scholarly journals How Income from Parental Contribution Helps Young Hungarians Become Consumers

2019 ◽  
Vol 15 (Special Issue) ◽  
pp. 473-492
Author(s):  
Szilárd Egyed
Keyword(s):  
Parental Contribution

scholarly journals Genetics, molar pregnancies and medieval ideas of monstrous births: the lump of flesh in The King of Tars

2018 ◽  
Vol 45 (1) ◽  
pp. 2-9
Author(s):  
Natalie Goodison ◽  
Deborah J G Mackay ◽  
I Karen Temple
Keyword(s):  
Hydatidiform Mole ◽  
Seed Transmission ◽  
Warning Sign ◽  
Parental Contribution ◽  
Medieval Text ◽  
Hydatidiform Moles

The medieval English romance The King of Tars gives an account of a birth of a lump of flesh. This has been considered as fantastic and monstrous in past literature, the horrific union of a Christian and Saracen. However, while the text certainly speaks to miscegenation, we propose that this lump of flesh is actually a hydatidiform mole. We trace the hydatidiform mole from antiquity, surrounding it with contextual medieval examples, from theology, history and medicine, that also describe abnormal births as ‘lumps of flesh’. By discussing medieval ideas of monsters as a warning sign, we interpret the lump of flesh in terms of abnormal births, seed transmission, parental contribution and sin. Ideas of warning, blame and intervention present themselves as a response to moles both in medieval texts as well as in modern reactions to hydatidiform moles. We explore the epigenetics of hydatidiform moles and relate them to the medieval text. In The King of Tars, the fault for the lump of flesh could reside with either parent; we find that this is also the case in the genetic formation of the hydatidiform mole; we also argue that the epigenetics supports medieval theories of seed transmission.

A maternal factor affecting mouse blastocyst formation

Development ◽  
1994 ◽  
Vol 120 (4) ◽  
pp. 797-802 ◽  
Author(s):  
J.P. Renard ◽  
P. Baldacci ◽  
V. Richoux-Duranthon ◽  
S. Pournin ◽  
C. Babinet
Keyword(s):  
Direct Evidence ◽  
Normal Development ◽  
Functional Inequality ◽  
Preimplantation Embryos ◽  
Mouse Blastocyst ◽  
Maternal Control ◽  
Paternal Genome ◽  
Functional Differences ◽  
Embryonic Genome ◽  
Parental Contribution

Normal development of the mouse embryo requires the presence of both paternal and maternal genomes. This is due to functional differences having their origin in a differential imprinting of parental genomes. Furthermore, several lines of evidence show that the very early interactions between egg cytoplasm and pronuclei may influence the programming of the embryonic genome and modulate the functional inequality of the parental contribution even during preimplantation stages. In this paper, we show that a factor present in ovulated oocytes of the mouse mutant strain DDK and therefore of maternal origin prevents the formation of the blastocyst. This factor, which acts via an interaction with the paternal genome, is present in oocytes as an RNA and is still active in preimplantation embryos. This is the first direct evidence of such a maternal control in the mouse.

Variance of Marker Estimates of Parental Contribution to F2 and BC1 -Derived Inbreds

Crop Science ◽  
2000 ◽  
Vol 40 (3) ◽  
pp. 659-665 ◽  
Author(s):  
Jiankang Wang ◽  
Rex Bernardo
Keyword(s):  
Parental Contribution

scholarly journals The parental contribution to the parent/health visitor relationship

2016 ◽  
Vol 4 (1) ◽  
pp. 48-55 ◽  
Author(s):  
Christine Bidmead ◽  
Sarah Cowley ◽  
Patricia Grocott
Keyword(s):  
Health Visitor ◽  
Parental Contribution ◽  
Parent Health

Parental contribution and coefficient of coancestry among maize inbreds: pedigree, RFLP, and SSR data

2000 ◽  
Vol 100 (3-4) ◽  
pp. 552-556
Author(s):  
R. Bernardo ◽  
J. Romero-Severson ◽  
J. Hauser ◽  
R. W. Doerge ◽  
G. Hookstra ◽  
...  
Keyword(s):  
Parental Contribution ◽  
Maize Inbreds ◽  
Coefficient Of Coancestry

Parental contribution analysis in hybrids bred for vascular streak dieback (VSD) disease resistance in cocoa

2016 ◽  
Vol 44 (1) ◽  
Author(s):  
J.S. Minimol, B. Suma, Mahiya Ummer P.A. Jayasree
Keyword(s):  
Disease Resistance ◽  
Relative Efficiency ◽  
Molecular Breeding ◽  
Disease Outbreak ◽  
Breeding Programme ◽  
Selection Differential ◽  
Chemical Application ◽  
Transfer Resistance ◽  
Parental Contribution ◽  
Mother Plants

<p>Vascular streak die back (VSD) disease caused by <em>Oncobasidium theobromae </em>(now renamed as <em>Ceratobasidium theobromae</em>), is one of the major threats in cocoa growing countries, mainly becauseit cannot be controlled by any chemical application. In India, this disease outbreak took place during 1990 and it became an unmanageable problem of cocoa trees. Knowing the importance of resistant hybrids for controlling this disease, Kerala Agricultural University took up resistant breeding programme. It was a massive breeding programme which resulted in production of 5921 hybrid seedlings. After nursery screening of these hybrids by subjecting to heavy inoculation load for more than two years from March 1996 to June 1998, only 566 seedlings survived and they were field planted during June 1998. When relative efficiency of mother plants to transfer resistance to their progenies was estimated, it was seen that G VI 126 (Scavina 6) showed maximum recovery of 32.6 per cent after fifteen years of screening. Comparison of efficacy of male parents indicated that G VI 55 showed maximum recovery of resistant parents (168 nos.). During the entire period of investigation, 267 hybrids were found free from the disease. Out of this, only 51 recorded satisfactory yield levels. All these hybrids showed high value for selection differential (S) indicating maximum genetic gain.These hybrids can be used as mother plants for establishing polyclonal garden, as VSD disease resistance is highly heritable and polygenically controlled. The resistant hybrids identified (both high and low yield) can be utilized for further studies employing conventional and molecular breeding methods.</p>

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