scholarly journals Response to Comment on Sjöholm et al. Weight Change–Adjusted Effects of Gastric Bypass Surgery on Glucose Metabolism: 2- and 10-Year Results From the Swedish Obese Subjects (SOS) Study. Diabetes Care 2016;39:625–631

Diabetes Care ◽  
2016 ◽  
Vol 39 (6) ◽  
pp. e85-e85
Author(s):  
Kajsa Sjöholm ◽  
Lena M.S. Carlsson ◽  
Markku Peltonen
Keyword(s):  
Gastric Bypass ◽  
Glucose Metabolism ◽  
Weight Change ◽  
Diabetes Care ◽  
Bypass Surgery ◽  
Gastric Bypass Surgery ◽  
Obese Subjects

Long-Term Effects of Sleeve Gastrectomy and Roux-en-Y Gastric Bypass Surgery on Type 2 Diabetes Mellitus in Morbidly Obese Subjects

2012 ◽  
Vol 256 (6) ◽  
pp. 1023-1029 ◽  
Author(s):  
Amanda Jiménez ◽  
Roser Casamitjana ◽  
Lílliam Flores ◽  
Judith Viaplana ◽  
Ricard Corcelles ◽  
...  
Keyword(s):  
Diabetes Mellitus ◽  
Type 2 Diabetes ◽  
Gastric Bypass ◽  
Bypass Surgery ◽  
Gastric Bypass Surgery ◽  
Morbidly Obese ◽  
Long Term Effects ◽  
Obese Subjects

Plasma amino acids, adiposity, and weight change after gastric bypass surgery: are amino acids associated with weight regain?

2017 ◽  
Vol 57 (7) ◽  
pp. 2629-2637 ◽  
Author(s):  
Susanna E. Hanvold ◽  
Kathrine J. Vinknes ◽  
Nasser E. Bastani ◽  
Cheryl Turner ◽  
Elin B. Løken ◽  
...  
Keyword(s):  
Amino Acids ◽  
Gastric Bypass ◽  
Weight Change ◽  
Weight Regain ◽  
Bypass Surgery ◽  
Gastric Bypass Surgery ◽  
Plasma Amino Acids

scholarly journals Vasoreactivity is rapidly improved in obese subjects after gastric bypass surgery

2009 ◽  
Vol 33 (12) ◽  
pp. 1390-1395 ◽  
Author(s):  
L Lind ◽  
B Zethelius ◽  
M Sundbom ◽  
B Edén Engström ◽  
F A Karlsson
Keyword(s):  
Gastric Bypass ◽  
Bypass Surgery ◽  
Gastric Bypass Surgery ◽  
Obese Subjects

Circulating Peptide YY, Weight Loss, and Glucose Homeostasis After Gastric Bypass Surgery in Morbidly Obese Subjects

2008 ◽  
Vol 247 (2) ◽  
pp. 270-275 ◽  
Author(s):  
Rosa Morínigo ◽  
Josep Vidal ◽  
Antonio M. Lacy ◽  
Salvadora Delgado ◽  
Roser Casamitjana ◽  
...  
Keyword(s):  
Weight Loss ◽  
Gastric Bypass ◽  
Glucose Homeostasis ◽  
Bypass Surgery ◽  
Peptide Yy ◽  
Gastric Bypass Surgery ◽  
Morbidly Obese ◽  
Obese Subjects

Follistatin secretion is enhanced by protein, but not glucose or fat ingestion in obese persons independently of previous gastric bypass surgery

2021 ◽  
Author(s):  
Kirstine N Bojsen-Møller ◽  
Maria S Svane ◽  
Christian Zinck Jensen ◽  
Sasha A.S. Kjeldsen ◽  
Jens J. Holst ◽  
...  
Keyword(s):  
Gastric Bypass ◽  
Protein Intake ◽  
Bypass Surgery ◽  
Gastric Bypass Surgery ◽  
Activin A ◽  
Strong Stimulus ◽  
Postoperative Changes ◽  
Glucose Intake ◽  
Obese Subjects ◽  
The Individual

Follistatin is secreted from the liver and is involved in the regulation of muscle mass and insulin sensitivity via inhibition of activin A in humans. The secretion of follistatin seems to be stimulated by glucagon and inhibited by insulin, but only limited knowledge on the postprandial regulation of follistatin exists. Moreover, results on postoperative changes after Roux-en-Y gastric bypass (RYGB) are conflicting with reports of increased, unaltered and lowered fasting concentrations of follistatin. In this study, we investigated postprandial follistatin and activin A concentrations after intake of isocaloric amounts of protein, fat or glucose in obese subjects with and without previous RYGB to explore the regulation of follistatin by the individual macronutrients. Protein intake enhanced follistatin concentrations similarly in the two groups, while glucose and fat ingestion did not change postprandial follistatin concentrations. Concentrations of activin A were lower after protein intake compared with glucose intake in RYGB. Glucagon concentrations were also particularly enhanced by protein intake and tended to correlate with follistatin in RYGB. In conclusion, we demonstrated that protein intake, but not glucose or fat, is a strong stimulus for follistatin secretion in obese subjects and that this regulation is maintained after RYGB surgery.

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