Experimental Transmission of the Rickettsiae of the Spotted Fevers of Brazil, Colombia, and the United States by the Argasid Tick Ornithodoros nicollei

1943 ◽  
Vol 58 (48) ◽  
pp. 1742 ◽  
Author(s):  
Gordon E. Davis
1931 ◽  
Vol 46 (42) ◽  
pp. 2481 ◽  
Author(s):  
R. E. Dyer ◽  
E. T. Ceder ◽  
R. D. Lillie ◽  
A. Rumreich ◽  
L. F. Badger

2005 ◽  
Vol 17 (1) ◽  
pp. 3-9 ◽  
Author(s):  
Amir N. Hamir ◽  
Robert A. Kunkle ◽  
Juergen A. Richt ◽  
Janice M. Miller ◽  
Randall C. Cutlip ◽  
...  

Parasitology ◽  
1933 ◽  
Vol 24 (4) ◽  
pp. 569-573 ◽  
Author(s):  
Charles W. Rees

It has been determined that anaplasmosis may be transmitted by Dernmcentor andersoni under the following conditions: (1) Larvae may acquire the infection by engorging on a bovine whose blood contains anaplasms and may transmit the infection as nymphs to susceptible bovines; and (2) in a similar manner the nymphs may acquire the infection and transmit it as adults. The test of “hereditary” transmission was negative, i.e. the larvae did not apparently acquire the infection from adult females which engorged on carriers of anaplasmosis. Conditions of transmission by D. andersoni are the same as those which have thus far been determined by me for D. variabilis. The incrimination of these two species of tick points to the possibility that anaplasmosis occurring anywhere within the known range of distribution of that disease in the United States may be tick-borne so far as a coincident distribution of known tick carriers can establish this possibility.


1997 ◽  
Vol 34 (3) ◽  
pp. 250-252 ◽  
Author(s):  
A. N. Hamir ◽  
J. R. Heidel ◽  
R. Picton ◽  
C. E. Rupprecht

Microscopic vacuolar changes in neuronal perikaryon are described in two free-ranging raccoons ( Procyon lotor) from different geographic locations in the United States. Both animals were negative for rabies and scrapie-associated antigens. Microscopically, lesions were not seen in the neuropil. Neuronal vacuolations have previously been documented in brains of normal animals and in diseases such as rabies and prion-associated encephalopathies. Although experimental transmission of a spongiform mink encephalopathy has been documented in raccoons, a naturally occurring spongiform encephalopathy has not been described in this species. The presence of neuronal vacuolations in the raccoons is novel and requires further investigation to elucidate the mechanism of this phenomenon.


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