scholarly journals Pre-administration of vitamin C reduces exercise-induced oxidative stress in untrained subjects

2014 ◽  
Vol 66 (3) ◽  
pp. 1179-1185 ◽  
Author(s):  
Florin-Petrut Trofin ◽  
Marin Chirazi ◽  
Cezar Honceriu ◽  
Paula Drosescu ◽  
Gabriela Grădinariu ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Vitamin C ◽  
Specific Activity ◽  
Serum Markers ◽  
Antioxidant Defenses ◽  
Dietary Vitamin ◽  
Bicycle Exercise ◽  
Physically Active ◽  
Markers Of Oxidative Stress ◽  
Exercise Induced

We investigated the effects of a 40 min bout of bicycle exercise, and the effects of vitamin C administration 12 h before exercise, on the serum markers of oxidative stress in young untrained subjects. Increased levels of malondialdehyde, the marker of the lipid peroxidation, and a decrease in specific activity of the antioxidant enzyme glutathione peroxidase that were observed as a result of exercise, pointed to the presence of exercise-induced oxidative stress. These markers were reduced by pre-administration of vitamin C. The results suggest that physically active subjects could increase their daily dietary vitamin C intake in order to reinforce their antioxidant defenses prior to exercise training.

Measurable serum markers of oxidative stress response in women with endometriosis

2009 ◽  
Vol 91 (1) ◽  
pp. 46-50 ◽  
Author(s):  
Irene V. Lambrinoudaki ◽  
Areti Augoulea ◽  
George E. Christodoulakos ◽  
Emmanuel V. Economou ◽  
George Kaparos ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Stress Response ◽  
Serum Markers ◽  
Oxidative Stress Response ◽  
Measurable Serum ◽  
Markers Of Oxidative Stress

Oxidants and antioxidants in exercise

1995 ◽  
Vol 79 (3) ◽  
pp. 675-686 ◽  
Author(s):  
C. K. Sen
Keyword(s):  
Oxidative Stress ◽  
Physical Activity ◽  
Exercise Training ◽  
Animal Studies ◽  
Dietary Habits ◽  
Antioxidant Defenses ◽  
Current State ◽  
Strenuous Physical Exercise ◽  
Long Duration ◽  
Exercise Induced

There is consistent evidence from human and animal studies that strenuous physical exercise may induce a state wherein the antioxidant defenses of several tissues are overwhelmed by excess reactive oxygen. A wide variety of physiological and dietary antioxidants act in concert to evade such a stress. Submaximal long-duration exercise training may augment the physiological antioxidant defenses in several tissues; however, this enhanced protection may not be sufficient to completely protect highly fit individuals from exhaustive exercise-induced oxidative stress. Regular physical activity in association with dietary habits that ensure adequate supply of a combination of appropriate antioxidants may be expected to yield desirable results. The significance of this area of research, current state of information, and possibilities of further investigation are briefly reviewed.

scholarly journals Roles of Superoxide dismutase(SOD) , Malondialdehyde (MDA), 8-iso-prostaglandinF2α ( 8-iso-PGF2α)as oxidative stress in development and progression of Brest cancer in Iraqi females patients

2020 ◽  
Vol 25 (1) ◽  
pp. 1-4
Author(s):  
Ferdous Abbas Jabir ◽  
Ahmed Sabah Shaker
Keyword(s):  
Breast Cancer ◽  
Oxidative Stress ◽  
Cancer Patients ◽  
Antioxidant Defenses ◽  
Blood Levels ◽  
P Value ◽  
Oxygen Species ◽  
Breast Cancer Patients ◽  
Markers Of Oxidative Stress ◽  
The Mean

               Oxidative stress occurs as a result of disturbance in the balance between the production of reactive oxygen species (free radicals) and antioxidant defenses. Markers of oxidative stress were measured the markers of oxidative stress in breast cancer patients after diagnosis of breast cancer and compared these plasma blood levels controls This study was conducted to three markers of oxidative stress ;these are (SOD) enzyme ,malondialdehyde (MDA)and8-iso-prostaglandinF2α plasma of patients with breast cancer and compare with controls .In this study ;  the mean MDA (ng/ml) levels for the breast cancer patients and the controls were55.91±3.31 and40.61±3.76  respectively, while the SOD (pg/ml) levels were1530.37±80.4 and1851.4 9±93.65  respectively and the 8-iso-PGF2α (ng/ml ) levels were 40.16±3.31 and 30.16±2.34  difference of the mean were statistically significant (p value <0.05).                                                                                                                       

Effects of Dietary Vitamin C and E Supplementation on Exercise-Induced Muscle Damage among Young Kelantan Weightlifters

2015 ◽  
Vol 11 (1) ◽  
pp. 41-53 ◽  
Author(s):  
SAWASH M. MOHAMMED ◽  
◽  
MOHD NIDZAM JAWIS ◽  
SUHAIR A. AHMED ◽  
OLEKSANDR KRASILSHCHIKOV ◽  
...  
Keyword(s):  
Vitamin C ◽  
Muscle Damage ◽  
Dietary Vitamin ◽  
Exercise Induced

Analysis of serum markers of oxidative stress response in women with infertility related to endometriosis

2009 ◽  
Vol 92 (3) ◽  
pp. S113
Author(s):  
J.K. Rodrigues ◽  
L.A. Dib ◽  
A.S. Melo ◽  
R.L. Cardoso ◽  
G.S. Romão ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Stress Response ◽  
Serum Markers ◽  
Oxidative Stress Response ◽  
Markers Of Oxidative Stress

Oxidative stress in colon tissue induced by vitamin E depletion

2004 ◽  
Vol 32 (6) ◽  
pp. 979-981 ◽  
Author(s):  
J.E. Drew ◽  
D.K. Mercer ◽  
C. Mayer ◽  
A.J. Farquharson ◽  
P.C. Morrice ◽  
...  
Keyword(s):  
Oxidative Stress ◽  
Vitamin E ◽  
Morphological Changes ◽  
Membrane Integrity ◽  
Dietary Vitamin ◽  
Colon Tissue ◽  
Cell Membrane Integrity ◽  
Markers Of Oxidative Stress ◽  
Lipid Soluble Antioxidant ◽  
Dietary Deficiency

Inflammatory disorders of the bowel and colon cancer are associated with elevated indices of oxidative stress. Analogous elevations in markers of oxidative stress and loss of cell-membrane integrity are also observed in the colons of rats deficient in vitamin E (D-α-tocopherol), the major lipid-soluble antioxidant in biological systems. The causal relationship between colon pathologies associated with oxidative stress and dietary deficiency in antioxidant vitamins such as vitamin E is still uncertain. Investigation of potential mechanisms by which lack of dietary vitamin E may lead to clinically relevant pathological changes in colon tissue was conducted using gene expression profiling strategies on vitamin E-sufficient and -deficient rats. Morphological changes and increased indices of lipid peroxidation were linked to vitamin E deficiency. These changes in colon tissue are potentially important in disease pathogenesis of the colon linked with oxidative stress or other direct consequences of inadequate levels of vitamin E.

scholarly journals Effects of Vitamin D and Simvastatin on Inflammatory and Oxidative Stress Markers of High-Fat Diet-Induced Obese Rats

2021 ◽  
Vol 2 (3) ◽  
pp. 39-50
Author(s):  
Abdel-Moniem A. Makhlouf ◽  
Atef M. Mahmoud ◽  
Rania G. Ibrahim ◽  
Yasmeen S. Abdel Aziz
Keyword(s):  
Oxidative Stress ◽  
Vitamin D ◽  
High Fat Diet ◽  
Male Rats ◽  
Antioxidant Defenses ◽  
Control Group ◽  
High Fat ◽  
Stress Markers ◽  
Obese Rats ◽  
Markers Of Oxidative Stress

Purpose: This study was aimed to evaluate the antioxidant and anti-inflammatory effects of vitamin D and Simvastatin (SIM) on a high-fat diet (HFD) induced-obese rats. Methods: 40 adult male rats were divided into four groups: control group, HFD, HFD + vitamin D, and HFD + SIM for 14 weeks. Vitamin D or SIM supplementation was done for the last 6 weeks. Vitamin D dosage was 500 IU/kg, while SIM dosage was 10 mg/kg. Interleukin-6 (IL-6) concentration and markers of oxidative stress including malondialdehyde (MDA), superoxide dismutase (SOD), glutathione peroxidase (GPx), and reduced glutathione(GSH) concentrations in serum were determined using ELISA kits and spectrophotometry methods, respectively. Results: Treatment with vitamin D or SIM could significantly reduce IL-6 and MDA and increases SOD, GPx activities, and GSH levels. Oxidative stress can result not only from increased ROS production but also from dysfunctional antioxidant defenses. Conclusion: From the experimental results, it was observed that SIM and vitamin D could attenuate oxidative stress and inflammation markers associated with obesity.

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