scholarly journals The quality of voice and speech before and after surgical treatment of bilateral recurrent laryngeal nerve paralysis

2017 ◽  
Vol 70 (5-6) ◽  
pp. 146-149
Author(s):  
Maja Bogdan ◽  
Rajko Jovic ◽  
Tanja Arbutina

Introduction. Bilateral recurrent laryngeal nerve paralysis usually occurs after thyroid surgery. In bilateral vocal cord paralysis, the voice is clear or slightly hoarse. The aim of this study was to determine whether the quality of voice and speech significantly deteriorates after the surgical treatment of bilateral recurrent laryngeal nerve paralysis. Material and Methods. The study included 16 patients with bilateral vocal cord paralysis and 16 age- and sex-matched healthy controls. The patients underwent partial arytenoidectomy with posterior cordectomy at the Clinical Center of Vojvodina in the period from January to April 2014. The quality of voice and speech was determined before and after surgical treatment by subjective, objective, aerodynamic voice analysis, and analysis of spontaneous speech, and then compared to the control group. Results. The results of this study showed that in patients with bilateral vocal cord paralysis the voice and speech quality was significantly worse compared to the healthy subjects. The results of subjective and aerodynamic analysis showed that there was a statistically significant deterioration in voice quality after the surgical treatment (p<0.05; p=0.001). The values of objective analysis and analysis of spontaneous speech parameters did not significantly change after the surgery (p>0.05; p=0.401). Conclusion. The patients with bilateral vocal cord paralysis have a poorer voice and speech quality compared to the healthy subjects. After the surgical treatment, patients presented with a lower voice quality, but there were no significant changes regarding the ability of spontaneous speech.

2018 ◽  
Vol 31 (Supplement_1) ◽  
pp. 103-103
Author(s):  
Fumiaki Kawano ◽  
Shinsuke Takeno ◽  
Kousei Tashiro ◽  
Rouko Hamada ◽  
Yasuyuki Miyazaki ◽  
...  

Abstract Background Recurrent laryngeal nerve paralysis in esophagectomy is one of the most concerned complications. In recent years, intraoperative neurostimulation monitoring system (IONM) in thyroid surgery have been widespread for identification of recurrent laryngeal nerve and assessment of soundness. Therefore, IONM is often used during esophagectomy in Japan. In this study, we examined the efficacy of IONM in the patients undergoing esophagectomy. Methods Of 66 patients underwent esophagectomy since April 2015 until December 2017, IONM used in 27 patients in the surgery for the examination of recurrent nerve paralysis. We retrospectively reviewed these cases for intraoperative findings, neurostimulation monitoring findings and their outcomes. Results Of 27 patients, 25 were male and two were female, and the median age at operation was 66 years old. Although IONM was used in cervical lymph node dissection, there were no vocal cord responses in 5 patients (left side in 4 and right side in 1) with stimulation of the vagus nerve. Because all patients had no vocal cord paralysis due to stimulation of the cervical recurrent laryngeal nerve, it was diagnosed that there was the recurrent laryngeal nerve injury due to thoracic para recurrent nerve lymph node dissection. IONM was able to facilitate the identification and preservation of cervical recurrent nerve in all patients. Three out of 5 patients with no vocal cord response by IONM were confirmed recurrent laryngeal nerve paralysis in postoperative endoscope. In patients with vocal cord paralysis by IONM, it was possible to carefully performed postoperative management. On the other hand, in patients without paralysis, extubation on the operation day seemed possible without the concern for aspiration. Conclusion By using IONM in esophagectomy, we were able to evaluate the damage of the recurrent laryngeal nerve in real-time. Confirming the intraoperative recurrent nerve injuries is important for postoperative management or prediction of postoperative aspiration pneumonia. IONM in esophagectomy was useful not only in terms of surgical procedures but also in the evaluation of postoperative management. Disclosure All authors have declared no conflicts of interest.


1989 ◽  
Vol 103 (6) ◽  
pp. 626-628 ◽  
Author(s):  
S. N. Krishnamurthy ◽  
K. O. Paulose

AbstractLeft recurrent laryngeal nerve runs a long intrathoracic course in close relationship to the aortic arch and adjacent heart structures and, hence, is liable to be injured by diseases affecting the heart and its great vessels.We report a case of Ebstein's anomaly causing left recurrent laryngeal nerve paralysis in a neonate. To our knowledge, this is the first reported case of vocal cord paralysis associated with this condition.


1993 ◽  
Vol 102 (10) ◽  
pp. 810-813 ◽  
Author(s):  
Yuval Zohar ◽  
Nora Buller ◽  
Ytzhak Shvilly

We present 2 patients with sudden onset of dysphonia due to acute unilateral vocal cord paralysis that occurred during transvenous insertion of a permanent endocardial pacemaker. Acute paralysis of the vocal cord due to this kind of medical intervention is a very unusual complication that has been rarely reported. We believe that traumatic insertion of a pacemaker through the subclavian vein triggered a dynamic process that led to vocal cord paralysis. Three hypothetic explanations are presented: 1) a direct vagus nerve lesion, 2) a direct inferior laryngeal nerve lesion, and 3) a central nervous system lesion caused by a cerebral microembolus. Physicians should be aware of the possibility that associated acute paralysis of the vocal cord may occur after transvenous insertion of an endocardial pacemaker.


Author(s):  
Punithakumar Ramasamy ◽  
Suresh Babu Kale ◽  
Senthilkumar Ramalingam ◽  
Murugesan Veerappa

<p>Rheumatic mitral stenosis is prevalent in the developing world and presents with a wide array of manifestations. Aphonia and hoarseness of voice secondary to recurrent laryngeal nerve paralysis (Ortner’s syndrome) is an uncommon manifestation of mitral stenosis. We present a case severe mitral stenosis with moderate to severe tricuspid valve regurgitation and severe pulmonary artery hypertension that was masquerading as aphonia and hoarseness of voice for eight months. Mitral valve replacement and tricuspid annuloplasty was done and the quality of voice improved post-surgery. </p>


1990 ◽  
Vol 100 (9) ◽  
pp. 1005???1008 ◽  
Author(s):  
??ke Geterud ◽  
Hasse Ejnell ◽  
Rune Stenborg ◽  
Bj??rn Bake

1986 ◽  
Vol 95 (1) ◽  
pp. 47-51 ◽  
Author(s):  
Randal A. Otto ◽  
William Davis

We have previously presented the concept of electrophysiologic pacing of bilaterally paralyzed vocal cord abductors as a solution to the difficult problem incurred in this clinical situation. Initially, we demonstrated that it was indeed feasible to electrophysiologically pace abduction of the vocal cords synchronously with respiration, employing the EMG activity of the diaphragm as a trigger stimulus. Further research has led us to evaluate other possible physiologic trigger stimuli to ascertain which of these will prove most suitable in long-term pacing studies. In this article, we will report our preliminary results, employing negative intrathoracic pressure occurring with respiration—as detected by an implanted pressure transducer as a trigger stimulus. This device was interfaced with a muscle stimulator attached to electrodes placed in the cricoarytenoid muscles in five canines whose recurrent laryngeal nerves had been sectioned bilaterally. In all animals, obvious physiologic synchrony of vocal cord abduction and a reduciton of negative inspiratory intratracheal pressure was achieved during electrical pacing. This reinforces our initial findings that it is indeed feasible to pace vocal cord abduction in bilaterial recurrent laryngeal nerve paralysis with resultant return of physiologic normality to the glottis. Thus, functional electrical stimulation offers an alternative approach to the difficult problems incurred in the patient with bilateral recurrent laryngeal nerve paralysis. It also demonstrates that physiologic negative intrathoracic pressure activity occurring with inspiration can be a trigger source.


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