scholarly journals THE EFFECTS OF FOOD DEPRIVATION AND SALINITY CHANGES ON REPRODUCTIVE FUNCTION IN THE ESTUARINE GOBIID FISH,GILLICHTHYS MIRABILIS

1971 ◽  
Vol 141 (3) ◽  
pp. 458-471 ◽  
Author(s):  
VICTOR L. DE VLAMING
Keyword(s):  
Food Deprivation ◽  
Reproductive Function ◽  
Gobiid Fish ◽  
Gillichthys Mirabilis ◽  
Salinity Changes

Interactive effects of food deprivation and exercise on reproductive function in female hamsters

1994 ◽  
Vol 267 (1) ◽  
pp. R185-R190 ◽  
Author(s):  
J. B. Powers ◽  
A. E. Jetton ◽  
G. N. Wade
Keyword(s):  
Food Deprivation ◽  
Reproductive Function ◽  
Hormonal Treatment ◽  
Estradiol Benzoate ◽  
Interactive Effects ◽  
Combined Effects ◽  
Inhibitory Effects ◽  
Estrous Cycles ◽  
Running Wheels ◽  
Estrous Behavior

Two experiments evaluated the combined effects of food deprivation and runningwheel access on estrous cycles and estrous behavior of female hamsters. In experiment 1, food deprivation on days 1 and 2 of the estrous cycle disrupted the next expected ovulation, and this effect was more, rather than less, robust in females allowed to exercise in running wheels while they were deprived. In experiment 2, a similar protocol was used except the females were ovariectomized and received sequential injections of estradiol benzoate (EB; 5 micrograms) and progesterone (P; 200 micrograms) separated by 48 h to induce lordosis, which was tested 4-5 after P. Food deprivation concomitant with hormonal treatment diminished lordosis durations, but this effect was significant only among the females that were permitted to run in activity wheels. Previous findings demonstrated that access to running wheels attenuated the inhibitory effects of short photoperiod exposure on hamster estrous cycles. In contrast, the present results indicate that this same manipulation exaggerates rather than diminishes the inhibitory effects of food deprivation on estrous cycles and hormone-induced behavioral estrus.

Reproductive Cycling in the Estuarine Gobiid Fish, Gillichthys mirabilis

Copeia ◽  
1972 ◽  
Vol 1972 (2) ◽  
pp. 278 ◽  
Author(s):  
Victor L. de Vlaming
Keyword(s):  
Gobiid Fish ◽  
Gillichthys Mirabilis

scholarly journals ENDOCRINE INFLUENCES ON SEMINAL VESICLES IN THE ESTUARINE GOBIID FISH,GILLICHTHYS MIRABILIS

1972 ◽  
Vol 142 (2) ◽  
pp. 243-250 ◽  
Author(s):  
VICTOR L. DE VLAMING ◽  
BANGALORE I. SUNDARARAJ
Keyword(s):  
Seminal Vesicles ◽  
Gobiid Fish ◽  
Gillichthys Mirabilis

Effects of leptin administration on lactational infertility in food-restricted rats depend on milk delivery

2004 ◽  
Vol 286 (1) ◽  
pp. R217-R225 ◽  
Author(s):  
Alfonso Abizaid ◽  
Diana Kyriazis ◽  
Barbara Woodside
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Milk Production ◽  
Food Deprivation ◽  
Food Restriction ◽  
Reproductive Function ◽  
Time Period ◽  
Lactating Females ◽  
The Central Nervous System ◽  
Direct Binding

Leptin administration has been shown to prevent the disruptive effects of acute food deprivation on reproductive function in cycling females and lactating females. We examined the ability of intracerebroventricular leptin administration to ameliorate the effects of food restriction for the first 2 wk postpartum on length of lactational infertility. Leptin administration did not reduce the effects of food restriction on reproductive function at either time period ( days 8-15 and 15-22 postpartum) or dose (1 and 10 μg/day) administered. Because of the sharp contrast between these results and the ability of leptin to offset the effects of acute food deprivation in lactating rats, the remaining studies investigated the possible causes of this difference. Both central and peripheral leptin administration eliminated food deprivation-induced prolongation of lactational infertility, suggesting that neither route of administration nor dose was a factor. However, we noticed that, whereas chronically food-restricted females continue to deliver milk to their young, acutely food-deprived females do not. To test the hypothesis that the continued energetic drain of milk production and delivery might prevent the ability of exogenous leptin administration to eliminate the effects of undernutrition, leptin was administered to food-restricted, lactating rats prevented from delivering milk. In this situation intracerebroventricular leptin treatment completely eliminated the effects of food restriction on lactational infertility, suggesting that leptin contributes to the maintenance of reproductive function via two pathways: direct binding in the central nervous system and through increasing the availability of oxidizable metabolic fuels.

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