scholarly journals Malignant Primary Meningeal Tumors

1974 ◽  
Vol 14pt1 (SUPPLEMENT) ◽  
pp. 35-46 ◽  
Author(s):  
Masashi FUKUI ◽  
Katsutoshi KITAMURA ◽  
Yukichi YONEMASU ◽  
Tosuke TAKAKI ◽  
Tohru SOEJIMA ◽  
...  
Keyword(s):  
Meningeal Tumors

The deposition of Hg203-chlormerodrin in experimental brain tumors

1971 ◽  
Vol 35 (3) ◽  
pp. 303-308 ◽  
Author(s):  
Tatsuya Kobayashi ◽  
Louis Bakay ◽  
Joseph C. Lee
Keyword(s):  
Brain Tumors ◽  
Tumor Cells ◽  
Normal Brain ◽  
Intracranial Tumors ◽  
Electron Microscopic ◽  
Electron Microscopic Autoradiography ◽  
Content Type ◽  
Meningeal Tumors ◽  
Vacuolar System ◽  
Fine Print

✓ The deposition of Hg203-chlormerodrin was studied in intracranial tumors in mice induced by implantation of 20-methyl cholanthrene by tissue assay, as well as light microscopic and electron microscopic autoradiography. The investigations were carried out in astrocytomas, glioblastomas, and meningeal tumors. The chlormerodrin content of the tumors exceeded that of normal brain with a significant tumor/brain ratio ranging from 5.8 to 22.5. It was found that the chlormerodrin molecule becomes rapidly incorporated in the tumor cells, with a preference for that portion of the cytoplasm associated with the vacuolar system.

scholarly journals Meningeal Tumors Induced in Calves with the Bovine Cutaneous Papilloma Virus

1969 ◽  
Vol 6 (2) ◽  
pp. 135-145 ◽  
Author(s):  
D. F. Brobst ◽  
G. C. Dulac
Keyword(s):  
Blood Vessels ◽  
Viral Antigen ◽  
Papilloma Virus ◽  
Meningeal Tumors ◽  
The Brain

Fibromatous tumors were induced in the meninges of calves by inoculating the meninges with a suspension of bovine cutaneous papillomas or by implanting bovine cutaneous papillomas into the brain. Meningeal tumors were observed to occur as early as 20 days after inoculation. Meningeal tumors from calves killed 90 and 145 days after inoculation extended into the brain along the course of blood vessels. Metastasis, however, was not observed. Evidence that the induced meningeal tumors contained viral antigen was lacking.

scholarly journals A Frontal Lobe Meningioma in a Child Leading to Visual Loss

2015 ◽  
Vol 2015 ◽  
pp. 1-3 ◽  
Author(s):  
Nedime Sahinoglu-Keşkek ◽  
Gokhan Soker ◽  
Şakir Özgür Keşkek ◽  
Sehire Sahinoglu ◽  
Figen Unal ◽  
...  
Keyword(s):  
Visual Loss ◽  
Clinical Presentation ◽  
Low Vision ◽  
Optic Chiasm ◽  
Intracranial Tumor ◽  
Intracranial Meningioma ◽  
Resonance Imaging ◽  
Blurred Vision ◽  
History Of ◽  
Meningeal Tumors

Objective. Meningiomas are benign primary meningeal tumors and are seen rare in children and adolescents.Clinical Presentation and Intervention. A 15-year-old Turkish boy reported a 1-month history of headache and blurred vision in both eyes. His visual acuity was 0.3 in both eyes with papilledema. Magnetic resonance imaging showed a77×97×77 mm intracranial-extra-axial frontal lesion which compresses the chiasm. He was diagnosed with intracranial meningioma and referred to neurosurgery clinic.Conclusion. Ophthalmologists should be aware of the fact that papilledema and low vision can be caused by an intracranial tumor which compresses optic chiasm.

Meningeal Tumors in Children

1994 ◽  
Vol 20 (3) ◽  
pp. 207-213 ◽  
Author(s):  
Luis Schut ◽  
Alexa I. Canady ◽  
Leslie N. Sutton ◽  
Derek A. Bruce
Keyword(s):  
Meningeal Tumors

Radical Removal of Recurrent Malignant Meningeal Tumors of the Cavernous Sinus in Combination with High-Flow Bypass

2015 ◽  
Vol 83 (4) ◽  
pp. 424-430 ◽  
Author(s):  
Masahiko Wanibuchi ◽  
Yukinori Akiyama ◽  
Takeshi Mikami ◽  
Satoshi Iihoshi ◽  
Kei Miyata ◽  
...  
Keyword(s):  
Cavernous Sinus ◽  
High Flow ◽  
Meningeal Tumors ◽  
Radical Removal

Microsurgical Anatomy of the Meningeal Branch of the Dorsolateral Medullary Plexus

2019 ◽  
Author(s):  
Ali Tayebi Meybodi ◽  
Xiaochun Zhao ◽  
Leandro Borba Moreira ◽  
Mark C Preul ◽  
Lea M Alhilali ◽  
...  
Keyword(s):  
Anatomic Variation ◽  
Posterior Inferior Cerebellar Artery ◽  
Microsurgical Anatomy ◽  
Anatomical Features ◽  
Arteriovenous Fistulas ◽  
Cerebellar Artery ◽  
Dural Arteriovenous Fistulas ◽  
Far Lateral ◽  
Meningeal Tumors ◽  
Actual Prevalence

Abstract BACKGROUND Meningeal branches originating from intradural arteries may be involved in several diseases such as meningeal tumors and arteriovenous lesions. These “pial-dural” arterial connections have been described for anterior cerebral, posterior cerebral, and cerebellar arteries. However, to the best of our knowledge, meningeal supply originating from the arterial plexus over the dorsolateral aspect of the medulla oblongata (dorsolateral medullary plexus [DLMP]) has not been described. OBJECTIVE To define the microsurgical anatomy of the meningeal branch of DLMP. METHODS A total of 20 cadaver heads (40 sides) underwent far-lateral craniotomy and the cerebellomedullary cisterns were explored to find the DLMP and any meningeal branches. Additionally, de-identified intraoperative images of 85 patients with vertebral artery (VA)/posterior inferior cerebellar artery aneurysms who had undergone far-lateral craniotomy were studied to find any meningeal branches of DLMP. RESULTS The meningeal branches of DLMP were identified in 4 cadavers/sides. These branches reached the region of jugular tubercle (JT) after crossing the accessory nerve. In 3 specimens, these branches were joined by a small twig from V4-VA before penetrating the dura. DLMP meningeal branches were found in 12 patients of the studied cohort (14%) with similar anatomical features as those found in the cadaveric study. CONCLUSION DLMP may give rise to meningeal branches to the adjacent dura of JT. The actual prevalence of this anatomic variation is difficult to estimate using our data. However, when present, these branches may have important clinical implications, ie, diseases such as dural arteriovenous fistulas, pial arteriovenous malformations (AVMs), and meningeal-based tumors.

scholarly journals A retrovirus carrying the K-fgf oncogene induces diffuse meningeal tumors and soft-tissue fibrosarcomas.

1993 ◽  
Vol 13 (4) ◽  
pp. 1998-2010 ◽  
Author(s):  
D Talarico ◽  
M M Ittmann ◽  
R Bronson ◽  
C Basilico
Keyword(s):  
Growth Factor ◽  
Soft Tissue ◽  
Fusion Protein ◽  
Murine Leukemia Virus ◽  
Leukemia Virus ◽  
Moloney Murine Leukemia Virus ◽  
Oncogenic Potential ◽  
Meningeal Tumors ◽  
Murine Leukemia

The K-fgf/hst oncogene encodes a growth factor of the fibroblast growth factor (FGF) family and transforms cells through an autocrine mechanism which requires extracellular activation of its receptor(s). To identify the cell and tissue targets of K-fgf oncogenic potential in vivo, we constructed a recombinant retrovirus carrying the human K-fgf cDNA and injected it, together with helper Moloney murine leukemia virus, into immunocompetent as well as nude mice. The original construct was highly transforming in tissue culture but produced no detectable pathologies in vivo with the exception of a single fibrosarcoma which arose after a long latency. The virus produced by this tumor appears to have undergone a complex series of recombination events involving the helper Moloney murine leukemia virus. It encodes an Env/K-FGF fusion protein whose expression is under the control of a hybrid long terminal repeat. This virus (designated MFS, for meningeal fibrosarcoma) induces tumors in mice with high frequency and short latency. These neoplasms consist of aggressive fibrosarcomas of soft tissue as well as diffuse meningeal tumors originating from the dura mater that surround the whole central nervous system and cause severe hydrocephalus. The Env/K-FGF fusion protein expressed by the MFS virus has retained all of the biological properties of native K-FGF, including secretion, mitogenic activity, heparin binding, and neutralization by anti-K-FGF antibodies. These and other results indicate that the tumors induced by the MFS virus result from the oncogenic potential of K-FGF.

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