Occupational and Environmental Lung Diseases Selected Pneumoconiosis

2017 ◽  
Author(s):  
Cora S Sack ◽  
Sverre Vedal ◽  
Joel D Kaufman

Environmental and occupational lung diseases encompass a diverse group of lung diseases caused by the inhalation of potentially harmful substances. This review provides an in-depth discussion of the pneumoconioses, hypersensitivity pneumonitis, chronic beryllium disease, and other occupational lung diseases that affect the parenchyma. For each disease, the review presents the epidemiology, biologic mechanisms when known, diagnosis, and clinical care. The review includes tables with different pneumoconioses, occupations and industries associated with silicosis and asbestosis, and some selected causes of hypersensitivity pneumonitis, as well as chest radiographs, computed tomographic scans, and/or pathologic slides of some selected diseases. This review contains 9 figures, 4 tables, and 68 references. Key words: asbestosis, asbestos-related pleural plaque, coal worker’s pneumoconiosis, chronic beryllium disease, hard metal pneumoconiosis, hypersensitivity pneumonitis, pneumoconiosis, progressive massive fibrosis, silicosis

2017 ◽  
Author(s):  
Cora S Sack ◽  
Sverre Vedal ◽  
Joel D Kaufman

Environmental and occupational lung diseases encompass a diverse group of lung diseases caused by the inhalation of potentially harmful substances. This review provides an in-depth discussion of the pneumoconioses, hypersensitivity pneumonitis, chronic beryllium disease, and other occupational lung diseases that affect the parenchyma. For each disease, the review presents the epidemiology, biologic mechanisms when known, diagnosis, and clinical care. The review includes tables with different pneumoconioses, occupations and industries associated with silicosis and asbestosis, and some selected causes of hypersensitivity pneumonitis, as well as chest radiographs, computed tomographic scans, and/or pathologic slides of some selected diseases. This review contains 9 figures, 4 tables, and 68 references. Key words: asbestosis, asbestos-related pleural plaque, coal worker’s pneumoconiosis, chronic beryllium disease, hard metal pneumoconiosis, hypersensitivity pneumonitis, pneumoconiosis, progressive massive fibrosis, silicosis


Author(s):  
Gareth Walters ◽  
Justin Mokhlis ◽  
Vicky Moore ◽  
Geraldine Burge ◽  
Alastair Robertson ◽  
...  

2021 ◽  
Vol 30 (160) ◽  
pp. 210076
Author(s):  
Iain R. Konigsberg ◽  
Lisa A. Maier ◽  
Ivana V. Yang

Epigenetic modifications are emerging as important regulatory mechanisms of gene expression in lung disease, given that they are influenced by environmental exposures and genetic variants, and that they regulate immune and fibrotic processes. In this review, we introduce these concepts with a focus on the study of DNA methylation and histone modifications and discuss how they have been applied to lung disease, and how they can be applied to sarcoidosis. This information has implications for other exposure and immunologically mediated lung diseases, such as chronic beryllium disease, hypersensitivity pneumonitis, and asbestosis.


2014 ◽  
pp. 473-491
Author(s):  
Joachim Müller-Quernheim ◽  
Gernot Zissel ◽  
Gian Kayser ◽  
Antje Prasse

2020 ◽  
Vol 6 (3) ◽  
pp. 79
Author(s):  
Ariani Permatasari ◽  
Reagen Irwan Kolibu

Indonesia is one of the third largest rice producers globally; therefore, rice dust-related lung diseases will be more significant. Rice dust is easily exposed to humans, triggers lung damage, and decline the lung function. Rice dust exposure is associated with occupational lung diseases, for instance, acute reversible airflow obstruction, chronic obstructive pulmonary disease, grain fever, organic dust syndrome, and hypersensitivity pneumonitis. Smoking and the environment might aggravate rice dust-related respiratory problems. The average tolerated dose of rice dust is about less than 3 mg/m3. Avoiding exposure to rice dust is primer prevention.


Sign in / Sign up

Export Citation Format

Share Document