The Fauna of Four Streams in the `Black Mountain' District of South Wales

10.2307/1609 ◽  
1948 ◽  
Vol 17 (1) ◽  
pp. 51 ◽  
Author(s):  
J. R. Erichsen Jones
Keyword(s):  
South Wales ◽  
Black Mountain

Persistent Places in the Mesolithic Landscape: an Example from the Black Mountain Uplands of South Wales

1995 ◽  
Vol 61 ◽  
pp. 81-116 ◽  
Author(s):  
R.N.E. Barton ◽  
P.J. Berridge ◽  
M.J.C. Walker ◽  
R.E. Bevins
Keyword(s):  
Lake Basin ◽  
Site Location ◽  
Long Distance ◽  
Hunter Gatherers ◽  
Mountain Ranges ◽  
Long Distance Transport ◽  
South Wales ◽  
Long Time ◽  
The Relationship ◽  
Black Mountain

Evidence for the early Postglacial use of upland environments in the Mesolithic in various parts Britain has been known for a long time. However, until relatively recently such evidence had been remarkably absent from upland south Wales, which includes some of the highest mountain ranges in southern Britain. In this paper we report on new work at the upland location of Waun Fignen Felen which consists of discrete Early and later Mesolithic artefact scatters on the edge of a former lake basin. In describing this example, we focus on the timing of Mesolithic movements into the interior uplands and examine the relationship between humans and the landscape, particularly in respect to the long distance transport of materials and factors likely to have influenced the choice of site location. Some comparative observations are made on the use and perception of landscapes by ethnographic hunter-gatherers.

β-Mannosidosis: Myelin and oligodendrocyte lesions in brain and spinal cord

1984 ◽  
Vol 42 ◽  
pp. 694-695
Author(s):  
Kathryn L. Lovell ◽  
Margaret Z. Jones
Keyword(s):  
Spinal Cord ◽  
Nervous System ◽  
Genetic Disorder ◽  
Neurological Deficits ◽  
Axonal Spheroids ◽  
South Wales ◽  
Pendular Nystagmus ◽  
Myelin Deficits ◽  
Recessive Defect ◽  
Brain And Spinal Cord

Caprine β-mannosidosis, an autosomal recessive defect of glycoprotein catabolism, is associated with a deficiency of tissue and plasma -mannosidase and with tissue accumulation and urinary excretion of oligosaccharides, including the trisaccharide Man(β1-4)GlcNAc(βl-4)GlcNAc and the disaccharide Man(β1-4)GlcNAc. This genetic disorder is evident at birth, with severe neurological deficits including a marked intention tremor, pendular nystagmus, ataxia and inability to stand. Major pathological characteristics described in Nubian goats in Michigan and in Anglo-Nubian goats in New South Wales include widespread cytoplasmic vacuolation in the nervous system and viscera, axonal spheroids, and severe myelin paucity in the brain but not spinal cord or peripheral nerves. Light microscopic examination revealed marked regional variation in the severity of central nervous system myelin deficits, with some brain areas showing nearly complete absence of myelin and other regions characterized by the presence of 25-50% of the control number of myelin sheaths.

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