Sequence of Precursor Polyprotein Gene (Segment A) of Infectious Bursal Disease Viruses Isolated in Korea

2000 ◽  
Vol 44 (3) ◽  
pp. 691 ◽  
Author(s):  
Hyuk Moo Kwon ◽  
Dae Kyu Kim ◽  
Tae Wook Hahn ◽  
Jeong Hee Han ◽  
Daral J. Jackwood
Author(s):  
Phạm Hồng Sơn ◽  
Phạm Hồng Kỳ ◽  
Nguyễn Thị Lan Hương ◽  
Phạm Thị Hồng Hà

. Using the method of shifting assay of standardized indirect agglutination (SSIA), the prevalence of Newcastle disease viruses (NDV) and infectious bursal disease viruses (IBDV) in chickens reared in several districts of Thua Thien Hue province in the Spring-Summer and Fall-Winter seasons was determined. In the Spring-Summer season of 2011, about 22.3% of the chickens were infected with NDV, in which A Luoi  accounted for the highest percentage of 25% of the infected chickens and Huong Thuy  the lowest  of 18.2%. Meanwhile, 36% of the same chickens were infected with IBDV, with the highest percentage (46.66%) also in A Luoi and the lowest (30.3%) also in Huong Thuy. The intensity of NDV infection in the Spring-Summer season in A Luoi and Phu Vang was highest (GMT = 1.45), and in Huong Thuy lowest (GMT = 1.31). In addition, in the Fall-Winter season, about 46% of the chickens were infected with NDV and 46.3% with IBDV in Huong Thuy and Phu Vang – two neighbouring districts of Hue City, in which NDV was detected in 54.4% of the chickens in Huong Thuy and 33.9% in Phu Vang. In contrast, IBDV was detected in 41.9% and 52.7% of the chickens respectively in the two districts. The infection was not inter-dependent. Methodically, although the differences in the infection rates were insignificant with the accuracy of 95%, faecal samples showed higher sensitivity in SSIA analyses for both cases of NDV and IBDV infection in comparision with mouth exudates. By SSIA method, results could be read clearly with unaided eyes for a long time after the performance, and it was also proven applicable for cases of haemagglutinating viruses if proper treatments for depletion of animal RBCs’ surface agglutinins could be applied.


2001 ◽  
Vol 45 (2) ◽  
pp. 297 ◽  
Author(s):  
N. Ikuta ◽  
J. El-Attrache ◽  
P. Villegas ◽  
M. Garcia ◽  
V. R. Lunge ◽  
...  

2013 ◽  
Vol 42 (4) ◽  
pp. 309-315
Author(s):  
Vijay Durairaj ◽  
Erich Linnemann ◽  
Alan H. Icard ◽  
Susan M. Williams ◽  
Holly S. Sellers ◽  
...  

2007 ◽  
Vol 152 (10) ◽  
pp. 1787-1797 ◽  
Author(s):  
Y. S. Wang ◽  
Z. C. Wang ◽  
Y. D. Tang ◽  
Z. L. Shi ◽  
K. W. He ◽  
...  

1978 ◽  
Vol 15 (3) ◽  
pp. 376-382 ◽  
Author(s):  
N. F. Cheville ◽  
W. Okazaki ◽  
P. D. Lukert ◽  
H. G. Purchase

Five groups of genetically susceptible chickens were inoculated at hatching with lymphoid leukosis virus; four of these were given infectious bursal viruses of varying virulence at 14 days of age and one group was not inoculated (control). All chickens in the control group developed evidence of lymphoid leukosis by 180 days. Two groups given relatively virulent bursal disease viruses, which destroyed bursal lymphoid cells, did not develop lymphoid leukosis. Treatment with avirulent vaccines had no visible effect on bursal morphology and did not significantly alter the incidence of lymphoid leukosis in two other groups, although the lime of development was delayed. Results of our study show that viral-induced destruction of the bursa of Fabricius eliminates the development of lymphoid leukosis but that infection without bursal destruction has little effect on lymphoid leukosis.


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