scholarly journals Relationship on the Pressure Pain Thresholds between the Masticatory Muscles and the Styloid Process Part 1. In Normal Subjects.

1998 ◽  
Vol 42 (3) ◽  
pp. 438-444 ◽  
Author(s):  
Kazuko Ishiura ◽  
Koichi Kimura ◽  
Makoto Matsushima ◽  
Che-cheng Chiang ◽  
Masahiro Tanaka ◽  
...  
2009 ◽  
Vol 88 (7) ◽  
pp. 658-662 ◽  
Author(s):  
M. Koutris ◽  
F. Lobbezoo ◽  
M. Naeije ◽  
K. Wang ◽  
P. Svensson ◽  
...  

Nociceptive substances, injected into the masseter muscle, induce pain and facilitate the jaw-stretch reflex. It is hypothesized that intense chewing would provoke similar effects. Fourteen men performed 20 bouts of 5-minute chewing. After each bout, 20 min and 24 hrs after the exercise, muscle fatigue and pain scores and the normalized reflex amplitude from the left masseter muscle were recorded. Before, 20 min, and 24 hrs after the exercise, signs of temporomandibular disorders and pressure-pain thresholds of the masticatory muscles were also recorded. Fatigue and pain scores had increased during the exercise (P < 0.001), but the reflex amplitude did not (P = 0.123). Twenty minutes after the exercises, 12 participants showed signs of myofascial pain or arthralgia. Pressure-pain thresholds were decreased after 20 min (P = 0.009) and 24 hrs (P = 0.049). Intense chewing can induce fatigue, pain, and decreased pressure-pain thresholds in the masticatory muscles, without concomitant changes in the jaw-stretch reflex amplitude.


1994 ◽  
Vol 40 (9) ◽  
pp. 974-980
Author(s):  
Koichi ASADA ◽  
Hiromasa NAGASHIMA ◽  
Yasuaki HASEGAWA ◽  
Yoichi NAKAGAWA ◽  
Katsunori ISHIBASHI

1995 ◽  
Vol 49 (4) ◽  
pp. 268-273
Author(s):  
Shinichi Masumi ◽  
Mayumi Ozamoto ◽  
Katsuaki Takeya ◽  
Masahiro Arita ◽  
Hirofumi Kido ◽  
...  

2021 ◽  
Vol 22 (1) ◽  
Author(s):  
René F. Castien ◽  
Michel W. Coppieters ◽  
Tom S. C. Durge ◽  
Gwendolyne G. M. Scholten-Peeters

Abstract Background Pressure pain thresholds (PPTs) are commonly assessed to quantify mechanical sensitivity in various conditions, including migraine. Digital and analogue algometers are used, but the concurrent validity between these algometers is unknown. Therefore, we assessed the concurrent validity between a digital and analogue algometer to determine PPTs in healthy participants and people with migraine. Methods Twenty-six healthy participants and twenty-nine people with migraine participated in the study. PPTs were measured interictally and bilaterally at the cephalic region (temporal muscle, C1 paraspinal muscles, and trapezius muscle) and extra-cephalic region (extensor carpi radialis muscle and tibialis anterior muscle). PPTs were first determined with a digital algometer, followed by an analogue algometer. Intraclass correlation coefficients (ICC3.1) and limits of agreement were calculated to quantify concurrent validity. Results The concurrent validity between algometers in both groups was moderate to excellent (ICC3.1 ranged from 0.82 to 0.99, with 95%CI: 0.65 to 0.99). Although PPTs measured with the analogue algometer were higher at most locations in both groups (p < 0.05), the mean differences between both devices were less than 18.3 kPa. The variation in methods, such as a hand-held switch (digital algometer) versus verbal commands (analogue algometer) to indicate when the threshold was reached, may explain these differences in scores. The limits of agreement varied per location and between healthy participants and people with migraine. Conclusion The concurrent validity between the digital and analogue algometer is excellent in healthy participants and moderate in people with migraine. Both types of algometer are well-suited for research and clinical practice but are not exchangeable within a study or patient follow-up.


2021 ◽  
Vol 0 (0) ◽  
Author(s):  
Morten Pallisgaard Støve ◽  
Rogerio Pessoto Hirata ◽  
Thorvaldur Skuli Palsson

Abstract Objectives The effect of stretching on joint range of motion is well documented, and although sensory perception has significance for changes in the tolerance to stretch following stretching the underlining mechanisms responsible for these changes is insufficiently understood. The aim of this study was to examine the influence of endogenous pain inhibitory mechanisms on stretch tolerance and to investigate the relationship between range of motion and changes in pain sensitivity. Methods Nineteen healthy males participated in this randomized, repeated-measures crossover study, conducted on 2 separate days. Knee extension range of motion, passive resistive torque, and pressure pain thresholds were recorded before, after, and 10 min after each of four experimental conditions; (i) Exercise-induced hypoalgesia, (ii) two bouts of static stretching, (iii) resting, and (iv) a remote, painful stimulus induced by the cold pressor test. Results Exercise-induced hypoalgesia and cold pressor test caused an increase in range of motion (p<0.034) and pressure pain thresholds (p<0.027). Moderate correlations in pressure pain thresholds were found between exercise-induced hypoalgesia and static stretch (Rho>0.507, p=0.01) and exercise-induced hypoalgesia and the cold pressor test (Rho=0.562, p=0.01). A weak correlation in pressure pain thresholds and changes in range of motion were found following the cold pressor test (Rho=0.460, p=0.047). However, a potential carryover hypoalgesic effect may have affected the results of the static stretch. Conclusions These results suggest that stretch tolerance may be linked with endogenous modulation of pain. Present results suggest, that stretch tolerance may merely be a marker for pain sensitivity which may have clinical significance given that stretching is often prescribed in the rehabilitation of different musculoskeletal pain conditions where reduced endogenous pain inhibition is frequently seen.


Pain ◽  
2014 ◽  
Vol 155 (10) ◽  
pp. 2134-2143 ◽  
Author(s):  
Gary D. Slade ◽  
Anne E. Sanders ◽  
Richard Ohrbach ◽  
Roger B. Fillingim ◽  
Ron Dubner ◽  
...  

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