In vivo determination of subcutaneous and abdominal adipose tissue depots in German Holstein dairy cattle1

2016 ◽  
Vol 94 (7) ◽  
pp. 2821-2834 ◽  
Author(s):  
C. Raschka ◽  
L. Ruda ◽  
P. Wenning ◽  
C.-I. von Stemm ◽  
C. Pfarrer ◽  
...  
Keyword(s):  
Adipose Tissue ◽  
Abdominal Adipose Tissue ◽  
Adipose Tissue Depots

An in vivo study of the cortisol-cortisone shuttle in subcutaneous abdominal adipose tissue

1999 ◽  
Vol 50 (1) ◽  
pp. 63-68 ◽  
Author(s):  
J. R. Katz ◽  
V. Mohamed-Ali ◽  
P. J. Wood ◽  
J. S. Yudkin ◽  
S. W. Coppack
Keyword(s):  
Adipose Tissue ◽  
In Vivo Study ◽  
Abdominal Adipose Tissue ◽  
Subcutaneous Abdominal Adipose Tissue

scholarly journals Effects of Hypo- and Hyperthyroidism on Noradrenergic Activity and Glycerol Concentrations in Human Subcutaneous Abdominal Adipose Tissue Assessed with Microdialysis

2003 ◽  
Vol 88 (12) ◽  
pp. 5605-5608 ◽  
Author(s):  
Martin Haluzik ◽  
Jara Nedvidkova ◽  
Vladimir Bartak ◽  
Ivana Dostalova ◽  
Petr Vlcek ◽  
...  
Keyword(s):  
Adipose Tissue ◽  
Thyroid Hormones ◽  
Relative Magnitude ◽  
In Vivo Microdialysis ◽  
Abdominal Adipose Tissue ◽  
Subcutaneous Abdominal Adipose Tissue ◽  
Hypothyroid Patients ◽  
Hyperthyroid Patients ◽  
Tissue Norepinephrine

Abstract Thyroid hormones play a major role in lipid metabolism. However, whether they directly affect lipolysis locally in the adipose tissue remains unknown. Therefore, we measured abdominal sc adipose tissue norepinephrine (NE), basal, and isoprenaline-stimulated lipolysis in 12 hypothyroid patients (HYPO), six hyperthyroid patients (HYPER), and 12 healthy controls by in vivo microdialysis. Adipose tissue NE was decreased in HYPO and increased in HYPER compared with controls (90.4 ± 2.9 and 458.0 ± 69.1 vs. 294.9 ± 19.5 pmol/liter, P < 0.01). Similarly, basal lipolysis, assessed by glycerol assay, was lower in HYPO and higher in HYPER than in controls (88.2 ± 9.9 and 566.0 ± 42.0 vs. 214.3 ± 5.1 μmol/liter P < 0.01). The relative magnitude of isoprenaline-induced glycerol increase was smaller in HYPO (39 ± 19.4%, P < 0.05 vs. basal) and higher in HYPER (277 ± 30.4%, P < 0.01) than in controls (117 ± 5.6%, P < 0.01). The corresponding changes in NE after isoprenaline stimulation were as follows: 120 ± 9.2% (P < 0.05), 503 ± 113% (P < 0.01), and 267 ± 17.2 (P < 0.01). In summary, by affecting local NE levels and adrenergic postreceptor signaling, thyroid hormones may influence the lipolysis rate in the abdominal sc adipose tissue.

scholarly journals On the existence of lipid peroxides in rat tissue

1972 ◽  
Vol 27 (1) ◽  
pp. 19-26 ◽  
Author(s):  
J. Glavind
Keyword(s):  
Adipose Tissue ◽  
Methyl Linoleate ◽  
Vitamin A ◽  
Rat Liver ◽  
Lipid Peroxides ◽  
Yellow Colour ◽  
Methyl Linoleate Hydroperoxide ◽  
Rat Tissue

1. The colorimetric micro-adaption of the iodometric method and the colorimetric thiocyanate method for the determination of lipoperoxides were compared. Similar results were obtained when methyl linoleate hydroperoxide was tested, but when lipid from rat liver, muscle, kindney and testes was examined, substantial amounts were found by the iodometric, but almost nothing by the thiocyanate method.2. The main reason for the discrepancy between the methods seems to be that the iodometric micromethod also estimates substances other than true lipoperoxides. The presence of ubiquinone and vitamin A in the organ extracts was shown to interfere in this way in the method.3. The yellow colour which develops when retinol and its esters are tested by the iodometric micromethod is due not to liberated iodine but to conversion products of retinol.4. It is concluded that the occurrence of substantial amounts of lipoperoxides in vivo has so far been demonstarted only in the adipose tissue, and not in the parenchymatous organs of the rat.

Inflammatory characteristics of distinct abdominal adipose tissue depots relate differently to metabolic risk factors for cardiovascular disease

2015 ◽  
Vol 239 (2) ◽  
pp. 419-427 ◽  
Author(s):  
Mariëtte E.G. Kranendonk ◽  
Joost A. van Herwaarden ◽  
Tereza Stupkova ◽  
Wilco de Jager ◽  
Aryan Vink ◽  
...  
Keyword(s):  
Risk Factors ◽  
Cardiovascular Disease ◽  
Adipose Tissue ◽  
Metabolic Risk Factors ◽  
Metabolic Risk ◽  
Abdominal Adipose Tissue ◽  
Adipose Tissue Depots

Obesity modifies expression profiles of metabolic markers in superficial and deep subcutaneous abdominal adipose tissue depots

Endocrine ◽  
2013 ◽  
Vol 46 (1) ◽  
pp. 99-106 ◽  
Author(s):  
Gillian E. Walker ◽  
Paolo Marzullo ◽  
Flavia Prodam ◽  
Gianni Bona ◽  
Anna Maria Di Blasio
Keyword(s):  
Adipose Tissue ◽  
Expression Profiles ◽  
Metabolic Markers ◽  
Abdominal Adipose Tissue ◽  
Adipose Tissue Depots ◽  
Subcutaneous Abdominal Adipose Tissue

scholarly journals Acute Hypercortisolemia Exerts Depot-Specific Effects on Abdominal and Femoral Adipose Tissue Function

2017 ◽  
Vol 102 (4) ◽  
pp. 1091-1101 ◽  
Author(s):  
Konstantinos N. Manolopoulos ◽  
Michael W. O’Reilly ◽  
Iwona J. Bujalska ◽  
Jeremy W. Tomlinson ◽  
Wiebke Arlt
Keyword(s):  
Adipose Tissue ◽  
Tissue Blood Flow ◽  
Healthy Male ◽  
Double Blind ◽  
Nonesterified Fatty Acids ◽  
Abdominal Adipose Tissue ◽  
Triglyceride Hydrolysis ◽  
Specific Effects ◽  
In Vivo Assessment

Abstract Context: Glucocorticoids have pleiotropic metabolic functions, and acute glucocorticoid excess affects fatty acid metabolism, increasing systemic lipolysis. Whether glucocorticoids exert adipose tissue depot-specific effects remains unclear. Objective: To provide an in vivo assessment of femoral and abdominal adipose tissue responses to acute glucocorticoid administration. Design and Outcome Measures: Nine healthy male volunteers were studied on two occasions, after a hydrocortisone infusion (0.2 mg/kg/min for 14 hours) and a saline infusion, respectively, given in randomized double-blind order. The subjects were studied in the fasting state and after a 75-g glucose drink with an in vivo assessment of femoral adipose tissue blood flow (ATBF) using radioactive xenon washout and of lipolysis and glucose uptake using the arteriovenous difference technique. In a separate study (same infusion design), eight additional healthy male subjects underwent assessment of fasting abdominal ATBF and lipolysis only. Lipolysis was assessed as the net release of nonesterified fatty acids (NEFAs) from femoral and abdominal subcutaneous adipose tissue. Results: Acute hypercortisolemia significantly increased basal and postprandial ATBF in femoral adipose tissue, but the femoral net NEFA release did not change. In abdominal adipose tissue, hypercortisolemia induced substantial increases in basal ATBF and NEFA release. Conclusions: Acute hypercortisolemia induces differential lipolysis and ATBF responses in abdominal and femoral adipose tissue, suggesting depot-specific glucocorticoid effects. Abdominal, but not femoral, adipose tissue contributes to the hypercortisolemia-induced systemic NEFA increase, with likely contributions from other adipose tissue sources and intravascular triglyceride hydrolysis.

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