scholarly journals Neuroendoscopic Transventricular Surgery for Suprasellar Cystic Mass Lesions Such as Cystic Craniopharyngioma and Rathke Cleft Cyst

2004 ◽  
Vol 44 (8) ◽  
pp. 408-415 ◽  
Author(s):  
Yukiko NAKAHARA ◽  
Hisao KOGA ◽  
Kenji MAEDA ◽  
Masashi TAKAGI ◽  
Kazuo TABUCHI
Neurosurgery ◽  
1991 ◽  
Vol 28 (2) ◽  
pp. 231-237 ◽  
Author(s):  
William T. Couldwell ◽  
Chi-Shing Zee ◽  
Michael L. J. Apuzzo

Abstract With increasing immigration from endemic regions, the incidence of neurocysticercosis in North America is rising. This retrospective study was undertaken to examine the role of surgery in those cases presenting with large cystic parenchymal and cisternal lesions in the current era of anthelminthic agents administered orally. A total of 237 patients presented with newly diagnosed neurocysticercosis to our institution over a recent 5-year period (mean age, 31.2 years). Among those who presented with cystic mass lesions predominantly affecting the brain parenchyma and cisternal spaces. 20 (8.4%; mean age, 40.2 years) with large cystic lesions subsequently underwent surgical intervention, either because of an emergent presentation or because they were refractory to medical management. Clinical presentation included increased intracranial pressure, focal neurological deficit, and seizure. Radiographic imaging (computed tomography and/or magnetic resonance imaging) demonstrated 12 cases with cisternal lesions, 7 with parenchymal lesions, and 1 involving both compartments. Based on imaging guidelines, 30 operative procedures (excluding shunt revisions) were performed (14 craniotomies, 8 cerebrospinal fluid diversions, 7 stereotactic procedures, and 1 burr hole drainage). Fifteen (75%) showed neurological or symptomatic improvement over a median follow-up period of 36.4 months. There were three surgery-related complications and no deaths.


2000 ◽  
Vol 92 (2) ◽  
pp. 350-354 ◽  
Author(s):  
Christopher B. Michael ◽  
Andrew G. Lee ◽  
James R. Patrinely ◽  
Samuel Stal ◽  
J. Bob Blacklock

✓ The authors present a case of visual loss associated with fibrous dysplasia of the anterior skull base and the surgical management of this case. Preoperative computerized tomography scanning in this patient demonstrated a patent optic foramen and a rapidly growing cystic mass within the orbit, which was responsible for the patient's visual loss. A literature review revealed that this case is typical, in that cystic mass lesions of various types are frequently responsible for visual loss associated with fibrous dysplasia. The authors did not find significant evidence in the literature to support the notion that visual loss associated with fibrous dysplasia is the result of progressive optic canal stenosis, thus raising questions about the value of prophylactic optic canal decompression. Instead, as demonstrated by this case and those uncovered in the literature review, most instances of visual loss result from the rapid growth of mass lesions of cystic fibrous dysplasia, mucoceles, or hemorrhage. Findings of the literature review and the present case of fibrous dysplasia of the anterior skull base support a role for extensive surgical resection in these cases and indicate a need for additional prospective analysis of a larger number of patients with this disease.


2016 ◽  
Vol 32 (9) ◽  
pp. 1625-1632 ◽  
Author(s):  
Yasuhiko Hayashi ◽  
Daisuke Kita ◽  
Issei Fukui ◽  
Yasuo Sasagawa ◽  
Masahiro Oishi ◽  
...  

2006 ◽  
Vol 66 (3) ◽  
pp. 246-250 ◽  
Author(s):  
Jaypal Sangala Reddy ◽  
Asht Mangal Mishra ◽  
Sanjay Behari ◽  
Mazhar Husain ◽  
Vikas Gupta ◽  
...  

2019 ◽  
Vol 41 (1) ◽  
pp. 106-110
Author(s):  
M. Azuma ◽  
Z.A. Khant ◽  
M. Kitajima ◽  
H. Uetani ◽  
T. Watanabe ◽  
...  

Author(s):  
S.L. Asa ◽  
K. Kovacs ◽  
J. M. Bilbao ◽  
R. G. Josse ◽  
K. Kreines

Seven cases of lymphocytic hypophysitis in women have been reported previously in association with various degrees of hypopituitarism. We report two pregnant patients who presented with mass lesions of the sella turcica, clinically mimicking pituitary adenoma. However, pathologic examination revealed extensive infiltration of the anterior pituitary by lymphocytes and plasma cells with destruction of the gland. To our knowledge, the ultrastructural features of lymphocytic hypophysitis have not been studied so far.For transmission electron microscopy, tissue from surgical specimens was fixed in glutaraldehyde, postfixed in OsO4, dehydrated and embedded in epoxy-resin. Ultrathin sections were stained with uranyl acetate and lead citrate and examined with a Philips 300 electron microscope.Electron microscopy revealed adenohypophysial cells of all types exhibiting varying degrees of injury. In the areas of most dense inflammatory cell infiltration pituitary cells contained large lysosomal bodies fusing with secretory granules (Fig. 1), as well as increased numbers of swollen mitochondria, indicating oncocytic transformation (Fig. 2).


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