threshold in the trained state persists for a long time (greater than 85 days) after training is stopped.


2016 ◽  
Vol 41 (11) ◽  
pp. 1197-1203 ◽  
Author(s):  
Felipe Mattioni Maturana ◽  
Daniel A. Keir ◽  
Kaitlin M. McLay ◽  
Juan M. Murias

Critical power (CP) conceptually represents the highest power output (PO) at physiological steady-state. In cycling exercise, CP is traditionally derived from the hyperbolic relationship of ∼5 time-to-exhaustion trials (TTE) (CPHYP). Recently, a 3-min all-out test (CP3MIN) has been proposed for estimation of CP as well the maximal lactate steady-state (MLSS). The aim of this study was to compare the POs derived from CPHYP, CP3MIN, and MLSS, and the oxygen uptake and blood lactate concentrations at MLSS. Thirteen healthy young subjects (age, 26 ± 3years; mass, 69.0 ± 9.2 kg; height, 174 ± 10 cm; maximal oxygen uptake, 60.4 ± 5.9 mL·kg−1·min−1) were tested. CPHYP was estimated from 5 TTE. CP3MIN was calculated as the mean PO during the last 30 s of a 3-min all-out test. MLSS was the highest PO during a 30-min ride where the variation in blood lactate concentration was ≤ 1.0 mmol·L−1 during the last 20 min. PO at MLSS (233 ± 41 W; coefficient of variation (CoV), 18%) was lower than CPHYP (253 ± 44 W; CoV, 17%) and CP3MIN (250 ± 51 W; CoV, 20%) (p < 0.05). Limits of agreement (LOA) from Bland–Altman plots between CPHYP and CP3MIN (–39 to 31 W), and CP3MIN and MLSS (–29 to 62 W) were wide, whereas CPHYP and MLSS presented the narrowest LOA (–7 to 48 W). MLSS yielded not only the maximum PO of stable blood lactate concentration, but also stable oxygen uptake. In conclusion, POs associated to CPHYP and CP3MIN were larger than those observed during MLSS rides. Although CPHYP and CP3MIN were not different, the wide LOA between these 2 tests and the discrepancy with PO at MLSS questions the ability of CP measures to determine the maximal physiological steady-state.


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