scholarly journals Damage Resistance and Damage Tolerance of Hybrid Carbon-Glass Laminates

Author(s):  
Andrew Rhead ◽  
Richard Butler ◽  
Hua Shi
2018 ◽  
Vol 38 (1) ◽  
pp. 31-45 ◽  
Author(s):  
Gaye Kaya

This study aims to compare the low-velocity impact and post-impact properties of intra-ply hybrid carbon/E-glass/polypropylene non-crimp thermoplastic composites with non-hybrid carbon/PP and E-glass/PP non-crimp thermoplastic composites. Impact test was performed at four energy levels as 15 J, 30 J, 45 J and 60 J. Post-impact properties of hybrid thermoplastic composites were tested by compression after impact method for each energy level to understand the impact damage tolerance of intra-ply hybrid carbon/E-glass/PP non-crimp thermoplastic composites. The effect of hybridization on energy absorption of composites was not significant, while C-scan results showed that the intra-ply hybrid non-crimp thermoplastic composites had smaller impact damage areas in comparison to the non-hybrid samples. Compression and compression after impact tests results confirmed that the intra-ply hybridization increased the toughness of the composite laminates. Also, the residual compression strength/modulus increased with hybridization which indicated to damage tolerance.


2013 ◽  
Vol 106 ◽  
pp. 96-103 ◽  
Author(s):  
T.A. Sebaey ◽  
E.V. González ◽  
C.S. Lopes ◽  
N. Blanco ◽  
J. Costa

2013 ◽  
Vol 106 ◽  
pp. 30-32 ◽  
Author(s):  
T.A. Sebaey ◽  
E.V. González ◽  
C.S. Lopes ◽  
N. Blanco ◽  
J. Costa

2013 ◽  
Vol 101 ◽  
pp. 255-264 ◽  
Author(s):  
T.A. Sebaey ◽  
E.V. González ◽  
C.S. Lopes ◽  
N. Blanco ◽  
P. Maimí ◽  
...  

Author(s):  
Masafumi Hayashi ◽  
Kenji Keyamura ◽  
Asami Yoshida ◽  
Mariko Ariyoshi ◽  
Genki Akanuma ◽  
...  

In eukaryotes, genomic DNA is packaged into nucleosomes, which are the basal components coordinating both the structures and functions of chromatin. Here we screened a collection of mutation for histone H3/H4 mutants in Saccharomyces cerevisiae that affect the DNA damage sensitivity of DNA damage tolerance (DDT)-deficient cells. We identified a class of histone H3/H4 mutations that suppress MMS sensitivity of DDT-deficient cells (hereafter we refer to as the histone SDD mutations), which likely cluster on a specific H3-H4 interface of the nucleosomes. The histone SDD mutations did not suppress the MMS sensitivity of DDT-deficient cells in the absence of Rad51, indicating that homologous recombination (HR) is responsible for DNA damage resistance. Furthermore, the histone SDD mutants showed reduced levels of PCNA ubiquitination after exposure to MMS or UV irradiation, consistent with a decreased MMS-induced mutagenesis relative to wild-type cells. We also found that histone SDD mutants lacking the INO80 chromatin remodeler impair HR-dependent recovery from MMS-induced replication arrest, resulting in defective S-phase progression and increased Rad52 foci. Taken together, our data provide novel insights into nucleosome functions, which link INO80-dependent chromatin remodeling to the regulation of DDT and HR during the recovery from replication blockage.


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