"The Relationship of Tubal Infertility to Barrier Method and Oral Contraceptive Use"

1988 ◽  
Vol 19 (6) ◽  
pp. 386
Author(s):  
Daniel W. Cramer ◽  
Marlene B. Goldman ◽  
Isaac Schiff ◽  
Serge Belisle ◽  
Bruce Albrecht ◽  
...  
Epidemiology ◽  
1990 ◽  
Vol 1 (2) ◽  
pp. 153-156 ◽  
Author(s):  
Mark A. Moskowitz ◽  
Susan S. Jick ◽  
Susan Burnside ◽  
Wayne Jack Wallis ◽  
John F. Dickson ◽  
...  

1999 ◽  
Vol 87 (1) ◽  
pp. 381-385 ◽  
Author(s):  
Nisha Charkoudian ◽  
John M. Johnson

To determine whether cardiovascular influences of exogenous female steroid hormones include effects on reflex thermoregulatory control of the adrenergic cutaneous vasoconstrictor system, we conducted ramp decreases in skin temperature (Tsk) in eight women in both high- and low (placebo)-progesterone/estrogen phases of oral contraceptive use. With the use of water-perfused suits, Tsk was held at 36°C for 10 min (to minimize initial vasoconstrictor activity) and was then decreased in a ramp, ∼0.2°C/min for 12–15 min. Subjects rested supine for 30–40 min before each experiment, and the protocol was terminated before the onset of shivering. Skin blood flow was monitored by laser-Doppler flowmetry and arterial pressure by finger photoplethysmography. In all experiments, cutaneous vasoconstriction began immediately with the onset of cooling, and cutaneous vascular conductance (CVC) decreased progressively with decreasing Tsk. Regression analysis of the relationship of CVC to Tsk showed no difference in slope between phases (low-hormone phase: 17.67 ± 5.57; high-hormone phase: 17.40 ± 8.00 %baseline/°C; P > 0.05). Additional studies involving local blockade confirmed this response as being solely due to the adrenergic vasoconstrictor system. Waking oral temperature (Tor) was significantly higher on high-hormone vs. low-hormone days (36.60 ± 0.11 vs. 36.37 ± 0.09 °C, respectively; P < 0.02). Integrative analysis of CVC in terms of simultaneous values for Tsk and Tor showed that the cutaneous vasoconstrictor response was shifted in the high-hormone phase such that a higher Tor was maintained throughout cooling ( P < 0.05). Thus reflex thermoregulatory control of the cutaneous vasoconstrictor system is shifted to higher internal temperatures by exogenous female reproductive hormones.


Cancer ◽  
1993 ◽  
Vol 71 (S4) ◽  
pp. 1506-1517 ◽  
Author(s):  
Phyllis A. Wingo ◽  
Nancy C. Lee ◽  
Howard W. Ory ◽  
Valerie Beral ◽  
Herbert B. Peterson ◽  
...  

Drug Safety ◽  
1990 ◽  
Vol 5 (6) ◽  
pp. 436-446 ◽  
Author(s):  
Carlo La Vecchia ◽  
Silvia Franceschi ◽  
Paolo Bruzzi ◽  
Fabio Parazzini ◽  
Peter Boyle

Cephalalgia ◽  
1995 ◽  
Vol 15 (2) ◽  
pp. 140-144 ◽  
Author(s):  
LM Cupini ◽  
M Matteis ◽  
E Troisi ◽  
P Calabresi ◽  
G Bernardi ◽  
...  

In this study, the relationship between hormonal-related events and migraine with aura (MA) and without aura (MO) was investigated. Subjects included 268 women suffering from MA (88) and MO (180). Data were collected on the relationship between sex-hormone-related events and migraine. Migraine during menses was observed in a significantly higher percentage of MO than MA patients ( p < 0.03). Menstrual migraine was significantly more common in MO than in MA patients ( p < 0.01). Migraine began during pregnancy in a significantly higher percentage of MA than of MO patients ( p < 0.01). No significant difference was observed between the two groups of patients regarding the onset of migraine at menarche, after menopause, in the postpartum period or during the early cycles of oral contraceptives. Also, both groups of patients showed a similar migraine course during pregnancy, oral contraceptive use and menopause. Eight patients with coexisting migraine with aura and migraine without aura attacks reported the appearance of the aura symptom for the first time in the early cycles of oral contraceptive intake. These findings suggest that gonadal hormone fluctuation may influence both types of migraine.


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